Temat: "glioma" - OpacWWW - Prolib Integro

Skocz do pozycji: 1.

Tytuł:: A novel cuproptosis-related gene signature to predict prognosis in Glioma.
Autorzy:: Zhang M; Department of Neurobiology, School of Life Sciences, China Medical University, Shenyang, 110122, China.; Key Laboratory of Neuro-Oncology in Liaoning Province, Shenyang, 110004, China.
Liu X; Key Laboratory of Neuro-Oncology in Liaoning Province, Shenyang, 110004, China.; Department of Neurosurgery, Shengjing Hospital of China Medical University, Shenyang, 110004, China.
Wang D; Key Laboratory of Neuro-Oncology in Liaoning Province, Shenyang, 110004, China.; Department of Neurosurgery, Shengjing Hospital of China Medical University, Shenyang, 110004, China.
Ruan X; Department of Neurobiology, School of Life Sciences, China Medical University, Shenyang, 110122, China.; Key Laboratory of Neuro-Oncology in Liaoning Province, Shenyang, 110004, China.
Wang P; Department of Neurobiology, School of Life Sciences, China Medical University, Shenyang, 110122, China.; Key Laboratory of Neuro-Oncology in Liaoning Province, Shenyang, 110004, China.
Liu L; Department of Neurobiology, School of Life Sciences, China Medical University, Shenyang, 110122, China.; Key Laboratory of Neuro-Oncology in Liaoning Province, Shenyang, 110004, China.
Xue Y; Department of Neurobiology, School of Life Sciences, China Medical University, Shenyang, 110122, China. .; Key Laboratory of Neuro-Oncology in Liaoning Province, Shenyang, 110004, China. .; Pokaż więcej
Źródło:: BMC cancer [BMC Cancer] 2023 Mar 13; Vol. 23 (1), pp. 237. Date of Electronic Publication: 2023 Mar 13.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis*
Glioma*/genetics
MicroRNAs*/genetics
Humans ; Cell Death ; Copper ; Nomograms ; Prognosis

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Skocz do pozycji: 2.

Tytuł:: Novel Cuproptosis-Related Gene Signature for Precise Identification of High-Risk Populations in Low-Grade Gliomas.
Autorzy:: Chen P; Department of Neurosurgery, The Second Affiliated Hospital of Hengyang Medical College, Hengyang, 421000 Hunan, China.; Department of Neurosurgery, China-Japan Union Hospital of Jilin University, Changchun, 130000 Jilin, China.
Han H; Department of Radiation Oncology, The Second Hospital of Jilin University, Changchun 130022, China.
Wang X; Department of Neurosurgery, China-Japan Union Hospital of Jilin University, Changchun, 130000 Jilin, China.
Wang B; Department of Neurosurgery, The Second Affiliated Hospital of Hengyang Medical College, Hengyang, 421000 Hunan, China.
Wang Z; Department of Neurosurgery, China-Japan Union Hospital of Jilin University, Changchun, 130000 Jilin, China.; Pokaż więcej
Źródło:: Mediators of inflammation [Mediators Inflamm] 2023 Feb 13; Vol. 2023, pp. 6232620. Date of Electronic Publication: 2023 Feb 13 (Print Publication: 2023).
Typ publikacji:: Journal Article
MeSH Terms:: Algorithms*
Glioma*
Apoptosis*
Humans ; Cell Death ; Tumor Microenvironment ; Copper

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Skocz do pozycji: 3.

Tytuł:: EREG is the core onco-immunological biomarker of cuproptosis and mediates the cross-talk between VEGF and CD99 signaling in glioblastoma.
Autorzy:: Zhou Y; Department of Neurosurgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1277 Jiefang Avenue, Wuhan, 430022, Hubei, China.
Xiao D; Department of Neurosurgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1277 Jiefang Avenue, Wuhan, 430022, Hubei, China.
Jiang X; Department of Neurosurgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1277 Jiefang Avenue, Wuhan, 430022, Hubei, China. .
Nie C; Department of Neurosurgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1277 Jiefang Avenue, Wuhan, 430022, Hubei, China. .; Pokaż więcej
Źródło:: Journal of translational medicine [J Transl Med] 2023 Jan 16; Vol. 21 (1), pp. 28. Date of Electronic Publication: 2023 Jan 16.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Epiregulin*
Glioblastoma*/genetics
Glioma*/genetics
Apoptosis*
Humans ; 12E7 Antigen ; Biomarkers ; Intercellular Signaling Peptides and Proteins ; Vascular Endothelial Growth Factor A

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Skocz do pozycji: 4.

