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Tytuł:
Cell origin and niche availability dictate the capacity of peritoneal macrophages to colonize the cavity and omentum.
Autorzy:
Louwe PA; Queens Medical Research Institute, University of Edinburgh Centre for Inflammation Research, Edinburgh, UK.; Laboratory of Myeloid Cell Biology in Tissue Damage and Inflammation, VIB Center for Inflammation Research, Ghent, Belgium.
Forbes SJ; Centre for Regenerative Medicine, University of Edinburgh, Edinburgh, UK.
Bénézech C; Queens Medical Research Institute, University of Edinburgh Centre for Cardiovascular Science, Edinburgh, UK.
Pridans C; Queens Medical Research Institute, University of Edinburgh Centre for Inflammation Research, Edinburgh, UK.; Simons Initiative for the Developing Brain, Centre for Discovery Brain Sciences, University of Edinburgh, Edinburgh, UK.
Jenkins SJ; Queens Medical Research Institute, University of Edinburgh Centre for Inflammation Research, Edinburgh, UK.
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Źródło:
Immunology [Immunology] 2022 Aug; Vol. 166 (4), pp. 458-474. Date of Electronic Publication: 2022 May 10.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Macrophages, Peritoneal*
Omentum*/metabolism
Macrophages ; Peritoneal Cavity
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CD5L attenuates allergic airway inflammation by expanding CD11c alveolar macrophages and inhibiting NLRP3 inflammasome activation via HDAC2.
Autorzy:
Weng D; Key Laboratory of Laboratory Medical Diagnostics Designated by the Ministry of Education, School of Laboratory Medicine, Chongqing Medical University, Chongqing, China.
Gao S; Department of Laboratory Medicine, Affiliated Hospital of Zunyi Medical University, School of Laboratory Medicine, Zunyi Medical University, Zunyi, Guizhou, China.
Shen H; Department of Laboratory Medicine, the First Affiliated Hospital, Chongqing Medical University, Chongqing, China.
Yao S; Key Laboratory of Laboratory Medical Diagnostics Designated by the Ministry of Education, School of Laboratory Medicine, Chongqing Medical University, Chongqing, China.
Huang Q; Key Laboratory of Laboratory Medical Diagnostics Designated by the Ministry of Education, School of Laboratory Medicine, Chongqing Medical University, Chongqing, China.
Zhang Y; Key Laboratory of Laboratory Medical Diagnostics Designated by the Ministry of Education, School of Laboratory Medicine, Chongqing Medical University, Chongqing, China.
Huang W; Key Laboratory of Laboratory Medical Diagnostics Designated by the Ministry of Education, School of Laboratory Medicine, Chongqing Medical University, Chongqing, China.
Wang Y; Key Laboratory of Laboratory Medical Diagnostics Designated by the Ministry of Education, School of Laboratory Medicine, Chongqing Medical University, Chongqing, China.
Zhang X; Key Laboratory of Laboratory Medical Diagnostics Designated by the Ministry of Education, School of Laboratory Medicine, Chongqing Medical University, Chongqing, China.
Yin Y; Key Laboratory of Laboratory Medical Diagnostics Designated by the Ministry of Education, School of Laboratory Medicine, Chongqing Medical University, Chongqing, China.
Xu W; Key Laboratory of Laboratory Medical Diagnostics Designated by the Ministry of Education, School of Laboratory Medicine, Chongqing Medical University, Chongqing, China.
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Źródło:
Immunology [Immunology] 2022 Nov; Vol. 167 (3), pp. 384-397. Date of Electronic Publication: 2022 Jul 26.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Asthma*
Macrophages, Alveolar*
Animals ; Apoptosis Regulatory Proteins/metabolism ; Bronchoalveolar Lavage Fluid ; CD11c Antigen/metabolism ; Disease Models, Animal ; Histone Deacetylase 2 ; Inflammasomes/metabolism ; Inflammation ; Lung ; Mice ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Ovalbumin ; Receptors, Scavenger/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
T-cell immunoglobulin- and mucin-domain-containing molecule-4 maintains adipose tissue homeostasis by orchestrating M2 macrophage polarization via nuclear factor kappa B pathway.
