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Tytuł :
Pharmacologic control of homeostatic and antigen-driven proliferation to target HIV-1 persistence.
Autorzy :
Innis EA; Division of Microbiology and Immunology, Department of Pathology, University of Utah School of Medicine, Salt Lake City, UT, USA.
Levinger C; Department of Microbiology, Immunology and Tropical Medicine, George Washington University, Washington, USA.
Szaniawski MA; Division of Microbiology and Immunology, Department of Pathology, University of Utah School of Medicine, Salt Lake City, UT, USA.
Williams ESCP; Division of Microbiology and Immunology, Department of Pathology, University of Utah School of Medicine, Salt Lake City, UT, USA.
Alcamí J; AIDS Immunopathology Unit, National Center of Microbiology (CNM), Instituto de Salud Carlos III (ISCIII), Madrid, Spain.
Bosque A; Department of Microbiology, Immunology and Tropical Medicine, George Washington University, Washington, USA.
Schiffer JT; Fred Hutchinson Cancer Research Center, Vaccine and Infectious Diseases Division, Seattle, WA 98109, USA.
Coiras M; AIDS Immunopathology Unit, National Center of Microbiology (CNM), Instituto de Salud Carlos III (ISCIII), Madrid, Spain.
Spivak AM; Division of Infectious Diseases, Department of Medicine, University of Utah School of Medicine, Salt Lake City, UT, USA. Electronic address: .
Planelles V; Division of Microbiology and Immunology, Department of Pathology, University of Utah School of Medicine, Salt Lake City, UT, USA. Electronic address: .
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Źródło :
Biochemical pharmacology [Biochem Pharmacol] 2021 Dec; Vol. 194, pp. 114816. Date of Electronic Publication: 2021 Oct 26.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Antigens, Viral*/metabolism
Cell Proliferation/*drug effects
Drug Delivery Systems/*methods
HIV-1/*drug effects
Homeostasis/*drug effects
Protein Kinase Inhibitors/*administration & dosage
Cell Proliferation/physiology ; Cells, Cultured ; Dasatinib/administration & dosage ; HIV Infections/drug therapy ; HIV Infections/immunology ; HIV Infections/metabolism ; HIV-1/immunology ; HIV-1/metabolism ; Homeostasis/physiology ; Humans ; Imidazoles/administration & dosage ; Leukocytes, Mononuclear/drug effects ; Leukocytes, Mononuclear/immunology ; Leukocytes, Mononuclear/metabolism ; Pyridazines/administration & dosage ; Pyridones/administration & dosage ; Pyrimidinones/administration & dosage
Czasopismo naukowe
Tytuł :
Hepatocyte Nuclear Factor 4-α (HNF4α) controls the insulin resistance-induced pancreatic β-cell mass expansion.
Autorzy :
Barth R; Islet Biology and Metabolism Lab - I.B.M. Lab, Department of Physiological Sciences, Center of Biological Sciences, Federal University of Santa Catarina - UFSC, Florianópolis 88040-900, Santa Catarina, Brazil; Multicenter Graduate Program in Physiological Sciences, Federal University of Santa Catarina (UFSC), 88040-900, Florianópolis, SC, Brazil.
Ruoso C; Islet Biology and Metabolism Lab - I.B.M. Lab, Department of Physiological Sciences, Center of Biological Sciences, Federal University of Santa Catarina - UFSC, Florianópolis 88040-900, Santa Catarina, Brazil; Multicenter Graduate Program in Physiological Sciences, Federal University of Santa Catarina (UFSC), 88040-900, Florianópolis, SC, Brazil.
Ferreira SM; Laboratory of endocrine pancreas and metabolism - LAPEM, Department of Structural and Functional Biology, Institute of Biology, State University of Campinas - UNICAMP, 13083-862 Campinas, Brazil.
de Ramos FC; Islet Biology and Metabolism Lab - I.B.M. Lab, Department of Physiological Sciences, Center of Biological Sciences, Federal University of Santa Catarina - UFSC, Florianópolis 88040-900, Santa Catarina, Brazil; Multicenter Graduate Program in Physiological Sciences, Federal University of Santa Catarina (UFSC), 88040-900, Florianópolis, SC, Brazil.
Lima FB; Islet Biology and Metabolism Lab - I.B.M. Lab, Department of Physiological Sciences, Center of Biological Sciences, Federal University of Santa Catarina - UFSC, Florianópolis 88040-900, Santa Catarina, Brazil; Multicenter Graduate Program in Physiological Sciences, Federal University of Santa Catarina (UFSC), 88040-900, Florianópolis, SC, Brazil.
Boschero AC; Laboratory of endocrine pancreas and metabolism - LAPEM, Department of Structural and Functional Biology, Institute of Biology, State University of Campinas - UNICAMP, 13083-862 Campinas, Brazil.
Santos GJD; Islet Biology and Metabolism Lab - I.B.M. Lab, Department of Physiological Sciences, Center of Biological Sciences, Federal University of Santa Catarina - UFSC, Florianópolis 88040-900, Santa Catarina, Brazil; Multicenter Graduate Program in Physiological Sciences, Federal University of Santa Catarina (UFSC), 88040-900, Florianópolis, SC, Brazil. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2022 Jan 15; Vol. 289, pp. 120213. Date of Electronic Publication: 2021 Dec 10.