Tytuł:: Endoplasmic Reticulum Stress Contributes to Gefitinib-Induced Apoptosis in Glioma.
Autorzy:: Chang CY; Department of Surgery, Feng Yuan Hospital, Taichung 420, Taiwan.
Pan PH; Department of Pediatrics, Tungs' Taichung MetroHarbor Hospital, Taichung 435, Taiwan.; Department of Veterinary Medicine, National Chung Hsing University, Taichung 402, Taiwan.
Wu CC; Department of Anesthesiology, Taichung Veterans General Hospital, Taichung 407, Taiwan.
Liao SL; Department of Medical Research, Taichung Veterans General Hospital, Taichung 407, Taiwan.
Chen WY; Department of Veterinary Medicine, National Chung Hsing University, Taichung 402, Taiwan.
Kuan YH; Department of Pharmacology, Chung Shan Medical University, Taichung 402, Taiwan.
Wang WY; Department of Nursing, HungKuang University, Taichung 433, Taiwan.
Chen CJ; Department of Medical Research, Taichung Veterans General Hospital, Taichung 407, Taiwan.; Department of Medical Laboratory Science and Biotechnology, China Medical University, Taichung 404, Taiwan.; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2021 Apr 11; Vol. 22 (8). Date of Electronic Publication: 2021 Apr 11.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Endoplasmic Reticulum Stress/*drug effects
Gefitinib/*pharmacology
Glioma/*drug therapy
Signal Transduction/*drug effects
Cell Line, Tumor ; Glioma/metabolism ; Glioma/pathology ; Humans ; Neoplasm Proteins/metabolism

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Skocz do pozycji: 5.

Tytuł:: Pterostilbene promotes mitochondrial apoptosis and inhibits proliferation in glioma cells.
Autorzy:: Gao H; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, Jilin Province, China.; Department of Neurosurgery, Fenyang Hospital of Shanxi Province, Fengyang, 032200, Shanxi Province, China.
Liu Z; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, Jilin Province, China.
Xu W; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, Jilin Province, China.
Wang Q; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, Jilin Province, China.
Zhang C; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, Jilin Province, China.
Ding Y; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, Jilin Province, China.
Nie W; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, Jilin Province, China.
Lai J; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, Jilin Province, China.
Chen Y; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, Jilin Province, China. chen_.
Huang H; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, Jilin Province, China. .; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2021 Mar 18; Vol. 11 (1), pp. 6381. Date of Electronic Publication: 2021 Mar 18.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis/*drug effects
Cell Proliferation/*drug effects
Glioma/*drug therapy
Stilbenes/*pharmacology
Animals ; Cell Line, Tumor ; Cell Survival/drug effects ; Gene Expression Regulation, Neoplastic/drug effects ; Glioma/genetics ; Glioma/pathology ; Humans ; MAP Kinase Signaling System/drug effects ; Membrane Potential, Mitochondrial/drug effects ; Mitochondria/drug effects ; Rats ; Reactive Oxygen Species/metabolism ; Xenograft Model Antitumor Assays

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Skocz do pozycji: 6.

Tytuł:: Tubeimoside‑1 induces apoptosis in human glioma U251 cells by suppressing PI3K/Akt‑mediated signaling pathways.
Autorzy:: Cao LJ; Department of Pediatrics, Affiliated Hospital of Inner Mongolia University for Nationalities, Tongliao, Inner Mongolia Autonomous Region 028007, P.R. China.
Xie HT; Department of Pediatrics, Affiliated Hospital of Inner Mongolia University for Nationalities, Tongliao, Inner Mongolia Autonomous Region 028007, P.R. China.
Chu ZX; Department of Pediatrics, Affiliated Hospital of Inner Mongolia University for Nationalities, Tongliao, Inner Mongolia Autonomous Region 028007, P.R. China.
Ma Y; Department of Pediatrics, Affiliated Hospital of Inner Mongolia University for Nationalities, Tongliao, Inner Mongolia Autonomous Region 028007, P.R. China.
Wang MM; Department of Pediatrics, Affiliated Hospital of Inner Mongolia University for Nationalities, Tongliao, Inner Mongolia Autonomous Region 028007, P.R. China.
Shi Z; Department of Mongolian Medicine Hand Foot Surgery, Affiliated Hospital of Inner Mongolia University for Nationalities, Tongliao, Inner Mongolia Autonomous Region 028007, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2020 Aug; Vol. 22 (2), pp. 1527-1535. Date of Electronic Publication: 2020 Jun 11.
Typ publikacji:: Journal Article
MeSH Terms:: Antineoplastic Agents, Phytogenic/*pharmacology
Apoptosis/*drug effects
Glioma/*pathology
Phosphatidylinositol 3-Kinases/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Saponins/*pharmacology
Triterpenes/*pharmacology
Astrocytes ; CDC2 Protein Kinase/metabolism ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Cyclin B1/metabolism ; G2 Phase Cell Cycle Checkpoints/drug effects ; Glioma/drug therapy ; Humans ; Proto-Oncogene Proteins c-bcl-2/metabolism

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Skocz do pozycji: 7.

