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Wyświetlanie 1-20 z 155
Tytuł:
NOX4-derived ROS-induced overexpression of FOXM1 regulates aerobic glycolysis in glioblastoma.
Autorzy:
Su X; Department of Neurosurgery, Shandong Provincial Hospital, Cheeloo College of Medicine, Shandong University, Jinan, 250012, Shandong, China.
Yang Y; Department of Neurosurgery, Shandong Provincial Hospital, Cheeloo College of Medicine, Shandong University, Jinan, 250012, Shandong, China.; Department of Neurosurgery, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan, 250012, Shandong, China.
Yang Q; Department of Radiation Oncology, The First Affiliated Hospital of Guangxi Medical University, Nanning, 530022, Guangxi, China.
Pang B; Department of Neurosurgery, Qilu Hospital, Cheeloo College of Medicine, Shandong University, Jinan, 250012, Shandong, China.
Sun S; Department of Neurosurgery, Shandong Provincial Hospital, Cheeloo College of Medicine, Shandong University, Jinan, 250012, Shandong, China.
Wang Y; Department of Neurosurgery, Shandong Provincial Hospital, Cheeloo College of Medicine, Shandong University, Jinan, 250012, Shandong, China.; Department of Neurosurgery, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan, 250012, Shandong, China.
Qiao Q; Department of Neurosurgery, Shandong Provincial Hospital, Cheeloo College of Medicine, Shandong University, Jinan, 250012, Shandong, China.
Guo C; Department of Neurosurgery, Shandong Provincial Hospital, Cheeloo College of Medicine, Shandong University, Jinan, 250012, Shandong, China.
Liu H; Department of Neurosurgery, Linyi People's Hospital, Cheeloo College of Medicine, Shandong University, Linyi, 276003, Shandong, China. liu_.
Pang Q; Department of Neurosurgery, Shandong Provincial Hospital, Cheeloo College of Medicine, Shandong University, Jinan, 250012, Shandong, China. .
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Źródło:
BMC cancer [BMC Cancer] 2021 Nov 05; Vol. 21 (1), pp. 1181. Date of Electronic Publication: 2021 Nov 05.
Typ publikacji:
Journal Article
MeSH Terms:
Warburg Effect, Oncologic*
Brain Neoplasms/*metabolism
Forkhead Box Protein M1/*metabolism
Glioblastoma/*metabolism
NADPH Oxidase 4/*metabolism
Reactive Oxygen Species/*metabolism
Adenosine Triphosphate/metabolism ; Animals ; Blotting, Western ; Brain/metabolism ; Brain Neoplasms/therapy ; Cell Line, Tumor ; Forkhead Box Protein M1/antagonists & inhibitors ; Glioblastoma/therapy ; Humans ; Hypoxia-Inducible Factor 1, alpha Subunit/metabolism ; Lactic Acid/biosynthesis ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Mitochondria/metabolism ; NADPH Oxidase 4/antagonists & inhibitors ; Neoplasm Proteins/metabolism ; Neoplasm Transplantation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Role of NADPH Oxidase 4 in Corneal Endothelial Cells Is Mediated by Endoplasmic Reticulum Stress and Autophagy
Autorzy:
Dae Joong Ma
Jin Sun Hwang
Kyung Bo Noh
Sun-Hee Oh
Kyoung Wook Kim
Young Joo Shin
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Temat:
NOX4
corneal-endothelial cells
autophagy
reactive oxygen species
senescence
in vivo transfection
Therapeutics. Pharmacology
RM1-950
Źródło:
Antioxidants, Vol 12, Iss 6, p 1228 (2023)
Opis pliku:
electronic resource
Relacje:
https://www.mdpi.com/2076-3921/12/6/1228; https://doaj.org/toc/2076-3921
Dostęp URL:
https://doaj.org/article/301f10275e1947bebd937e88d560cb5f  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Tytuł:
NADPH Oxidase 4: A Potential Therapeutic Target of Malignancy
Autorzy:
Shulei Gong
Shiyang Wang
Mingrui Shao
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Temat:
reactive oxygen species
tumor microenevironment
NOX4
cancer
metabolism
Biology (General)
QH301-705.5
Źródło:
Frontiers in Cell and Developmental Biology, Vol 10 (2022)
Opis pliku:
electronic resource
Relacje:
https://www.frontiersin.org/articles/10.3389/fcell.2022.884412/full; https://doaj.org/toc/2296-634X
Dostęp URL:
https://doaj.org/article/f878bb4c61a140809e9a1b219b88a49d  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Tytuł:
APX-115A, a pan-NADPH Oxidase Inhibitor, Induces Caspase-dependent Cell Death by Suppressing NOX4-ROS Signaling in EBV-infected Retinal Epithelial Cells.