Typ publikacji :
Journal Article
MeSH Terms :
Insulin Resistance*
Cell Proliferation/*drug effects
Dexamethasone/*pharmacology
Hepatocyte Nuclear Factor 4/*metabolism
Insulin Secretion/*drug effects
Insulin-Secreting Cells/*metabolism
Animals ; Basic Helix-Loop-Helix Transcription Factors/biosynthesis ; Basic Helix-Loop-Helix Transcription Factors/genetics ; Cell Proliferation/genetics ; Gene Expression Regulation/drug effects ; Hepatocyte Nuclear Factor 4/genetics ; Homeodomain Proteins/biosynthesis ; Homeodomain Proteins/genetics ; Insulin Secretion/genetics ; Mice ; Mice, Knockout ; Nerve Tissue Proteins/biosynthesis ; Nerve Tissue Proteins/genetics ; Paired Box Transcription Factors/biosynthesis ; Paired Box Transcription Factors/genetics ; Trans-Activators/biosynthesis ; Trans-Activators/genetics
Czasopismo naukowe
Tytuł :
The effects of UVB irradiance on aberrant epidermal proliferation: Novel insights on how to improve currently available sunscreens.
Autorzy :
Lai HC; Graduate Institute of Medicine, College of Medicine, Kaohsiung Medical University, Kaohsiung, Taiwan.
Lin CS; Graduate Institute of Medicine, College of Medicine, Kaohsiung Medical University, Kaohsiung, Taiwan; Department of Biological Sciences, National Sun Yat-sen University, Kaohsiung, Taiwan.
Wu CS; Graduate Institute of Medicine, College of Medicine, Kaohsiung Medical University, Kaohsiung, Taiwan; Department of Medical Laboratory Science and Biotechnology, Kaohsiung Medical University, Kaohsiung, Taiwan; Institute of Biomedical Sciences, National Sun Yat-Sen University, Kaohsiung, Taiwan.
Lan CE; Graduate Institute of Medicine, College of Medicine, Kaohsiung Medical University, Kaohsiung, Taiwan; Department of Dermatology, Kaohsiung Medical University Hospital, Kaohsiung, Taiwan; Department of Dermatology, College of Medicine, Kaohsiung Medical University, Kaohsiung, Taiwan. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2022 Jan 01; Vol. 288, pp. 120181. Date of Electronic Publication: 2021 Nov 27.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Proliferation*
Sunscreening Agents*
Epidermis/*pathology
Keratinocytes/*pathology
Skin/*pathology
Ultraviolet Rays/*adverse effects
Animals ; Cell Cycle ; Epidermis/radiation effects ; Keratinocytes/radiation effects ; Mice ; Mice, Hairless ; Phosphorylation ; Proto-Oncogene Proteins c-akt/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; Skin/radiation effects
Czasopismo naukowe
Tytuł :
Silencing KLF16 inhibits oral squamous cell carcinoma cell proliferation by arresting the cell cycle and inducing apoptosis.
Autorzy :
Yang L; Department of Orthodontics, Dongfeng Stomatological Hospital, Hubei University of Medicine, Shiyan, Hubei Province, China.
Shi YL; Department of Orthodontics, Dongfeng Stomatological Hospital, Hubei University of Medicine, Shiyan, Hubei Province, China.
Ma Y; Department of Orthodontics, Dongfeng Stomatological Hospital, Hubei University of Medicine, Shiyan, Hubei Province, China.
Ren WW; Department of Pediatric Stomatology, Dongfeng Stomatological Hospital, Hubei University of Medicine, Shiyan, Hubei Province, China.
Pang GM; Department of Orthodontics, Dongfeng Stomatological Hospital, Hubei University of Medicine, Shiyan, Hubei Province, China.
Liu J; Department of Pediatric Stomatology, Dongfeng Stomatological Hospital, Hubei University of Medicine, Shiyan, Hubei Province, China.
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Źródło :
APMIS : acta pathologica, microbiologica, et immunologica Scandinavica [APMIS] 2022 Jan; Vol. 130 (1), pp. 43-52. Date of Electronic Publication: 2021 Nov 25.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Cell Cycle*
Cell Proliferation*
Carcinoma, Squamous Cell/*metabolism
Kruppel-Like Transcription Factors/*metabolism
Mouth Neoplasms/*metabolism
Animals ; Carcinoma, Squamous Cell/genetics ; Cell Line, Tumor ; Cell Movement ; Gene Expression Regulation, Neoplastic ; Gene Silencing ; Humans ; Kruppel-Like Transcription Factors/genetics ; Mice ; Mice, Nude ; Mouth Neoplasms/genetics ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
RAF1-MEK/ERK pathway-dependent ARL4C expression promotes ameloblastoma cell proliferation and osteoclast formation.
Autorzy :
Fujii S; Laboratory of Oral Pathology, Division of Maxillofacial Diagnostic and Surgical Sciences, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.
Ishibashi T; Laboratory of Oral Pathology, Division of Maxillofacial Diagnostic and Surgical Sciences, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.
Kokura M; Laboratory of Oral Pathology, Division of Maxillofacial Diagnostic and Surgical Sciences, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.
Fujimoto T; Laboratory of Oral Pathology, Division of Maxillofacial Diagnostic and Surgical Sciences, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.
Matsumoto S; Department of Molecular Biology and Biochemistry, Graduate School of Medicine, Osaka University, Suita, Japan.; Integrated Frontier Research for Medical Science Division, Institute for Open and Transdisciplinary Research Initiatives (OTRI), Osaka University, Suita, Japan.
Shidara S; Laboratory of Oral Pathology, Division of Maxillofacial Diagnostic and Surgical Sciences, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.
Kurppa KJ; Institute of Biomedicine and MediCity Research Laboratories, University of Turku, and Turku Bioscience Centre, University of Turku and Åbo Akademi University, Turku, Finland.