Tytuł:: Effects of small nucleolar RNA SNORD44 on the proliferation, apoptosis and invasion of glioma cells.
Autorzy:: Xia XR; Department of Clinical Laboratory, Taihe Hospital, Hubei University of Medicine, No. 32, South Renmin Road, Shiyan, 442000, Hubei, China.
Li WC; Department of Clinical Laboratory, Taihe Hospital, Hubei University of Medicine, No. 32, South Renmin Road, Shiyan, 442000, Hubei, China.
Yu ZT; Department of Clinical Laboratory, Taihe Hospital, Hubei University of Medicine, No. 32, South Renmin Road, Shiyan, 442000, Hubei, China.
Li J; Department of Clinical Laboratory, Taihe Hospital, Hubei University of Medicine, No. 32, South Renmin Road, Shiyan, 442000, Hubei, China.
Peng CY; Department of Clinical Laboratory, Taihe Hospital, Hubei University of Medicine, No. 32, South Renmin Road, Shiyan, 442000, Hubei, China.
Jin L; Department of Clinical Laboratory, Taihe Hospital, Hubei University of Medicine, No. 32, South Renmin Road, Shiyan, 442000, Hubei, China.
Yuan GL; Department of Clinical Laboratory, Taihe Hospital, Hubei University of Medicine, No. 32, South Renmin Road, Shiyan, 442000, Hubei, China. yuan_g_.; Pokaż więcej
Źródło:: Histochemistry and cell biology [Histochem Cell Biol] 2020 Apr; Vol. 153 (4), pp. 257-269. Date of Electronic Publication: 2020 Feb 15.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis*
Central Nervous System Neoplasms/*metabolism
Central Nervous System Neoplasms/*pathology
Glioma/*metabolism
Glioma/*pathology
RNA, Small Nucleolar/*metabolism
Animals ; Cell Line ; Cell Proliferation ; Central Nervous System Neoplasms/genetics ; Female ; Glioma/genetics ; Humans ; Male ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Middle Aged ; RNA, Long Noncoding/genetics ; RNA, Long Noncoding/metabolism ; RNA, Small Nucleolar/genetics

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Skocz do pozycji: 8.

Tytuł:: Differential suitability of reactive oxygen species and the role of glutathione in regulating paradoxical behavior in gliomas: A mathematical perspective.
Autorzy:: Bhowmick R; Chemical Engineering and Process Development Division, CSIR-National Chemical Laboratory, Pune, Maharashtra, India.; Academy of Scientific & Innovative Research (AcSIR), Ghaziabad, India.
Sarkar RR; Chemical Engineering and Process Development Division, CSIR-National Chemical Laboratory, Pune, Maharashtra, India.; Academy of Scientific & Innovative Research (AcSIR), Ghaziabad, India.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2020 Jun 25; Vol. 15 (6), pp. e0235204. Date of Electronic Publication: 2020 Jun 25 (Print Publication: 2020).
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis*
Models, Biological*
Glioma/*metabolism
Glutathione/*metabolism
Reactive Oxygen Species/*metabolism
Glioma/pathology ; Humans ; Neoplasm Proteins/metabolism ; Oxidoreductases/metabolism

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Skocz do pozycji: 9.

Tytuł:: microRNA cluster MC-let-7a-1~let-7d promotes autophagy and apoptosis of glioma cells by down-regulating STAT3.
Autorzy:: Yang ZY; Department of Neurosurgery, Xiangya Hospital Central South University, Changsha, China.
Wang Y; Department of Pathology, Xiangya Medical School of Central South University & Xiangya Hospital Central South University, Changsha, China.
Liu Q; Department of Neurosurgery, Xiangya Hospital Central South University, Changsha, China.
Wu M; Department of Neurosurgery, Xiangya Hospital Central South University, Changsha, China.; Pokaż więcej
Źródło:: CNS neuroscience & therapeutics [CNS Neurosci Ther] 2020 Mar; Vol. 26 (3), pp. 319-331. Date of Electronic Publication: 2019 Dec 23.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis/*physiology
Autophagy/*physiology
Brain Neoplasms/*metabolism
Glioma/*metabolism
MicroRNAs/*metabolism
STAT3 Transcription Factor/*metabolism
Adolescent ; Adult ; Aged ; Animals ; Brain Neoplasms/pathology ; Cell Line, Tumor ; Down-Regulation/physiology ; Female ; Glioma/pathology ; Humans ; Male ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Middle Aged ; STAT3 Transcription Factor/antagonists & inhibitors ; Young Adult

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Skocz do pozycji: 10.