Autorzy:
Hong SW; Department of Anatomy, Inje University College of Medicine , Pusan, Republic of Korea.; Department of Convergence Medicine, University of Ulsan College of Medicine, Asan Medical Center , Seoul, Republic of Korea.
Noh MH; Department of Anatomy, Inje University College of Medicine , Pusan, Republic of Korea.
Kim YS; Department of Anatomy, Inje University College of Medicine , Pusan, Republic of Korea.
Jin DH; Department of Convergence Medicine, University of Ulsan College of Medicine, Asan Medical Center , Seoul, Republic of Korea.
Moon SH; AptaBio Therapeutics Incorporation , Gyeonggi-do, Republic of Korea.
Yang JW; Department of Ophthalmology, Inje University Pusan Paik Hospital , Pusan, Republic of Korea.
Hur DY; Department of Anatomy, Inje University College of Medicine , Pusan, Republic of Korea.
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Źródło:
Current eye research [Curr Eye Res] 2020 Sep; Vol. 45 (9), pp. 1136-1143. Date of Electronic Publication: 2020 Feb 04.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Caspases/*metabolism
Herpesvirus 4, Human/*physiology
NADPH Oxidase 4/*metabolism
Pyrazoles/*pharmacology
Pyridines/*pharmacology
Reactive Oxygen Species/*metabolism
Retinal Pigment Epithelium/*pathology
Blotting, Western ; Cell Line ; Cell Proliferation ; Down-Regulation ; Enzyme Inhibitors/pharmacology ; Flow Cytometry ; Humans ; NADPH Oxidases/antagonists & inhibitors ; Retinal Pigment Epithelium/enzymology ; Retinal Pigment Epithelium/virology ; Signal Transduction/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
NADPH oxidase inhibition rescues keratinocytes from elevated oxidative stress in a 2D atopic dermatitis and psoriasis model.
Autorzy:
Emmert H; Department of Dermatology, Allergology and Venereology, University Hospital Schleswig-Holstein, Kiel, Germany.
Fonfara M; Department of Dermatology, Allergology and Venereology, University Hospital Schleswig-Holstein, Kiel, Germany.
Rodriguez E; Department of Dermatology, Allergology and Venereology, University Hospital Schleswig-Holstein, Kiel, Germany.
Weidinger S; Department of Dermatology, Allergology and Venereology, University Hospital Schleswig-Holstein, Kiel, Germany.
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Źródło:
Experimental dermatology [Exp Dermatol] 2020 Aug; Vol. 29 (8), pp. 749-758. Date of Electronic Publication: 2020 Jul 22.