Pape J; Division of Surgery and Interventional Science, Department of Targeted Intervention, Centre for 3D Models of Health and Disease, University College London, London, UK.
Caton J; Department of Anatomy and Embryology, Faculty of Medicine, University Complutense Madrid, Madrid, Spain.
Morgan PR; Head & Neck Pathology, King's College London, Guy's Hospital, London, UK.
Heikinheimo K; Department of Oral and Maxillofacial Surgery, Institute of Dentistry, University of Turku and Turku University Hospital, Turku, Finland.
Kikuchi A; Department of Molecular Biology and Biochemistry, Graduate School of Medicine, Osaka University, Suita, Japan.
Jimi E; Oral Health/Brain Health/Total Health Research Center, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.; Laboratory of Molecular and Cellular Biochemistry, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.
Kiyoshima T; Laboratory of Oral Pathology, Division of Maxillofacial Diagnostic and Surgical Sciences, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.
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Źródło :
The Journal of pathology [J Pathol] 2022 Jan; Vol. 256 (1), pp. 119-133. Date of Electronic Publication: 2021 Nov 16.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
ADP-Ribosylation Factors/*metabolism
Ameloblastoma/*metabolism
Cell Proliferation/*genetics
Extracellular Signal-Regulated MAP Kinases/*metabolism
Osteoclasts/*pathology
Ameloblastoma/genetics ; Cell Proliferation/physiology ; Cell Transformation, Neoplastic/genetics ; Female ; Gene Expression Regulation, Neoplastic/genetics ; Humans ; MAP Kinase Signaling System/physiology ; Male ; Mitogen-Activated Protein Kinase Kinases/metabolism ; Osteoclasts/metabolism ; Protein Serine-Threonine Kinases/genetics ; Protein Serine-Threonine Kinases/metabolism ; Wnt Signaling Pathway/genetics
Czasopismo naukowe
Tytuł :
Antiproliferative and antiangiogenic effects of ammonium tetrathiomolybdate in a model of endometriosis.
Autorzy :
Delsouc MB; Facultad de Química, Bioquímica y Farmacia, Universidad Nacional de San Luis (UNSL), San Luis, Argentina; Instituto Multidisciplinario de Investigaciones Biológicas de San Luis (IMIBIO-SL-CONICET), San Luis, Argentina.
Conforti RA; Facultad de Química, Bioquímica y Farmacia, Universidad Nacional de San Luis (UNSL), San Luis, Argentina; Instituto Multidisciplinario de Investigaciones Biológicas de San Luis (IMIBIO-SL-CONICET), San Luis, Argentina.
Vitale DL; Laboratorio de Microambiente Tumoral, Centro de Investigaciones Básicas y Aplicadas (CIBA), Universidad Nacional del Noroeste de la Provincia de Buenos Aires (UNNOBA), Junín, Buenos Aires, Argentina; Centro de Investigaciones y Transferencia del Noroeste de la Provincia de Buenos Aires (CITNOBA), UNNOBA-UNSAdA-CONICET, Junín, Buenos Aires, Argentina.
Alaniz L; Laboratorio de Microambiente Tumoral, Centro de Investigaciones Básicas y Aplicadas (CIBA), Universidad Nacional del Noroeste de la Provincia de Buenos Aires (UNNOBA), Junín, Buenos Aires, Argentina; Centro de Investigaciones y Transferencia del Noroeste de la Provincia de Buenos Aires (CITNOBA), UNNOBA-UNSAdA-CONICET, Junín, Buenos Aires, Argentina.
Pacheco P; Facultad de Química, Bioquímica y Farmacia, Universidad Nacional de San Luis (UNSL), San Luis, Argentina; Instituto de Química de San Luis (INQUISAL-CONICET), San Luis, Argentina.
Andujar S; Facultad de Química, Bioquímica y Farmacia, Universidad Nacional de San Luis (UNSL), San Luis, Argentina; Instituto Multidisciplinario de Investigaciones Biológicas de San Luis (IMIBIO-SL-CONICET), San Luis, Argentina.
Vallcaneras SS; Facultad de Química, Bioquímica y Farmacia, Universidad Nacional de San Luis (UNSL), San Luis, Argentina; Instituto Multidisciplinario de Investigaciones Biológicas de San Luis (IMIBIO-SL-CONICET), San Luis, Argentina.
Casais M; Facultad de Química, Bioquímica y Farmacia, Universidad Nacional de San Luis (UNSL), San Luis, Argentina; Instituto Multidisciplinario de Investigaciones Biológicas de San Luis (IMIBIO-SL-CONICET), San Luis, Argentina. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2021 Dec 15; Vol. 287, pp. 120099. Date of Electronic Publication: 2021 Oct 29.
Typ publikacji :
Journal Article
MeSH Terms :
Disease Models, Animal*
Angiogenesis Inhibitors/*therapeutic use
Cell Proliferation/*drug effects
Endometriosis/*drug therapy
Molybdenum/*therapeutic use
Angiogenesis Inhibitors/pharmacology ; Animals ; Cell Proliferation/physiology ; Endometriosis/pathology ; Female ; Mice ; Mice, Inbred C57BL ; Molybdenum/pharmacology
Czasopismo naukowe
Tytuł :
VEGFB Promotes Myoblasts Proliferation and Differentiation through VEGFR1-PI3K/Akt Signaling Pathway.