Autorzy:
Ding L; Key Laboratory for Experimental Teratology of Ministry of Education, Shandong Key Laboratory of Infection and Immunity, and Department of Immunology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, People's Republic of China.
Liang Y; Key Laboratory for Experimental Teratology of Ministry of Education, Shandong Key Laboratory of Infection and Immunity, and Department of Immunology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, People's Republic of China.; Department of Emergency Medicine, Qilu Hospital of Shandong University, Jinan, Shandong, People's Republic of China.
Wang Y; Key Laboratory for Experimental Teratology of Ministry of Education, Shandong Key Laboratory of Infection and Immunity, and Department of Immunology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, People's Republic of China.
Tong Z; Key Laboratory for Experimental Teratology of Ministry of Education, Shandong Key Laboratory of Infection and Immunity, and Department of Immunology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, People's Republic of China.
Liu W; Key Laboratory for Experimental Teratology of Ministry of Education, Shandong Key Laboratory of Infection and Immunity, and Department of Immunology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, People's Republic of China.
Tan S; Key Laboratory for Experimental Teratology of Ministry of Education, Shandong Key Laboratory of Infection and Immunity, and Department of Immunology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, People's Republic of China.
Zhang J; Key Laboratory for Experimental Teratology of Ministry of Education, Shandong Key Laboratory of Infection and Immunity, and Department of Immunology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, People's Republic of China.
Wang Y; Key Laboratory for Experimental Teratology of Ministry of Education, Shandong Key Laboratory of Infection and Immunity, and Department of Immunology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, People's Republic of China.
Wu Z; Key Laboratory for Experimental Teratology of Ministry of Education, Shandong Key Laboratory of Infection and Immunity, and Department of Immunology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, People's Republic of China.
Liang X; Key Laboratory for Experimental Teratology of Ministry of Education, Shandong Key Laboratory of Infection and Immunity, and Department of Immunology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, People's Republic of China.
Ma C; Key Laboratory for Experimental Teratology of Ministry of Education, Shandong Key Laboratory of Infection and Immunity, and Department of Immunology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, People's Republic of China.
Gao L; Key Laboratory for Experimental Teratology of Ministry of Education, Shandong Key Laboratory of Infection and Immunity, and Department of Immunology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, People's Republic of China.
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Źródło:
Immunology [Immunology] 2023 Jan; Vol. 168 (1), pp. 49-62. Date of Electronic Publication: 2022 Aug 22.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adipose Tissue*
Macrophages*/metabolism
Membrane Proteins*
Animals ; Mice ; Homeostasis ; Immunoglobulins/metabolism ; Inflammation/metabolism ; Mice, Inbred C57BL ; Mice, Knockout ; NF-kappa B/metabolism ; Obesity/metabolism ; T-Lymphocytes/metabolism ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Macrophage metabolism in the intestine is compartment specific and regulated by the microbiota.
Autorzy:
Scott NA; Division of Infection, Immunity and Respiratory Medicine, Faculty of Biology, Medicine and Health, School of Biological Sciences, University of Manchester, Manchester Academic Health Science Centre, Lydia Becker Institute of Immunology and Inflammation, Manchester, UK.
Lawson MAE; Division of Infection, Immunity and Respiratory Medicine, Faculty of Biology, Medicine and Health, School of Biological Sciences, University of Manchester, Manchester Academic Health Science Centre, Lydia Becker Institute of Immunology and Inflammation, Manchester, UK.; Gut Microbes & Health, Quadram Institute Bioscience, Norwich Research Park, Norwich, UK.