Tytuł:: Endoplasmic Reticulum Stress Contributes to Indomethacin-Induced Glioma Apoptosis.
Autorzy:: Chang CY; Department of Surgery, Feng Yuan Hospital, Taichung City 420, Taiwan.
Li JR; Division of Urology, Taichung Veterans General Hospital, Taichung City 407, Taiwan.
Wu CC; Department of Anesthesiology, Taichung Veterans General Hospital, Taichung City 407, Taiwan.; Department of Financial Engineering, Providence University, Taichung City 433, Taiwan.; Department of Data Science and Big Data Analytics, Providence University, Taichung City 433, Taiwan.
Wang JD; Children's Medical Center, Taichung Veterans General Hospital, Taichung City 407, Taiwan.; Department of Industrial Engineering and Enterprise Information, Tunghai University, Taichung City 407, Taiwan.
Liao SL; Department of Medical Research, Taichung Veterans General Hospital, Taichung City 407, Taiwan.
Chen WY; Department of Veterinary Medicine, National Chung Hsing University, Taichung City 402, Taiwan.
Wang WY; Department of Nursing, HungKuang University, Taichung City 433, Taiwan.
Chen CJ; Department of Medical Research, Taichung Veterans General Hospital, Taichung City 407, Taiwan.; Department of Medical Laboratory Science and Biotechnology, China Medical University, Taichung City 404, Taiwan.; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2020 Jan 15; Vol. 21 (2). Date of Electronic Publication: 2020 Jan 15.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Endoplasmic Reticulum Stress/*drug effects
Glioma/*metabolism
Indomethacin/*pharmacology
Anti-Inflammatory Agents, Non-Steroidal/pharmacology ; Calcium/metabolism ; Caspases/metabolism ; Cell Line, Tumor ; Enzyme Activation/drug effects ; Glioma/pathology ; Humans ; MAP Kinase Kinase Kinase 5/metabolism ; Mitochondria/drug effects ; Mitochondria/metabolism ; Protein Phosphatase 2/metabolism ; Reactive Oxygen Species/metabolism ; Signal Transduction/drug effects

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Skocz do pozycji: 11.

Tytuł:: Second Generation mTOR Inhibitors as a Double-Edged Sword in Malignant Glioma Treatment.
Autorzy:: Heinzen D; Dr. Senckenberg Institute of Neurooncology, University Hospital Frankfurt, Schleusenweg 2-16, 60528 Frankfurt am Main, Germany. .
Divé I; Dr. Senckenberg Institute of Neurooncology, University Hospital Frankfurt, Schleusenweg 2-16, 60528 Frankfurt am Main, Germany. .; University Cancer Center Frankfurt (UCT), University Hospital Frankfurt, Theodor-Stern-Kai 7, 60590 Frankfurt am Main, Germany. .; German Cancer Consortium (DKTK), Partner Site Frankfurt/Mainz, Theodor-Stern-Kai 7, 60590 Frankfurt am Main, Germany. .
Lorenz NI; Dr. Senckenberg Institute of Neurooncology, University Hospital Frankfurt, Schleusenweg 2-16, 60528 Frankfurt am Main, Germany. .; University Cancer Center Frankfurt (UCT), University Hospital Frankfurt, Theodor-Stern-Kai 7, 60590 Frankfurt am Main, Germany. .; German Cancer Consortium (DKTK), Partner Site Frankfurt/Mainz, Theodor-Stern-Kai 7, 60590 Frankfurt am Main, Germany. .
Luger AL; Dr. Senckenberg Institute of Neurooncology, University Hospital Frankfurt, Schleusenweg 2-16, 60528 Frankfurt am Main, Germany. .; University Cancer Center Frankfurt (UCT), University Hospital Frankfurt, Theodor-Stern-Kai 7, 60590 Frankfurt am Main, Germany. .; German Cancer Consortium (DKTK), Partner Site Frankfurt/Mainz, Theodor-Stern-Kai 7, 60590 Frankfurt am Main, Germany. .
Steinbach JP; Dr. Senckenberg Institute of Neurooncology, University Hospital Frankfurt, Schleusenweg 2-16, 60528 Frankfurt am Main, Germany. .; University Cancer Center Frankfurt (UCT), University Hospital Frankfurt, Theodor-Stern-Kai 7, 60590 Frankfurt am Main, Germany. .; German Cancer Consortium (DKTK), Partner Site Frankfurt/Mainz, Theodor-Stern-Kai 7, 60590 Frankfurt am Main, Germany. .; Frankfurt Cancer Institute (FCI), University Hospital Frankfurt, Paul-Ehrlich-Straße 42-44, 60596 Frankfurt am Main, Germany. .
Ronellenfitsch MW; Dr. Senckenberg Institute of Neurooncology, University Hospital Frankfurt, Schleusenweg 2-16, 60528 Frankfurt am Main, Germany. .; University Cancer Center Frankfurt (UCT), University Hospital Frankfurt, Theodor-Stern-Kai 7, 60590 Frankfurt am Main, Germany. .; German Cancer Consortium (DKTK), Partner Site Frankfurt/Mainz, Theodor-Stern-Kai 7, 60590 Frankfurt am Main, Germany. .; Frankfurt Cancer Institute (FCI), University Hospital Frankfurt, Paul-Ehrlich-Straße 42-44, 60596 Frankfurt am Main, Germany. .; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2019 Sep 10; Vol. 20 (18). Date of Electronic Publication: 2019 Sep 10.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Cell Proliferation/*drug effects
Glioma/*metabolism
Protein Kinase Inhibitors/*pharmacology
Signal Transduction/*drug effects
TOR Serine-Threonine Kinases/*antagonists & inhibitors
Benzoxazoles/pharmacology ; Cell Cycle Checkpoints/drug effects ; Cell Line, Tumor ; Glioma/drug therapy ; Glioma/pathology ; Humans ; Imidazoles/pharmacology ; Mechanistic Target of Rapamycin Complex 1/antagonists & inhibitors ; Mechanistic Target of Rapamycin Complex 1/metabolism ; Mechanistic Target of Rapamycin Complex 2/antagonists & inhibitors ; Mechanistic Target of Rapamycin Complex 2/metabolism ; Naphthyridines/pharmacology ; Pyrimidines/pharmacology ; Quinolines/pharmacology ; Sirolimus/pharmacology ; TOR Serine-Threonine Kinases/metabolism

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Skocz do pozycji: 12.