Autorzy :
Ling M; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Quan L; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Lai X; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Lang L; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Li F; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Yang X; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Fu Y; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Feng S; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Yi X; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Zhu C; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Gao P; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Zhu X; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Wang L; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Shu G; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Jiang Q; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
Wang S; Guangdong Provincial Key Laboratory of Animal Nutrition Control, College of Animal Science, South China Agricultural University, Guangzhou 510642, China.; National Engineering Research Center for the Breeding Swine Industry, South China Agricultural University, Guangzhou 510642, China.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Dec 12; Vol. 22 (24). Date of Electronic Publication: 2021 Dec 12.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Differentiation*
Cell Proliferation*
Signal Transduction*
Myoblasts/*metabolism
Phosphatidylinositol 3-Kinases/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Vascular Endothelial Growth Factor B/*metabolism
Vascular Endothelial Growth Factor Receptor-1/*metabolism
Animals ; Cell Line ; Mice ; Vascular Endothelial Growth Factor B/pharmacology
Czasopismo naukowe
Tytuł :
Increased Susceptibility of the CD57 NK Cells Expressing KIR2DL2/3 and NKG2C to iCasp9 Gene Retroviral Transduction and the Relationships with Proliferative Potential, Activation Degree, and Death Induction Response.
Autorzy :
Palamarchuk AI; Shemyakin and Ovchinnikov Institute of Bioorganic Chemistry, Russian Academy of Sciences, st. Miklukho-Maklaya, 16/10, 117997 Moscow, Russia.
Alekseeva NA; Shemyakin and Ovchinnikov Institute of Bioorganic Chemistry, Russian Academy of Sciences, st. Miklukho-Maklaya, 16/10, 117997 Moscow, Russia.
Streltsova MA; Shemyakin and Ovchinnikov Institute of Bioorganic Chemistry, Russian Academy of Sciences, st. Miklukho-Maklaya, 16/10, 117997 Moscow, Russia.
Ustiuzhanina MO; Shemyakin and Ovchinnikov Institute of Bioorganic Chemistry, Russian Academy of Sciences, st. Miklukho-Maklaya, 16/10, 117997 Moscow, Russia.; Center of Life Sciences, Skolkovo Institute of Science and Technology, 121205 Moscow, Russia.
Kobyzeva PA; Shemyakin and Ovchinnikov Institute of Bioorganic Chemistry, Russian Academy of Sciences, st. Miklukho-Maklaya, 16/10, 117997 Moscow, Russia.
Kust SA; Shemyakin and Ovchinnikov Institute of Bioorganic Chemistry, Russian Academy of Sciences, st. Miklukho-Maklaya, 16/10, 117997 Moscow, Russia.
Grechikhina MV; Shemyakin and Ovchinnikov Institute of Bioorganic Chemistry, Russian Academy of Sciences, st. Miklukho-Maklaya, 16/10, 117997 Moscow, Russia.
Boyko AA; Shemyakin and Ovchinnikov Institute of Bioorganic Chemistry, Russian Academy of Sciences, st. Miklukho-Maklaya, 16/10, 117997 Moscow, Russia.
Shustova OA; Shemyakin and Ovchinnikov Institute of Bioorganic Chemistry, Russian Academy of Sciences, st. Miklukho-Maklaya, 16/10, 117997 Moscow, Russia.
Sapozhnikov AM; Shemyakin and Ovchinnikov Institute of Bioorganic Chemistry, Russian Academy of Sciences, st. Miklukho-Maklaya, 16/10, 117997 Moscow, Russia.
Kovalenko EI; Shemyakin and Ovchinnikov Institute of Bioorganic Chemistry, Russian Academy of Sciences, st. Miklukho-Maklaya, 16/10, 117997 Moscow, Russia.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Dec 11; Vol. 22 (24). Date of Electronic Publication: 2021 Dec 11.
Typ publikacji :
Journal Article
MeSH Terms :
CRISPR-Cas Systems*
Cell Proliferation*
Gene Expression Regulation*
Genetic Vectors*
Lymphocyte Activation*
Retroviridae*
Transduction, Genetic*
NK Cell Lectin-Like Receptor Subfamily C/*biosynthesis
Receptors, KIR2DL2/*biosynthesis
Receptors, KIR2DL3/*biosynthesis
Cell Death ; Humans ; K562 Cells ; Killer Cells, Natural ; NK Cell Lectin-Like Receptor Subfamily C/genetics ; Receptors, KIR2DL2/genetics ; Receptors, KIR2DL3/genetics
Czasopismo naukowe
Tytuł :
Validation of a Lab-on-Chip Assay for Measuring Sorafenib Effectiveness on HCC Cell Proliferation.
Autorzy :
Piccinno E; National Institute of Gastroenterology 'Saverio de Bellis', Research Hospital, Castellana Grotte, 70013 Bari, Italy.
Monteduro AG; Omnics Research Group, Department of Mathematics and Physics 'Ennio De Giorgi', Institute of Nanotechnology CNR-Nanotec and INFN Sezione di Lecce, University of Salento, Via per Monteroni, 73100 Lecce, Italy.
Dituri F; National Institute of Gastroenterology 'Saverio de Bellis', Research Hospital, Castellana Grotte, 70013 Bari, Italy.
Rizzato S; Omnics Research Group, Department of Mathematics and Physics 'Ennio De Giorgi', Institute of Nanotechnology CNR-Nanotec and INFN Sezione di Lecce, University of Salento, Via per Monteroni, 73100 Lecce, Italy.
Giannelli G; National Institute of Gastroenterology 'Saverio de Bellis', Research Hospital, Castellana Grotte, 70013 Bari, Italy.
Maruccio G; Omnics Research Group, Department of Mathematics and Physics 'Ennio De Giorgi', Institute of Nanotechnology CNR-Nanotec and INFN Sezione di Lecce, University of Salento, Via per Monteroni, 73100 Lecce, Italy.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Dec 03; Vol. 22 (23). Date of Electronic Publication: 2021 Dec 03.