Hodgetts RJ; Division of Infection, Immunity and Respiratory Medicine, Faculty of Biology, Medicine and Health, School of Biological Sciences, University of Manchester, Manchester Academic Health Science Centre, Lydia Becker Institute of Immunology and Inflammation, Manchester, UK.; Department of Immunology, Weizmann Institute of Science, Rehovot, Israel.
Le Gall G; Norwich Medical School, University of East Anglia, Norwich Research Park, Norwich, UK.
Hall LJ; Gut Microbes & Health, Quadram Institute Bioscience, Norwich Research Park, Norwich, UK.; ZIEL - Institute for Food & Health, Technical University of Munich, Freising, Germany.
Mann ER; Division of Infection, Immunity and Respiratory Medicine, Faculty of Biology, Medicine and Health, School of Biological Sciences, University of Manchester, Manchester Academic Health Science Centre, Lydia Becker Institute of Immunology and Inflammation, Manchester, UK.
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Źródło:
Immunology [Immunology] 2022 May; Vol. 166 (1), pp. 138-152. Date of Electronic Publication: 2022 Mar 11.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gastrointestinal Microbiome*
Macrophages*/metabolism
Animals ; Anti-Bacterial Agents/pharmacology ; Colon ; Fatty Acids/metabolism ; Mammals ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
IL-20 is involved in obesity by modulation of adipogenesis and macrophage dysregulation.
Autorzy:
Hsu YH; Institute of Clinical Medicine, College of Medicine, National Cheng Kung University, Tainan, Taiwan.; Research Center of Clinical Medicine, National Cheng Kung University Hospital, College of Medicine, National Cheng Kung University, Tainan, Taiwan.
Wu CH; Department of Family Medicine, National Cheng Kung University Hospital, College of Medicine, National Cheng Kung University, Tainan, Taiwan.
Chiu CJ; Graduate Institute of Clinical Medicine, College of Medicine, National Taiwan University, Taipei, Taiwan.
Chen WT; Department of Biochemistry and Molecular Biology, College of Medicine, National Cheng Kung University, Tainan, Taiwan.
Chang YC; Department of Biochemistry and Molecular Biology, College of Medicine, National Cheng Kung University, Tainan, Taiwan.
Wabitsch M; Division of Pediatric Endocrinology and Diabetes, Department of Pediatrics and Adolescent Medicine, Ulm University Medical Center, Ulm, Germany.
Chang MS; Department of Biochemistry and Molecular Biology, College of Medicine, National Cheng Kung University, Tainan, Taiwan.
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Źródło:
Immunology [Immunology] 2021 Dec; Vol. 164 (4), pp. 817-833. Date of Electronic Publication: 2021 Aug 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adipogenesis*/genetics
Disease Susceptibility*
Interleukins/*metabolism
Macrophages/*metabolism
Obesity/*etiology
Obesity/*metabolism
Adipocytes/metabolism ; Adult ; Aged ; Aged, 80 and over ; Animals ; Biomarkers ; Chemotaxis, Leukocyte ; Cytokines/metabolism ; Disease Models, Animal ; Female ; Gene Expression ; Glucose/metabolism ; Humans ; Insulin/metabolism ; Interleukins/genetics ; Macrophages/immunology ; Male ; Mice ; Middle Aged ; Obesity/pathology ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
The IL-23/IL-17 axis promotes the formation of retinal neovascularization by activating the NLRP3 inflammasome in macrophages in an experimental retinopathy mouse model.
Autorzy:
Sui A; The Department of Ophthalmology, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Chen X; The Department of Ophthalmology, Zhongshan Hospital, Fudan University, Shanghai, China.