Tytuł:: Transcriptional CDK Inhibitors CYC065 and THZ1 Induce Apoptosis in Glioma Stem Cells Derived from Recurrent GBM.
Autorzy:: Juric V; Department of Physiology & Medical Physics, Royal College of Surgeons in Ireland, University of Medicine and Health Sciences, Dublin 2, Ireland.
Düssmann H; Department of Physiology & Medical Physics, Royal College of Surgeons in Ireland, University of Medicine and Health Sciences, Dublin 2, Ireland.; Centre for Systems Medicine, Royal College of Surgeons in Ireland, University of Medicine and Health Sciences, Dublin 2, Ireland.
Lamfers MLM; Brain Tumor Center, Department of Neurosurgery, Erasmus MC, 3015 CN Rotterdam, The Netherlands.
Prehn JHM; Department of Physiology & Medical Physics, Royal College of Surgeons in Ireland, University of Medicine and Health Sciences, Dublin 2, Ireland.; Centre for Systems Medicine, Royal College of Surgeons in Ireland, University of Medicine and Health Sciences, Dublin 2, Ireland.
Rehm M; Institute of Cell Biology and Immunology, University of Stuttgart, 70569 Stuttgart, Germany.; Stuttgart Research Center Systems Biology, University of Stuttgart, 70569 Stuttgart, Germany.
Murphy BM; Department of Physiology & Medical Physics, Royal College of Surgeons in Ireland, University of Medicine and Health Sciences, Dublin 2, Ireland.; Pokaż więcej
Źródło:: Cells [Cells] 2021 May 12; Vol. 10 (5). Date of Electronic Publication: 2021 May 12.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Drug Resistance, Neoplasm*
Adenosine/*analogs & derivatives
Apoptosis/*drug effects
Brain Neoplasms/*drug therapy
Glioma/*drug therapy
Temozolomide/*administration & dosage
AC133 Antigen/metabolism ; Adenosine/administration & dosage ; Adult ; Antineoplastic Agents/pharmacology ; Cell Cycle Checkpoints/drug effects ; Cell Line, Tumor/cytology ; Cell Proliferation/drug effects ; Humans ; Hyaluronan Receptors/metabolism ; Male ; Middle Aged ; Neoplasm Recurrence, Local/metabolism ; Neoplastic Stem Cells/pathology

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Skocz do pozycji: 13.

Tytuł:: Circ‑TOP2A acts as a ceRNA for miR‑346 and contributes to glioma progression via the modulation of sushi domain‑containing 2.
Autorzy:: Sang J; Department of Magnetic Resonance Imaging, The Second Affiliated Hospital of Qiqihar Medical University, Qiqihar, Heilongjiang 161000, P.R. China.
Li X; Department of Ultrasonics, The Second Affiliated Hospital of Qiqihar Medical University, Qiqihar, Heilongjiang 161000, P.R. China.
Lv L; Department of Neurology, The Second Affiliated Hospital of Qiqihar Medical University, Qiqihar, Heilongjiang 161000, P.R. China.
Zhang C; Department of Magnetic Resonance Imaging, The Second Affiliated Hospital of Qiqihar Medical University, Qiqihar, Heilongjiang 161000, P.R. China.
Zhang X; Department of Magnetic Resonance Imaging, The Second Affiliated Hospital of Qiqihar Medical University, Qiqihar, Heilongjiang 161000, P.R. China.
Li G; Department of Magnetic Resonance Imaging, The Second Affiliated Hospital of Qiqihar Medical University, Qiqihar, Heilongjiang 161000, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2021 Apr; Vol. 23 (4). Date of Electronic Publication: 2021 Feb 04.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis*
Cell Movement*
Membrane Glycoproteins/*metabolism
MicroRNAs/*metabolism
Neoplasm Proteins/*metabolism
RNA, Circular/*metabolism
RNA, Neoplasm/*metabolism
Cell Line, Tumor ; Glioma ; Humans ; Membrane Glycoproteins/genetics ; MicroRNAs/genetics ; Neoplasm Metastasis ; Neoplasm Proteins/genetics ; RNA, Circular/genetics ; RNA, Neoplasm/genetics

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Skocz do pozycji: 14.