Typ publikacji :
Journal Article; Validation Study
MeSH Terms :
Cell Proliferation*
Lab-On-A-Chip Devices*
Carcinoma, Hepatocellular/*drug therapy
Drug Evaluation, Preclinical/*methods
Liver Neoplasms/*drug therapy
Sorafenib/*pharmacology
Antineoplastic Agents/pharmacology ; Carcinoma, Hepatocellular/physiopathology ; Fibroblasts/drug effects ; Fibroblasts/physiology ; Humans ; Liver Neoplasms/physiopathology
Czasopismo naukowe
Tytuł :
IRF4 L116R mutation promotes proliferation of chronic lymphocytic leukemia B cells inducing MYC.
Autorzy :
Benatti S; Division of Hematology, Department of Medical and Surgical Sciences, University of Modena and Reggio Emilia, Modena, Italy.
Atene CG; Division of Hematology, Department of Medical and Surgical Sciences, University of Modena and Reggio Emilia, Modena, Italy.
Fiorcari S; Division of Hematology, Department of Medical and Surgical Sciences, University of Modena and Reggio Emilia, Modena, Italy.
Mesini N; Division of Hematology, Department of Medical and Surgical Sciences, University of Modena and Reggio Emilia, Modena, Italy.
Martinelli S; Division of Hematology, Department of Medical and Surgical Sciences, University of Modena and Reggio Emilia, Modena, Italy.
Zucchini P; Division of Hematology, Department of Medical and Surgical Sciences, University of Modena and Reggio Emilia, Modena, Italy.
Bacchelli F; Division of Hematology, Department of Medical and Surgical Sciences, University of Modena and Reggio Emilia, Modena, Italy.
Maccaferri M; Department of Oncology and Hematology, Hematology Division, Azienda Ospedaliero-Universitaria of Modena-Policlinico, Modena, Italy.
Debbia G; Division of Hematology, Department of Medical and Surgical Sciences, University of Modena and Reggio Emilia, Modena, Italy.
Potenza L; Division of Hematology, Department of Medical and Surgical Sciences, University of Modena and Reggio Emilia, Modena, Italy.
Rossi D; Hematology, Oncology Institute of Southern Switzerland and Institute of Oncology Research, Bellinzona, Switzerland.
Vallisa D; Division of Hematology, Guglielmo da Saliceto Hospital, Piacenza, Italy.
Trentin L; Hematology and Clinical Immunology, Department of Medicine, University of Padua, Padua, Italy.
Gaidano G; Division of Hematology, Department of Translational Medicine, University of Eastern Piedmont, Novara, Italy.
Luppi M; Division of Hematology, Department of Medical and Surgical Sciences, University of Modena and Reggio Emilia, Modena, Italy.
Marasca R; Division of Hematology, Department of Medical and Surgical Sciences, University of Modena and Reggio Emilia, Modena, Italy.
Maffei R; Department of Oncology and Hematology, Hematology Division, Azienda Ospedaliero-Universitaria of Modena-Policlinico, Modena, Italy.
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Źródło :
Hematological oncology [Hematol Oncol] 2021 Dec; Vol. 39 (5), pp. 707-711. Date of Electronic Publication: 2021 Aug 25.
Typ publikacji :
Case Reports; Letter
MeSH Terms :
Cell Proliferation*
Mutation*
Biomarkers, Tumor/*metabolism
Interferon Regulatory Factors/*genetics
Leukemia, Lymphocytic, Chronic, B-Cell/*pathology
Proto-Oncogene Proteins c-myc/*metabolism
Aged ; Biomarkers, Tumor/genetics ; Cohort Studies ; Follow-Up Studies ; Gene Expression Regulation, Neoplastic ; Humans ; Interferon Regulatory Factors/metabolism ; Leukemia, Lymphocytic, Chronic, B-Cell/genetics ; Leukemia, Lymphocytic, Chronic, B-Cell/metabolism ; Male ; Prognosis ; Proto-Oncogene Proteins c-myc/genetics ; Tumor Cells, Cultured
Raport
Tytuł :
Deletion of Nf2 in neural crest-derived tongue mesenchyme alters tongue shape and size, Hippo signalling and cell proliferation in a region- and stage-specific manner.
Autorzy :
Ishan M; Regenerative Bioscience Center, University of Georgia, Athens, GA, USA.; Department of Animal and Dairy Science, College of Agricultural and Environmental Sciences, University of Georgia, Athens, GA, USA.
Chen G; Regenerative Bioscience Center, University of Georgia, Athens, GA, USA.; Department of Animal and Dairy Science, College of Agricultural and Environmental Sciences, University of Georgia, Athens, GA, USA.
Yu W; Regenerative Bioscience Center, University of Georgia, Athens, GA, USA.; Department of Animal and Dairy Science, College of Agricultural and Environmental Sciences, University of Georgia, Athens, GA, USA.
Wang Z; Regenerative Bioscience Center, University of Georgia, Athens, GA, USA.; Department of Animal and Dairy Science, College of Agricultural and Environmental Sciences, University of Georgia, Athens, GA, USA.
Giovannini M; Department of Head and Neck Surgery, David Geffen School of Medicine, University of California Los Angeles, Los Angeles, CA, USA.
Cao X; Department of Developmental Neurobiology, St Jude Children's Research Hospital, Memphis, TN, USA.
Liu HX; Regenerative Bioscience Center, University of Georgia, Athens, GA, USA.; Department of Animal and Dairy Science, College of Agricultural and Environmental Sciences, University of Georgia, Athens, GA, USA.