Typ publikacji:
Journal Article
MeSH Terms:
Dermatitis, Atopic/*metabolism
Keratinocytes/*metabolism
NADPH Oxidases/*metabolism
Oxidative Stress/*drug effects
Psoriasis/*metabolism
Reactive Oxygen Species/*metabolism
Cells, Cultured ; DNA Damage/drug effects ; Enzyme Inhibitors/pharmacology ; Humans ; Hydrogen Peroxide/pharmacology ; In Vitro Techniques ; NADPH Oxidase 1/metabolism ; NADPH Oxidase 4/metabolism ; NADPH Oxidases/antagonists & inhibitors ; Onium Compounds/pharmacology ; Oxidants/pharmacology ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
RNA interference targeting NOX4 protects visual function in an experimental model of retinal detachment by alleviating blood-retinal barrier damage
Autorzy:
Kai Dong
Nan Yang
Jie Ding
Yuan-Ye Yan
Li Lu
Yi-Sai Wang
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Temat:
nox4
adeno-associated virus
retinal detachment
blood-retinal barrier
reactive oxygen species
Ophthalmology
RE1-994
Źródło:
International Journal of Ophthalmology, Vol 14, Iss 1, Pp 50-56 (2021)
Opis pliku:
electronic resource
Relacje:
http://ies.ijo.cn/en_publish/2021/1/20210107.pdf; https://doaj.org/toc/2222-3959; https://doaj.org/toc/2227-4898
Dostęp URL:
https://doaj.org/article/14b78ea0306b44ddb1485758677ce392  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Tytuł:
Sestrin2 Attenuates Cellular Senescence by Inhibiting NADPH Oxidase 4 Expression
Autorzy:
Chae Young Hwang
Ying-Hao Han
Seung-Min Lee
Sang-Mi Cho
Dae-Yeul Yu
Ki-Sun Kwon
Pokaż więcej
Temat:
nox4
reactive oxygen species
senescence
sestrin2
Medicine
Geriatrics
RC952-954.6
Źródło:
Annals of Geriatric Medicine and Research, Vol 24, Iss 4, Pp 297-304 (2020)
Opis pliku:
electronic resource
Relacje:
http://www.e-agmr.org/upload/pdf/agmr-20-0051.pdf; https://doaj.org/toc/2508-4798; https://doaj.org/toc/2508-4909
Dostęp URL:
https://doaj.org/article/07b45251ed8e4b5bb8b08789ff54a68a  Link otwiera się w nowym oknie
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Down-Regulation of NOX4 Expression in Dorsal Horn of Spinal Cord Could Alleviate Cancer-Induced Bone Pain in Rats by Reducing Oxidative Stress Response
Autorzy:
Long H
Zheng H
Ai L
Osman K
Liu Z
Pokaż więcej
Temat:
cancer induced bone pain
nox4
reactive oxygen species
spinal cord sensitization
Neoplasms. Tumors. Oncology. Including cancer and carcinogens
RC254-282
Źródło:
Cancer Management and Research, Vol Volume 12, Pp 10929-10938 (2020)
Opis pliku:
electronic resource
Relacje:
https://www.dovepress.com/down-regulation-of-nox4-expression-in-dorsal-horn-of-spinal-cord-could-peer-reviewed-article-CMAR; https://doaj.org/toc/1179-1322
Dostęp URL:
https://doaj.org/article/620863c1381c4b2b9a61cd37c0052fd0  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Tytuł:
The effect of nicotinamide adenine dinucleotide phosphate oxidase 4 on migration and invasion of fibroblast-like synoviocytes in rheumatoid arthritis
Autorzy:
Ha-Reum Lee
Su-Jin Yoo
Jinhyun Kim
In Seol Yoo
Chan Keol Park
Seong Wook Kang
Pokaż więcej
Temat:
Rheumatoid arthritis
Osteoarthritis
Fibroblast-like synoviocytes
Reactive oxygen species
NADPH
NOX4
Diseases of the musculoskeletal system
RC925-935
Źródło:
Arthritis Research & Therapy, Vol 22, Iss 1, Pp 1-11 (2020)
Opis pliku:
electronic resource
Relacje:
http://link.springer.com/article/10.1186/s13075-020-02204-0; https://doaj.org/toc/1478-6362
Dostęp URL:
https://doaj.org/article/d065e484e9ab4fc19de3a0a6bc68add9  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Tytuł:
A mitochondrial ROS pathway controls matrix metalloproteinase 9 levels and invasive properties in RAS-activated cancer cells.
Autorzy:
Mori K; Division of Cancer Cell Biology, Department of Pharmaceutical Sciences, Showa University School of Pharmacy, Tokyo, Japan.
Uchida T; Division of Cancer Cell Biology, Department of Pharmaceutical Sciences, Showa University School of Pharmacy, Tokyo, Japan.
Yoshie T; Division of Cancer Cell Biology, Department of Pharmaceutical Sciences, Showa University School of Pharmacy, Tokyo, Japan.
Mizote Y; Division of Cancer Cell Biology, Department of Pharmaceutical Sciences, Showa University School of Pharmacy, Tokyo, Japan.