Yao Y; The Department of Ophthalmology, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Yao Y; The Department of Ophthalmology, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Shen X; The Department of Ophthalmology, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Zhu Y; The Department of Ophthalmology, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Xie B; The Department of Ophthalmology, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
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Źródło:
Immunology [Immunology] 2021 Dec; Vol. 164 (4), pp. 803-816. Date of Electronic Publication: 2021 Aug 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Diabetic Retinopathy/*etiology
Diabetic Retinopathy/*metabolism
Inflammasomes/*metabolism
Interleukin-17/*metabolism
Interleukin-23/*metabolism
Macrophages/*metabolism
NLR Family, Pyrin Domain-Containing 3 Protein/*metabolism
Retinal Neovascularization/*etiology
Retinal Neovascularization/*metabolism
Animals ; Biomarkers ; Diabetic Retinopathy/pathology ; Disease Models, Animal ; Disease Susceptibility ; Immunohistochemistry ; Macrophage Activation/genetics ; Macrophage Activation/immunology ; Macrophages/immunology ; Mice ; Retinal Neovascularization/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
PAQR11 modulates monocyte-to-macrophage differentiation and pathogenesis of rheumatoid arthritis.
Autorzy:
Lin Y; CAS Key Laboratory of Nutrition, Metabolism and Food Safety, Shanghai Institute of Nutrition and Health, University of Chinese Academy of Sciences, Chinese Academy of Sciences, Shanghai, China.
Huang M; CAS Key Laboratory of Nutrition, Metabolism and Food Safety, Shanghai Institute of Nutrition and Health, University of Chinese Academy of Sciences, Chinese Academy of Sciences, Shanghai, China.
Wang S; CAS Key Laboratory of Nutrition, Metabolism and Food Safety, Shanghai Institute of Nutrition and Health, University of Chinese Academy of Sciences, Chinese Academy of Sciences, Shanghai, China.
You X; CAS Key Laboratory of Nutrition, Metabolism and Food Safety, Shanghai Institute of Nutrition and Health, University of Chinese Academy of Sciences, Chinese Academy of Sciences, Shanghai, China.
Zhang L; CAS Key Laboratory of Nutrition, Metabolism and Food Safety, Shanghai Institute of Nutrition and Health, University of Chinese Academy of Sciences, Chinese Academy of Sciences, Shanghai, China.
Chen Y; CAS Key Laboratory of Nutrition, Metabolism and Food Safety, Shanghai Institute of Nutrition and Health, University of Chinese Academy of Sciences, Chinese Academy of Sciences, Shanghai, China.
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Źródło:
Immunology [Immunology] 2021 May; Vol. 163 (1), pp. 60-73. Date of Electronic Publication: 2021 Jan 24.
Typ publikacji:
Journal Article
MeSH Terms:
Cell Differentiation*
Arthritis, Experimental/*metabolism
Arthritis, Rheumatoid/*metabolism
Intracellular Signaling Peptides and Proteins/*metabolism
Macrophages/*metabolism
Membrane Proteins/*metabolism
Monocytes/*metabolism
Animals ; Apoptosis ; Arthritis, Experimental/genetics ; Arthritis, Experimental/immunology ; Arthritis, Experimental/pathology ; Arthritis, Rheumatoid/genetics ; Arthritis, Rheumatoid/immunology ; Arthritis, Rheumatoid/pathology ; CCAAT-Enhancer-Binding Protein-beta/metabolism ; Disease Progression ; Gene Expression Regulation ; HEK293 Cells ; Humans ; Intracellular Signaling Peptides and Proteins/genetics ; Macrophages/immunology ; Macrophages/pathology ; Membrane Proteins/genetics ; Mice ; Mitogen-Activated Protein Kinases/metabolism ; Monocytes/immunology ; Monocytes/pathology ; Phosphorylation ; Signal Transduction ; THP-1 Cells
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Schistosome and intestinal helminth modulation of macrophage immunometabolism.
Autorzy:
Cortes-Selva D; Division of Microbiology and Immunology, Department of Pathology, University of Utah, Salt Lake City, UT, USA.; Janssen Biotherapeutics, Janssen R&D, Spring House, PA, USA.
Fairfax K; Division of Microbiology and Immunology, Department of Pathology, University of Utah, Salt Lake City, UT, USA.