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Źródło :
Cell proliferation [Cell Prolif] 2021 Dec; Vol. 54 (12), pp. e13144. Date of Electronic Publication: 2021 Oct 26.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Proliferation*
Gene Deletion*
Hippo Signaling Pathway*
Mesoderm/*embryology
NF-E2-Related Factor 2/*deficiency
Neural Crest/*embryology
Tongue/*embryology
Animals ; Mice ; Mice, Transgenic ; NF-E2-Related Factor 2/metabolism ; Organ Size
Czasopismo naukowe
Tytuł :
Proliferation in the developing intestine is regulated by the endosomal protein Endotubin.
Autorzy :
Wu MH; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, AZ, USA. Electronic address: .
Padilla-Rodriguez M; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, AZ, USA. Electronic address: .
Blum I; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, AZ, USA. Electronic address: .
Camenisch A; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, AZ, USA. Electronic address: .
Figliuolo da Paz V; Departments of Pediatrics and Immunobiology, University of Arizona, Tucson, AZ, USA. Electronic address: .
Ollerton M; Department of Medicine, University of Arizona, Tucson, AZ, USA. Electronic address: .
Muller J; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, AZ, USA. Electronic address: .
Momtaz S; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, AZ, USA. Electronic address: .
Mitchell SAT; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, AZ, USA. Electronic address: .
Kiela P; Departments of Pediatrics and Immunobiology, University of Arizona, Tucson, AZ, USA; Steele Children's Research Center, University of Arizona, Tucson, AZ, USA. Electronic address: .
Thorne C; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, AZ, USA; The University of Arizona Cancer Center, University of Arizona, Tucson, AZ, USA; Bio5 Institute, University of Arizona, Tucson, AZ, USA; Steele Children's Research Center, University of Arizona, Tucson, AZ, USA. Electronic address: .
Wilson JM; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, AZ, USA; The University of Arizona Cancer Center, University of Arizona, Tucson, AZ, USA; Bio5 Institute, University of Arizona, Tucson, AZ, USA. Electronic address: .
Cox CM; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, AZ, USA. Electronic address: .
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Źródło :
Developmental biology [Dev Biol] 2021 Dec; Vol. 480, pp. 50-61. Date of Electronic Publication: 2021 Aug 17.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Cell Proliferation/*genetics
Glycoproteins/*genetics
Intestines/*embryology
Animals ; Cell Differentiation/genetics ; Cell Proliferation/physiology ; Endosomes/metabolism ; Enterocytes/metabolism ; Female ; Glycoproteins/metabolism ; Intestinal Mucosa/metabolism ; Intestines/metabolism ; Male ; Mice ; Mice, Knockout ; Microfilament Proteins/genetics ; Wnt Proteins/metabolism ; Wnt Signaling Pathway/physiology
Czasopismo naukowe
Tytuł :
Olfactory Receptor OR7A17 Expression Correlates with All- Trans Retinoic Acid (ATRA)-Induced Suppression of Proliferation in Human Keratinocyte Cells.
Autorzy :
Kim H; Molecular Dermatology Laboratory, Department of Integrative Biotechnology, College of Biotechnology and Bioengineering, Sungkyunkwan University, Suwon City 16419, Gyunggi Do, Korea.
Park SH; Department of Bio and Chemical Engineering, Hongik University, Sejong City 30016, Korea.
Oh SW; Molecular Dermatology Laboratory, Department of Integrative Biotechnology, College of Biotechnology and Bioengineering, Sungkyunkwan University, Suwon City 16419, Gyunggi Do, Korea.
Kwon K; Molecular Dermatology Laboratory, Department of Integrative Biotechnology, College of Biotechnology and Bioengineering, Sungkyunkwan University, Suwon City 16419, Gyunggi Do, Korea.
Park SJ; Molecular Dermatology Laboratory, Department of Integrative Biotechnology, College of Biotechnology and Bioengineering, Sungkyunkwan University, Suwon City 16419, Gyunggi Do, Korea.
Yu E; Molecular Dermatology Laboratory, Department of Integrative Biotechnology, College of Biotechnology and Bioengineering, Sungkyunkwan University, Suwon City 16419, Gyunggi Do, Korea.
Yang S; Molecular Dermatology Laboratory, Department of Integrative Biotechnology, College of Biotechnology and Bioengineering, Sungkyunkwan University, Suwon City 16419, Gyunggi Do, Korea.
Park JY; Molecular Dermatology Laboratory, Department of Integrative Biotechnology, College of Biotechnology and Bioengineering, Sungkyunkwan University, Suwon City 16419, Gyunggi Do, Korea.
Choi S; Molecular Dermatology Laboratory, Department of Integrative Biotechnology, College of Biotechnology and Bioengineering, Sungkyunkwan University, Suwon City 16419, Gyunggi Do, Korea.
Yang S; Molecular Dermatology Laboratory, Department of Integrative Biotechnology, College of Biotechnology and Bioengineering, Sungkyunkwan University, Suwon City 16419, Gyunggi Do, Korea.
Han SB; Molecular Dermatology Laboratory, Department of Integrative Biotechnology, College of Biotechnology and Bioengineering, Sungkyunkwan University, Suwon City 16419, Gyunggi Do, Korea.
Song M; Integrative Research of T Cells Laboratory, Department of Integrative Biotechnology, College of Biotechnology and Bioengineering, Sungkyunkwan University, Suwon City 16419, Gyunggi Do, Korea.