Ishikawa F; Division of Cancer Cell Biology, Department of Pharmaceutical Sciences, Showa University School of Pharmacy, Tokyo, Japan.
Katsuyama M; Radioisotope Center, Kyoto Prefectural University of Medicine, Japan.
Shibanuma M; Division of Cancer Cell Biology, Department of Pharmaceutical Sciences, Showa University School of Pharmacy, Tokyo, Japan.
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Źródło:
The FEBS journal [FEBS J] 2019 Feb; Vol. 286 (3), pp. 459-478. Date of Electronic Publication: 2018 Oct 13.
Typ publikacji:
Editorial; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression Regulation, Neoplastic*
Breast Neoplasms/*genetics
Intracellular Signaling Peptides and Proteins/*genetics
LIM Domain Proteins/*genetics
Lung Neoplasms/*genetics
Mitochondria/*metabolism
NADPH Oxidase 4/*genetics
Reactive Oxygen Species/*metabolism
Animals ; Breast Neoplasms/metabolism ; Breast Neoplasms/pathology ; Cell Line, Tumor ; Cellular Senescence ; Epithelial Cells/metabolism ; Epithelial Cells/pathology ; Extracellular Matrix/metabolism ; Extracellular Matrix/pathology ; Female ; Focal Adhesions/metabolism ; Focal Adhesions/pathology ; Heterografts ; Humans ; Intracellular Signaling Peptides and Proteins/antagonists & inhibitors ; Intracellular Signaling Peptides and Proteins/metabolism ; LIM Domain Proteins/antagonists & inhibitors ; LIM Domain Proteins/metabolism ; Lung Neoplasms/metabolism ; Lung Neoplasms/secondary ; Matrix Metalloproteinase 9/genetics ; Matrix Metalloproteinase 9/metabolism ; Mice ; Mice, Inbred NOD ; Mitochondria/pathology ; NADPH Oxidase 4/metabolism ; Neoplasm Invasiveness ; Oxidative Stress ; Proto-Oncogene Proteins B-raf/genetics ; Proto-Oncogene Proteins B-raf/metabolism ; Proto-Oncogene Proteins p21(ras)/genetics ; Proto-Oncogene Proteins p21(ras)/metabolism ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; RNA, Small Interfering/genetics ; RNA, Small Interfering/metabolism ; Signal Transduction
Raport
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
TRPC1 Deficiency Exacerbates Cerebral Ischemia/Reperfusion-Induced Neurological Injury by Potentiating Nox4-Derived Reactive Oxygen Species Generation
Autorzy:
Ning Xu
Hao Meng
Tianyi Liu
Yingli Feng
Yuan Qi
Honglei Wang
Pokaż więcej
Temat:
Ischemia/reperfusion injury
Neurological dysfunction
Reactive oxygen species
NADPH
Nox4
TRPC1
Physiology
QP1-981
Biochemistry
QD415-436
Źródło:
Cellular Physiology and Biochemistry, Vol 51, Iss 4, Pp 1723-1738 (2018)
Opis pliku:
electronic resource
Relacje:
https://www.karger.com/Article/FullText/495676; https://doaj.org/toc/1015-8987; https://doaj.org/toc/1421-9778
Dostęp URL:
https://doaj.org/article/c51c6c6ad3e3426db6b528743a77d3b6  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Tytuł:
Oxidative Stress Induced by Reactive Oxygen Species (ROS) and NADPH Oxidase 4 (NOX4) in the Pathogenesis of the Fibrotic Process in Systemic Sclerosis: A Promising Therapeutic Target
Autorzy:
Sonsoles Piera-Velazquez
Sergio A. Jimenez
Pokaż więcej
Temat:
fibrosis
systemic sclerosis
oxidative stress
reactive oxygen species
NADPH
NOX4
Medicine
Źródło:
Journal of Clinical Medicine, Vol 10, Iss 20, p 4791 (2021)
Opis pliku:
electronic resource
Relacje:
https://www.mdpi.com/2077-0383/10/20/4791; https://doaj.org/toc/2077-0383
Dostęp URL:
https://doaj.org/article/3221c03ecedf48fe994eee0387d004bf  Link otwiera się w nowym oknie
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Wyświetlanie 1-20 z 155

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