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Źródło:
Immunology [Immunology] 2021 Feb; Vol. 162 (2), pp. 123-134. Date of Electronic Publication: 2020 Jul 27.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Helminthiasis/*immunology
Helminths/*immunology
Intestines/*immunology
Intestines/*parasitology
Macrophages/*immunology
Schistosoma/*immunology
Schistosomiasis/*immunology
Animals ; Cytokines/immunology ; Humans ; Macrophages/parasitology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Ectopic endometriotic stromal cells-derived lactate induces M2 macrophage polarization via Mettl3/Trib1/ERK/STAT3 signalling pathway in endometriosis.
Autorzy:
Gou Y; Department of Obstetrics and Gynecology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
Wang H; Department of Obstetrics and Gynecology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
Wang T; Department of Obstetrics and Gynecology, Harbin Red Cross Central Hospital, Harbin, China.
Wang H; Department of Obstetrics and Gynecology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
Wang B; Department of Obstetrics and Gynecology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
Jiao N; Department of Obstetrics and Gynecology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
Yu Y; Department of Obstetrics and Gynecology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
Cao Y; Department of Obstetrics and Gynecology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
Wang H; Department of Obstetrics and Gynecology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
Zhang Z; Department of Obstetrics and Gynecology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
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Źródło:
Immunology [Immunology] 2023 Mar; Vol. 168 (3), pp. 389-402. Date of Electronic Publication: 2022 Oct 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Lactic Acid*
Endometriosis*/metabolism
Endometriosis*/pathology
Humans ; Female ; Animals ; Mice ; Signal Transduction ; Macrophages/metabolism ; Stromal Cells ; Methyltransferases/metabolism ; Protein Serine-Threonine Kinases/metabolism ; Intracellular Signaling Peptides and Proteins/metabolism ; STAT3 Transcription Factor/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Ataxia telangiectasia mutated kinase inhibition promotes irradiation-induced PD-L1 expression in tumour-associated macrophages through IFN-I/JAK signalling pathway.
Autorzy:
Gao Y; Department of Radiation and Medical Oncology, Zhongnan Hospital of Wuhan University, Wuhan, China.
Li Y; Department of Radiation and Medical Oncology, Zhongnan Hospital of Wuhan University, Wuhan, China.
Lin Z; Department of Gastric and Colorectal Surgical Oncology, Zhongnan Hospital of Wuhan University, Wuhan, China.
Zeng Y; Department of Radiation and Medical Oncology, Zhongnan Hospital of Wuhan University, Wuhan, China.
Huang Z; Department of Radiation and Medical Oncology, Zhongnan Hospital of Wuhan University, Wuhan, China.; Department of Biological Repositories, Zhongnan Hospital of Wuhan University, Wuhan, China.
Han L; Department of Radiation and Medical Oncology, Zhongnan Hospital of Wuhan University, Wuhan, China.
Zhong Y; Department of Radiation and Medical Oncology, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Key Laboratory of Tumor Biological Behaviors, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Cancer Clinical Study Center, Zhongnan Hospital of Wuhan University, Wuhan, China.
Gong Y; Department of Biological Repositories, Zhongnan Hospital of Wuhan University, Wuhan, China.
Wu Q; Department of Radiation and Medical Oncology, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Key Laboratory of Tumor Biological Behaviors, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Cancer Clinical Study Center, Zhongnan Hospital of Wuhan University, Wuhan, China.
Xie C; Department of Radiation and Medical Oncology, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Key Laboratory of Tumor Biological Behaviors, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Cancer Clinical Study Center, Zhongnan Hospital of Wuhan University, Wuhan, China.
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Źródło:
Immunology [Immunology] 2023 Feb; Vol. 168 (2), pp. 346-361. Date of Electronic Publication: 2022 Nov 16.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Ataxia Telangiectasia*
Neoplasms*
Humans ; Animals ; Mice ; Interferons ; Tumor-Associated Macrophages ; B7-H1 Antigen/metabolism ; Signal Transduction ; Tumor Microenvironment
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CD5L attenuates allergic airway inflammation by expanding CD11c alveolar macrophages and inhibiting NLRP3 inflammasome activation via HDAC2.