Cho JY; Molecular Immunology Laboratory, Department of Integrative Biotechnology, College of Biotechnology and Bioengineering, Sungkyunkwan University, Suwon City 16419, Gyunggi Do, Korea.
Lee J; Molecular Dermatology Laboratory, Department of Integrative Biotechnology, College of Biotechnology and Bioengineering, Sungkyunkwan University, Suwon City 16419, Gyunggi Do, Korea.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Nov 14; Vol. 22 (22). Date of Electronic Publication: 2021 Nov 14.
Typ publikacji :
Journal Article
MeSH Terms :
Gene Expression*
Cell Proliferation/*drug effects
Keratinocytes/*drug effects
Receptors, Odorant/*genetics
Tretinoin/*pharmacology
Antineoplastic Agents/pharmacology ; Blotting, Western ; Calcium/metabolism ; Cell Line ; Cell Proliferation/genetics ; Down-Regulation/drug effects ; Down-Regulation/genetics ; Humans ; Keratinocytes/cytology ; Keratinocytes/metabolism ; Receptors, Odorant/metabolism ; Reverse Transcriptase Polymerase Chain Reaction
Czasopismo naukowe
Tytuł :
ROR2 has a protective role in melanoma by inhibiting Akt activity, cell-cycle progression, and proliferation.
Autorzy :
Castro MV; Centro de Estudios Biomédicos, Básicos, Aplicados y Desarrollo (CEBBAD), Universidad Maimónides, 1405, Buenos Aires, Argentina.; Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), 1425, Buenos Aires, Argentina.
Barbero GA; Centro de Estudios Biomédicos, Básicos, Aplicados y Desarrollo (CEBBAD), Universidad Maimónides, 1405, Buenos Aires, Argentina.; Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), 1425, Buenos Aires, Argentina.
Villanueva MB; Centro de Estudios Biomédicos, Básicos, Aplicados y Desarrollo (CEBBAD), Universidad Maimónides, 1405, Buenos Aires, Argentina.; Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), 1425, Buenos Aires, Argentina.
Grumolato L; INSERM U982, Institute for Research and Innovation in Biomedicine, University of Rouen, 76183, Rouen, France.
Nsengimana J; Biostatistics Research Group, Population Health Sciences Institute, Faculty of Medical Sciences, Newcastle University, Newcastle upon Tyne, NE2 4HH, UK.
Newton-Bishop J; Leeds Institute of Medical Research, Leeds, LS2 9JT, UK.
Illescas E; Centro de Estudios Biomédicos, Básicos, Aplicados y Desarrollo (CEBBAD), Universidad Maimónides, 1405, Buenos Aires, Argentina.
Quezada MJ; Centro de Estudios Biomédicos, Básicos, Aplicados y Desarrollo (CEBBAD), Universidad Maimónides, 1405, Buenos Aires, Argentina.; Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), 1425, Buenos Aires, Argentina.
Lopez-Bergami P; Centro de Estudios Biomédicos, Básicos, Aplicados y Desarrollo (CEBBAD), Universidad Maimónides, 1405, Buenos Aires, Argentina. .; Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), 1425, Buenos Aires, Argentina. .; Centro de Estudios Biomédicos, Biotecnológicos, Ambientales y Diagnóstico, Universidad Maimonides, Hidalgo 775, 6th Floor, Lab 602., 1405, Buenos Aires, Argentina. .
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Źródło :
Journal of biomedical science [J Biomed Sci] 2021 Nov 13; Vol. 28 (1), pp. 76. Date of Electronic Publication: 2021 Nov 13.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Cycle*
Cell Proliferation*
Melanoma/*genetics
Proto-Oncogene Proteins c-akt/*genetics
Receptor Tyrosine Kinase-like Orphan Receptors/*genetics
Proto-Oncogene Proteins c-akt/metabolism
Czasopismo naukowe
Tytuł :
Early Adventitial Activation and Proliferation in a Mouse Model of Arteriovenous Stenosis: Opportunities for Intervention.
Autorzy :
Chan JS; Division of Nephrology, Department of Internal Medicine, Armed Forces Taoyuan General Hospital, Taoyuan 325, Taiwan.; Division of Nephrology, Department of Medicine, Tri-Service General Hospital, National Defense Medical Center, Taipei 114, Taiwan.; School of Medicine, National Defense Medical Center, Taipei 114, Taiwan.
Wang Y; Division of Nephrology, Department of Internal Medicine, University of Cincinnati, Cincinnati, OH 45267, USA.
Cornea V; Department of Pathology, University of Cincinnati, Cincinnati, OH 45267, USA.
Roy-Chaudhury P; Department of Medicine, Division of Nephrology and Hypertension, University of North Carolina, Chapel Hill, NC 27599, USA.; WG (Bill) Hefner Salisbury VA Medical Center, Salisbury, NC 27284, USA.
Campos B; Division of Nephrology, Department of Internal Medicine, University of Cincinnati, Cincinnati, OH 45267, USA.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Nov 13; Vol. 22 (22). Date of Electronic Publication: 2021 Nov 13.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Proliferation*
Adventitia/*metabolism
Endothelial Cells/*metabolism
Macrophages/*metabolism
Neointima/*metabolism
Adventitia/pathology ; Animals ; Constriction, Pathologic/metabolism ; Constriction, Pathologic/pathology ; Disease Models, Animal ; Endothelial Cells/pathology ; Macrophages/pathology ; Mice ; Neointima/pathology
Czasopismo naukowe
Tytuł :
Fukutin Protein Participates in Cell Proliferation by Enhancing Cyclin D1 Expression through Binding to the Transcription Factor Activator Protein-1: An In Vitro Study.