Tytuł:: Silencing of PRDM5 increases cell proliferation and inhibits cell apoptosis in glioma.
Autorzy:: Wang X; Department of Neurosurgery, Taizhou People's Hospital, Taizhou, Jiangsu, China.
Chang H; Department of Neurosurgery, Taizhou People's Hospital, Taizhou, Jiangsu, China.
Gao G; Department of Neurosurgery, Taizhou People's Hospital, Taizhou, Jiangsu, China.
Su B; Department of Neurosurgery, Taizhou People's Hospital, Taizhou, Jiangsu, China.
Deng Q; Department of Neurosurgery, Taizhou People's Hospital, Taizhou, Jiangsu, China.
Zhou H; Department of Pathology, Taizhou People's Hospital, Taizhou, Jiangsu, China.
Wang Q; Department of Neurosurgery, Wuxi Second Hospital Affiliated to Nanjing Medical University, Wuxi, Jiangsu, China.
Lin Y; Department of Neurosurgery, Wuxi Second Hospital Affiliated to Nanjing Medical University, Wuxi, Jiangsu, China.
Ding Y; Department of Neurosurgery, Taizhou People's Hospital, Taizhou, Jiangsu, China.; Pokaż więcej
Źródło:: The International journal of neuroscience [Int J Neurosci] 2021 Feb; Vol. 131 (2), pp. 144-153. Date of Electronic Publication: 2020 Mar 05.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis*
Cell Proliferation*
Brain Neoplasms/*metabolism
DNA-Binding Proteins/*metabolism
Glioma/*metabolism
Transcription Factors/*metabolism
Cell Cycle Checkpoints ; Cells, Cultured ; Female ; Humans ; Male

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Skocz do pozycji: 15.

Tytuł:: Tim-3 expression in glioma cells is associated with drug resistance.
Autorzy:: Zhang J; Department of Neurosurgery, State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Zhu ZQ; Department of Neurosurgery, Tumor Hospital Affiliated of Xinjiang Medical University, Xinshi District, China.
Li YX; Department of Rehabilitation, The Second Affiliated Hospital of Xinjiang Medical University, Urumqi, China.
Zhuang QF; Department of Neurosurgery, State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Lai Y; Department of Urology, State Key Laboratory of Oncology in South China, Sun Yat-sen University Cancer Center, Collaborative Innovation Center for Cancer Medicine, Guangzhou, China.
Li SF; Department of Urology, State Key Laboratory of Oncology in South China, Sun Yat-sen University Cancer Center, Collaborative Innovation Center for Cancer Medicine, Guangzhou, China.
Xu XB; Department of Neurosurgery, Shunde Hospital, Southern Medical University (The First People's Hospital of Shunde Foshan), Foshan, China.
Liu JM; Department of Neurosurgery, The First Affiliated Hospital of Guangzhou University of Traditional Chinese Medicine, Guangzhou, China.; Pokaż więcej
Źródło:: Journal of cancer research and therapeutics [J Cancer Res Ther] 2019; Vol. 15 (4), pp. 882-888.
Typ publikacji:: Journal Article
MeSH Terms:: Drug Resistance, Neoplasm*
Apoptosis/*drug effects
Brain Neoplasms/*pathology
Gene Expression Regulation, Neoplastic/*drug effects
Glioma/*pathology
Hepatitis A Virus Cellular Receptor 2/*metabolism
Temozolomide/*pharmacology
Antineoplastic Agents, Alkylating/pharmacology ; Brain Neoplasms/drug therapy ; Brain Neoplasms/genetics ; Brain Neoplasms/metabolism ; Cell Proliferation/drug effects ; Glioma/drug therapy ; Glioma/genetics ; Glioma/metabolism ; Hepatitis A Virus Cellular Receptor 2/antagonists & inhibitors ; Hepatitis A Virus Cellular Receptor 2/genetics ; Humans ; RNA, Small Interfering/genetics ; Tumor Cells, Cultured

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Skocz do pozycji: 16.

Tytuł:: [Effects of FPR2 gene silencing on the proliferation, migration and invasion of human glioma U87 cells].
Autorzy:: Liu L; Deparment of Respiration, Wuhan Children's Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430015, China.
Li X; Department of Neurobiology, School of Basic Medicine, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China.
Shi J; Department of Neurobiology, School of Basic Medicine, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China.
Li L; Department of Pathology, The Central Hospital of Wuhan, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430014, China.
Wang J; Department of Medical Laboratory, the Central Hospital of Wuhan, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430014, China.
Luo ZZ; Department of Medical Laboratory, the Central Hospital of Wuhan, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430014, China.; Pokaż więcej
Źródło:: Zhonghua zhong liu za zhi [Chinese journal of oncology] [Zhonghua Zhong Liu Za Zhi] 2018 Sep 23; Vol. 40 (9), pp. 659-666.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis*
Cell Movement*
Cell Proliferation*
Brain Neoplasms/*pathology
Gene Silencing/*physiology
Glioma/*pathology
Receptors, Formyl Peptide/*metabolism
Receptors, Lipoxin/*metabolism
Animals ; Brain/metabolism ; Brain Neoplasms/genetics ; Cell Cycle ; Cell Line, Tumor ; Cyclin D1/metabolism ; Glioma/genetics ; Glioma/metabolism ; Humans ; Mice ; Neoplasm Invasiveness ; Neuroglia/metabolism ; RNA, Messenger/metabolism ; RNA, Small Interfering ; Real-Time Polymerase Chain Reaction ; Receptors, Formyl Peptide/genetics ; Receptors, Lipoxin/genetics ; Signal Transduction ; beta Catenin/metabolism

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Skocz do pozycji: 17.