Autorzy:
Weng, Danlin (AUTHOR)
Gao, Song (AUTHOR)
Shen, Hailan (AUTHOR)
Yao, Shifei (AUTHOR)
Huang, Qi (AUTHOR)
Zhang, Yanyu (AUTHOR)
Huang, Wenjie (AUTHOR)
Wang, Yan (AUTHOR)
Zhang, Xuemei (AUTHOR)
Yin, Yibing (AUTHOR)
Xu, Wenchun (AUTHOR)
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Źródło:
Immunology. Nov2022, Vol. 167 Issue 3, p384-397. 14p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Pneumolysin suppresses the initial macrophage pro‐inflammatory response to Streptococcus pneumoniae.
Autorzy:
Periselneris, Jimstan (AUTHOR)
Turner, Carolin T. (AUTHOR)
Ercoli, Giuseppe (AUTHOR)
Szylar, Gabriella (AUTHOR)
Weight, Caroline M. (AUTHOR)
Thurston, Teresa (AUTHOR)
Whelan, Matthew (AUTHOR)
Tomlinson, Gillian (AUTHOR)
Noursadeghi, Mahdad (AUTHOR)
Brown, Jeremy (AUTHOR)
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Źródło:
Immunology. Nov2022, Vol. 167 Issue 3, p413-427. 15p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Tissue-resident innate immunity in the lung.
Autorzy:
Ardain A; Africa Health Research Institute, KwaZulu-Natal, South Africa.; College of Health Sciences, University of KwaZulu-Natal, Durban, South Africa.
Marakalala MJ; Africa Health Research Institute, KwaZulu-Natal, South Africa.; Department of Infection and Immunity, University College London, London, UK.
Leslie A; Africa Health Research Institute, KwaZulu-Natal, South Africa.; Department of Infection and Immunity, University College London, London, UK.
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Źródło:
Immunology [Immunology] 2020 Mar; Vol. 159 (3), pp. 245-256. Date of Electronic Publication: 2019 Nov 21.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Immunity, Innate*
Dendritic Cells/*immunology
Lung/*immunology
Lung Diseases/*immunology
Lymphocytes/*immunology
Macrophages/*immunology
Animals ; Cellular Microenvironment ; Dendritic Cells/metabolism ; Dendritic Cells/pathology ; Humans ; Killer Cells, Natural/immunology ; Lung/metabolism ; Lung/pathology ; Lung Diseases/metabolism ; Lung Diseases/pathology ; Lymphocytes/metabolism ; Lymphocytes/pathology ; Macrophages/metabolism ; Macrophages/pathology ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
TRIM21 controls Toll-like receptor 2 responses in bone-marrow-derived macrophages.
Autorzy:
Sjöstrand M; Unit of Rheumatology, Center for Molecular Medicine L8:03, Department of Medicine, Karolinska Institutet, Karolinska University Hospital, Stockholm, Sweden.
Carow B; Department of Microbiology, Tumor and Cell Biology, Karolinska Institutet, Stockholm, Sweden.
Nyberg WA; Unit of Rheumatology, Center for Molecular Medicine L8:03, Department of Medicine, Karolinska Institutet, Karolinska University Hospital, Stockholm, Sweden.
Covacu R; Unit of Neuroimmunology, Department of Clinical Neuroscience, Center for Molecular Medicine, Karolinska Institutet, Karolinska University Hospital, Stockholm, Sweden.
Rottenberg ME; Department of Microbiology, Tumor and Cell Biology, Karolinska Institutet, Stockholm, Sweden.
Espinosa A; Unit of Rheumatology, Center for Molecular Medicine L8:03, Department of Medicine, Karolinska Institutet, Karolinska University Hospital, Stockholm, Sweden.