Tytuł:: C 18 H 17 NO 6 and Its Combination with Scutellarin Suppress the Proliferation and Induce the Apoptosis of Human Glioma Cells via Upregulation of Fas-Associated Factor 1 Expression.
Autorzy:: He XY; Institute of Neurological Disease, Department of Anesthesiology, Translational Neuroscience Center, West China Hospital, Sichuan University, Chengdu, China.
Xiong LL; Department of Anesthesiology, Traditional Chinese Medicine Hospital of Southwest Medical University, Luzhou, China.
Xia QJ; Institute of Neurological Disease, Department of Anesthesiology, Translational Neuroscience Center, West China Hospital, Sichuan University, Chengdu, China.
Wang YY; Institute of Neurological Disease, Department of Anesthesiology, Translational Neuroscience Center, West China Hospital, Sichuan University, Chengdu, China.
Zhao XM; Department of Histology, Embryology and Neurobiology, West China School of Preclinical and Forensic Medicine, Sichuan University, Chengdu, China.
Du RL; Institute of Neurological Disease, Department of Anesthesiology, Translational Neuroscience Center, West China Hospital, Sichuan University, Chengdu, China.
Huang J; Institute of Neuroscience, Laboratory Zoology Department, Kunming Medical University, Kunming, China.
He XQ; School of Public Health, Kunming Medical University, Kunming, China.
Jia-Liu; Institute of Neuroscience, Laboratory Zoology Department, Kunming Medical University, Kunming, China.
Wang TH; Institute of Neurological Disease, Department of Anesthesiology, Translational Neuroscience Center, West China Hospital, Sichuan University, Chengdu, China.; Institute of Neuroscience, Laboratory Zoology Department, Kunming Medical University, Kunming, China.; Pokaż więcej
Źródło:: BioMed research international [Biomed Res Int] 2019 Feb 20; Vol. 2019, pp. 6821219. Date of Electronic Publication: 2019 Feb 20 (Print Publication: 2019).
Typ publikacji:: Journal Article
MeSH Terms:: Brain Neoplasms*/drug therapy
Brain Neoplasms*/metabolism
Brain Neoplasms*/pathology
Glioma*/drug therapy
Glioma*/metabolism
Glioma*/pathology
Adaptor Proteins, Signal Transducing/*biosynthesis
Apigenin/*pharmacology
Apoptosis/*drug effects
Cell Proliferation/*drug effects
Gene Expression Regulation, Neoplastic/*drug effects
Glucuronates/*pharmacology
Neoplasm Proteins/*biosynthesis
Apoptosis Regulatory Proteins ; Cell Line, Tumor ; Dose-Response Relationship, Drug ; Humans

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Skocz do pozycji: 18.

Tytuł:: β-Asarone Induces Apoptosis and Cell Cycle Arrest of Human Glioma U251 Cells via Suppression of HnRNP A2/B1-Mediated Pathway In Vitro and In Vivo.
Autorzy:: Li L; College of Pharmaceutical Sciences, Southwest University, Chongqing 400716, China. .
Yang Y; College of Pharmaceutical Sciences, Southwest University, Chongqing 400716, China. .
Wu M; College of Pharmaceutical Sciences, Southwest University, Chongqing 400716, China. zhangww_.
Yu Z; College of Pharmaceutical Sciences, Southwest University, Chongqing 400716, China. .
Wang C; College of Pharmaceutical Sciences, Southwest University, Chongqing 400716, China. .
Dou G; College of Pharmaceutical Sciences, Southwest University, Chongqing 400716, China. .
He H; College of Pharmaceutical Sciences, Southwest University, Chongqing 400716, China. .
Wang H; College of Pharmaceutical Sciences, Southwest University, Chongqing 400716, China. .
Yang N; Institute of Laboratory Animal Sciences, Sichuan Academy of Medical Sciences and Sichuan Provincial People's Hospital, Chengdu 610212, China. .
Qi H; College of Pharmaceutical Sciences, Southwest University, Chongqing 400716, China. .
Xu X; College of Pharmaceutical Sciences, Southwest University, Chongqing 400716, China. .; Pokaż więcej
Źródło:: Molecules (Basel, Switzerland) [Molecules] 2018 May 03; Vol. 23 (5). Date of Electronic Publication: 2018 May 03.
Typ publikacji:: Journal Article
MeSH Terms:: Anisoles/*pharmacology
Apoptosis/*drug effects
Brain Neoplasms/*metabolism
Cell Cycle Checkpoints/*drug effects
Glioma/*metabolism
Heterogeneous-Nuclear Ribonucleoprotein Group A-B/*metabolism
Signal Transduction/*drug effects
Allylbenzene Derivatives ; Animals ; Brain Neoplasms/genetics ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Disease Models, Animal ; Glioma/genetics ; Humans ; Mice ; Mitochondria/drug effects ; Mitochondria/metabolism ; Proto-Oncogene Proteins c-bcl-2/genetics ; Proto-Oncogene Proteins c-bcl-2/metabolism ; RNA Splicing ; Xenograft Model Antitumor Assays

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Skocz do pozycji: 19.