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Źródło:
Immunology [Immunology] 2020 Mar; Vol. 159 (3), pp. 335-343. Date of Electronic Publication: 2019 Dec 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cytokines/*metabolism
Inflammation Mediators/*metabolism
Macrophages/*metabolism
Ribonucleoproteins/*metabolism
Toll-Like Receptor 2/*metabolism
Animals ; Cell Differentiation ; Cells, Cultured ; Host-Pathogen Interactions ; Lipopeptides/pharmacology ; Macrophage Colony-Stimulating Factor/pharmacology ; Macrophages/drug effects ; Macrophages/immunology ; Macrophages/microbiology ; Mice, Inbred C57BL ; Mice, Knockout ; Mycobacterium bovis/immunology ; Mycobacterium bovis/pathogenicity ; Phenotype ; Ribonucleoproteins/deficiency ; Ribonucleoproteins/genetics ; Signal Transduction ; Toll-Like Receptor 2/agonists ; Toll-Like Receptor 2/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Monocyte isolation techniques significantly impact the phenotype of both isolated monocytes and derived macrophages in vitro.
Autorzy:
Nielsen MC; Department of Clinical Biochemistry, Aarhus University Hospital, Aarhus, Denmark.
Andersen MN; Department of Clinical Biochemistry, Aarhus University Hospital, Aarhus, Denmark.; Department of Biomedicine, Aarhus University, Aarhus, Denmark.
Møller HJ; Department of Clinical Biochemistry, Aarhus University Hospital, Aarhus, Denmark.
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Źródło:
Immunology [Immunology] 2020 Jan; Vol. 159 (1), pp. 63-74. Date of Electronic Publication: 2019 Nov 27.
Typ publikacji:
Comparative Study; Journal Article
MeSH Terms:
Cell Differentiation*
Flow Cytometry*
Cell Separation/*methods
Lipopolysaccharide Receptors/*metabolism
Macrophages/*physiology
Monocytes/*physiology
Antigens, CD/metabolism ; Antigens, Differentiation, Myelomonocytic/metabolism ; Biomarkers ; Cell Adhesion ; Cells, Cultured ; Humans ; Interleukin-6/metabolism ; Lectins, C-Type/metabolism ; Macrophages/immunology ; Macrophages/metabolism ; Mannose Receptor ; Mannose-Binding Lectins/metabolism ; Monocytes/immunology ; Monocytes/metabolism ; Phenotype ; Receptors, Cell Surface/metabolism ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The immunosuppressive functions of two novel tick serpins, HlSerpin-a and HlSerpin-b, from Haemaphysalis longicornis.
Autorzy:
Wang F; Institutes of Biology and Medical Sciences, Jiangsu Key Laboratory of Infection and Immunity, Soochow University, Suzhou, China.
Song Z; Institutes of Biology and Medical Sciences, Jiangsu Key Laboratory of Infection and Immunity, Soochow University, Suzhou, China.
Chen J; NHC Key Lab of Reproduction Regulation (Shanghai Institute of Planned Parenthood Research), Fudan University, Shanghai, China.
Wu Q; NHC Key Lab of Reproduction Regulation (Shanghai Institute of Planned Parenthood Research), Fudan University, Shanghai, China.
Zhou X; School of Biology and Basic Medical Sciences, Soochow University, Suzhou, China.
Ni X; NHC Key Lab of Reproduction Regulation (Shanghai Institute of Planned Parenthood Research), Fudan University, Shanghai, China.
Dai J; Institutes of Biology and Medical Sciences, Jiangsu Key Laboratory of Infection and Immunity, Soochow University, Suzhou, China.
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Źródło:
Immunology [Immunology] 2020 Jan; Vol. 159 (1), pp. 109-120. Date of Electronic Publication: 2019 Nov 10.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Arthritis, Experimental/*prevention & control
Arthropod Proteins/*pharmacology
Dendritic Cells/*drug effects
Immunosuppressive Agents/*pharmacology
Ixodidae/*metabolism
Joints/*drug effects
Macrophages/*drug effects
Serpins/*pharmacology
Animals ; Arthritis, Experimental/immunology ; Arthritis, Experimental/metabolism ; Arthritis, Experimental/pathology ; Arthropod Proteins/genetics ; Arthropod Proteins/isolation & purification ; Cytokines/metabolism ; Dendritic Cells/immunology ; Dendritic Cells/metabolism ; Immunosuppressive Agents/isolation & purification ; Ixodidae/genetics ; Joints/immunology ; Joints/metabolism ; Joints/pathology ; Macrophages/immunology ; Macrophages/metabolism ; Male ; Mice ; Mice, Inbred DBA ; Protein Conformation ; RAW 264.7 Cells ; Saliva/metabolism ; Serpins/genetics ; Serpins/isolation & purification ; Structure-Activity Relationship
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Histone hyperacetylation mediates enhanced IL-1β production in LPS/IFN-γ-stimulated macrophages.
Autorzy:
Dong Z; Department of Cardiology, Zhongshan Hospital, Fudan University, Shanghai Institute of Cardiovascular Diseases, Shanghai, China.
Li R; Department of Cardiology, Huashan Hospital, Fudan University, Shanghai, China.
Xu L; Department of Cardiology, Zhongshan Hospital, Fudan University, Shanghai Institute of Cardiovascular Diseases, Shanghai, China.
Xin K; Department of Cardiology, Zhongshan Hospital, Fudan University, Shanghai Institute of Cardiovascular Diseases, Shanghai, China.
Xu Y; Department of Cardiology, Zhongshan Hospital, Fudan University, Shanghai Institute of Cardiovascular Diseases, Shanghai, China.
Shi H; Department of Cardiology, Huashan Hospital, Fudan University, Shanghai, China.
Sun A; Department of Cardiology, Zhongshan Hospital, Fudan University, Shanghai Institute of Cardiovascular Diseases, Shanghai, China.; Institute of Biomedical Science, Fudan University, Shanghai, China.
Ge J; Department of Cardiology, Zhongshan Hospital, Fudan University, Shanghai Institute of Cardiovascular Diseases, Shanghai, China.; Institute of Biomedical Science, Fudan University, Shanghai, China.
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Źródło:
Immunology [Immunology] 2020 Jun; Vol. 160 (2), pp. 183-197. Date of Electronic Publication: 2020 Apr 07.
Typ publikacji:
Journal Article
MeSH Terms:
Macrophage Activation*
Histones/*metabolism
Interleukin-1beta/*metabolism
Macrophages/*immunology
Acetylation ; Animals ; Cells, Cultured ; Glycolysis/immunology ; Interferon-gamma/immunology ; Lipopolysaccharides/immunology ; Macrophages/metabolism ; Mice ; Primary Cell Culture ; Recombinant Proteins/immunology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Elucidation of the signalling pathways for enhanced exosome release from Mycobacterium-infected macrophages and subsequent induction of differentiation.
Autorzy:
Singh A; School of Biological Sciences, Indian Association for the Cultivation of Science, Kolkata, India.
Das K; School of Biological Sciences, Indian Association for the Cultivation of Science, Kolkata, India.
Banerjee S; School of Biological Sciences, Indian Association for the Cultivation of Science, Kolkata, India.
Sen P; School of Biological Sciences, Indian Association for the Cultivation of Science, Kolkata, India.
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Źródło:
Immunology [Immunology] 2023 Jan; Vol. 168 (1), pp. 63-82. Date of Electronic Publication: 2022 Oct 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Exosomes*/metabolism
Mycobacterium tuberculosis*
Proto-Oncogene Proteins c-akt/metabolism ; Macrophages/metabolism ; Cell Differentiation ; Endosomal Sorting Complexes Required for Transport/genetics ; Endosomal Sorting Complexes Required for Transport/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Wyświetlanie 1-20 z 4975

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