Tytuł:: Dp44mT, an iron chelator, suppresses growth and induces apoptosis via RORA-mediated NDRG2-IL6/JAK2/STAT3 signaling in glioma.
Autorzy:: Zhou J; Department of Neurosurgery, the First Hospital of China Medical University, NO.155, North Nanjing Street, Heping District, Shenyang, 110001, Liaoning Province, People's Republic of China.
Jiang Y; Department of Neurosurgery, the First Hospital of China Medical University, NO.155, North Nanjing Street, Heping District, Shenyang, 110001, Liaoning Province, People's Republic of China.; Department of Neurosurgery, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Zhao J; Department of Neurosurgery, the First Hospital of China Medical University, NO.155, North Nanjing Street, Heping District, Shenyang, 110001, Liaoning Province, People's Republic of China.
Zhang H; International Education College, Liaoning University of Traditional Chinese Medicine, Shenyang, Liaoning, China.
Fu J; Department of Neurosurgery, the First Hospital of China Medical University, NO.155, North Nanjing Street, Heping District, Shenyang, 110001, Liaoning Province, People's Republic of China.
Luo P; Department of Neurosurgery, the First Hospital of China Medical University, NO.155, North Nanjing Street, Heping District, Shenyang, 110001, Liaoning Province, People's Republic of China.
Ma Y; Department of Oncology, Liaoning Province Tumor Hospital, Shenyang, Liaoning, China.
Zou D; The First Laboratory of Cancer Institute, the First Hospital of China Medical University, Shenyang, Liaoning, China.
Gao H; College of Life and Health Sciences, Northeastern University, Shenyang, Liaoning, China.
Hu J; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Zhang Y; The First Laboratory of Cancer Institute, the First Hospital of China Medical University, Shenyang, Liaoning, China.
Jing Z; Department of Neurosurgery, the First Hospital of China Medical University, NO.155, North Nanjing Street, Heping District, Shenyang, 110001, Liaoning Province, People's Republic of China. .; Pokaż więcej
Źródło:: Cellular oncology (Dordrecht) [Cell Oncol (Dordr)] 2020 Jun; Vol. 43 (3), pp. 461-475. Date of Electronic Publication: 2020 Mar 23.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Glioma/*pathology
Iron Chelating Agents/*pharmacology
Janus Kinase 2/*metabolism
Nuclear Receptor Subfamily 1, Group F, Member 1/*metabolism
STAT3 Transcription Factor/*metabolism
Thiosemicarbazones/*pharmacology
Tumor Suppressor Proteins/*metabolism
Animals ; Carcinogenesis/drug effects ; Carcinogenesis/metabolism ; Carcinogenesis/pathology ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Female ; Humans ; Interleukin-6/metabolism ; Mice, Inbred BALB C ; Models, Biological ; Neoplastic Stem Cells/drug effects ; Neoplastic Stem Cells/metabolism ; Neoplastic Stem Cells/pathology ; Signal Transduction/drug effects ; Transcription, Genetic/drug effects ; Tumor Suppressor Proteins/genetics

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Skocz do pozycji: 20.

Tytuł:: Effect of CCL2 siRNA on proliferation and apoptosis in the U251 human glioma cell line.
Autorzy:: Lu B; Department of Neurosurgery, Huzhou Central Hospital, Huzhou, Zhejiang 313000, P.R. China.
Zhou Y; Department of Neurosurgery, Huzhou Central Hospital, Huzhou, Zhejiang 313000, P.R. China.
Su Z; Department of Neurosurgery, Huzhou Central Hospital, Huzhou, Zhejiang 313000, P.R. China.
Yan A; Department of Neurosurgery, Huzhou Central Hospital, Huzhou, Zhejiang 313000, P.R. China.
Ding P; Department of Neurosurgery, The First Affiliated Hospital of Kunming Medical College, Kunming, Yunnan 650032, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2017 Sep; Vol. 16 (3), pp. 3387-3394. Date of Electronic Publication: 2017 Jul 15.
Typ publikacji:: Journal Article; Retracted Publication
MeSH Terms:: Apoptosis*/genetics
Brain Neoplasms/*metabolism
Brain Neoplasms/*pathology
Chemokine CCL2/*metabolism
Glioma/*metabolism
Glioma/*pathology
RNA, Small Interfering/*metabolism
Brain Neoplasms/enzymology ; Brain Neoplasms/genetics ; Cell Cycle Checkpoints/genetics ; Cell Line, Tumor ; Chemokine CCL2/genetics ; Down-Regulation ; Extracellular Signal-Regulated MAP Kinases/metabolism ; Female ; Gene Expression Regulation, Neoplastic ; Glioma/enzymology ; Glioma/genetics ; Humans ; MAP Kinase Signaling System ; Male ; Middle Aged ; Receptors, CCR2/metabolism

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