Autorzy :
Okamura Y; Division of Human Pathology & Pathological Neuroscience, Department of Pathology, Graduate School of Medicine, Tokyo Women's Medical University, Tokyo 162-8666, Japan.
Yamamoto T; Division of Human Pathology & Pathological Neuroscience, Department of Pathology, Graduate School of Medicine, Tokyo Women's Medical University, Tokyo 162-8666, Japan.
Tsukui R; Division of Human Pathology & Pathological Neuroscience, Department of Pathology, Graduate School of Medicine, Tokyo Women's Medical University, Tokyo 162-8666, Japan.
Kato Y; Division of Human Pathology & Pathological Neuroscience, Department of Pathology, Graduate School of Medicine, Tokyo Women's Medical University, Tokyo 162-8666, Japan.
Shibata N; Division of Human Pathology & Pathological Neuroscience, Department of Pathology, Graduate School of Medicine, Tokyo Women's Medical University, Tokyo 162-8666, Japan.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Nov 10; Vol. 22 (22). Date of Electronic Publication: 2021 Nov 10.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Proliferation*
Gene Expression Regulation*
Astrocytes/*metabolism
Cyclin D1/*biosynthesis
Membrane Proteins/*metabolism
Transcription Factor AP-1/*metabolism
Cell Line, Tumor ; Humans ; Proto-Oncogene Proteins c-jun/metabolism
Czasopismo naukowe
Tytuł :
Nkx2-3 induces autophagy inhibiting proliferation and migration of vascular smooth muscle cells via AMPK/mTOR signaling pathway.
Autorzy :
Zheng H; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Zhai W; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Zhong C; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Hong Q; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Li H; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Rui B; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Zhu X; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Que D; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Feng L; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Yu B; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Huang G; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Yin J; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Li J; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Yan J; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
Yang P; Department of Cardiology, Laboratory of Heart Center, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Guangdong Provincial Biomedical Engineering Technology Research Center for Cardiovascular Disease, Guangzhou, Guangdong, China.; Heart Center of Zhujiang Hospital, Sino-Japanese Cooperation Platform for Translational Research in Heart Failure, Guangzhou, Guangdong, China.
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Źródło :
Journal of cellular physiology [J Cell Physiol] 2021 Nov; Vol. 236 (11), pp. 7342-7355. Date of Electronic Publication: 2021 Apr 30.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Autophagy*/drug effects
Cell Movement*/drug effects
Cell Proliferation*/drug effects
AMP-Activated Protein Kinases/*metabolism
Carotid Artery Injuries/*enzymology
Muscle, Smooth, Vascular/*enzymology
Myocytes, Smooth Muscle/*enzymology
TOR Serine-Threonine Kinases/*metabolism
Animals ; Becaplermin/pharmacology ; Carotid Artery Injuries/genetics ; Carotid Artery Injuries/pathology ; Carotid Artery Injuries/prevention & control ; Cells, Cultured ; Disease Models, Animal ; Male ; Muscle, Smooth, Vascular/drug effects ; Muscle, Smooth, Vascular/ultrastructure ; Myocytes, Smooth Muscle/drug effects ; Myocytes, Smooth Muscle/ultrastructure ; Neointima ; Rats, Sprague-Dawley ; Signal Transduction ; Vascular Remodeling
Czasopismo naukowe
Tytuł :
Reg4 regulates pancreatic regeneration following pancreatitis via modulating the Notch signaling.
Autorzy :
Dai J; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Department of Intensive Care Unit, State Key Laboratory of Oncology in South China, Sun Yat-sen University Cancer Center, Collaborative Innovation Center for Cancer Medicine, Guangzhou, China.
He Y; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Jiang M; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Department of Head and Neck, State Key Laboratory of Oncology in South China, Sun Yat-sen University Cancer Center, Collaborative Innovation Center for Cancer Medicine, Guangzhou, China.
Niu M; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Li B; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Wu Z; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Bao J; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Wen L; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Wang X; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Hu G; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
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Źródło :
Journal of cellular physiology [J Cell Physiol] 2021 Nov; Vol. 236 (11), pp. 7565-7577. Date of Electronic Publication: 2021 Apr 26.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Proliferation*
Regeneration*
Pancreas/*metabolism
Pancreatitis/*metabolism
Pancreatitis-Associated Proteins/*metabolism
Receptors, Notch/*metabolism
Animals ; Disease Models, Animal ; HEK293 Cells ; Humans ; Male ; Metaplasia ; Mice, Inbred C57BL ; Pancreas/pathology ; Pancreatitis/genetics ; Pancreatitis/pathology ; Signal Transduction
Czasopismo naukowe
Tytuł :
[Effects of miR-155-3p on the degradation rate of EAF1 mRNA and malignant proliferation in HANK1 cells].
Autorzy :
Liu L; Weifang NO. 2 People's Hospital, Weifang 261041.
Xu K; Sunshine Union Hospital, Weifang 261000.
Wang LQ; Sunshine Union Hospital, Weifang 261000.
Xu ML; Weifang NO. 2 People's Hospital, Weifang 261041, China.
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Źródło :
Zhongguo ying yong sheng li xue za zhi = Zhongguo yingyong shenglixue zazhi = Chinese journal of applied physiology [Zhongguo Ying Yong Sheng Li Xue Za Zhi] 2021 Nov; Vol. 37 (6), pp. 601-605.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Proliferation*
MicroRNAs*/genetics
Transcription Factors*/genetics
Cell Line, Tumor ; Gene Expression Regulation, Neoplastic ; Humans ; Proto-Oncogene Proteins c-myc/genetics ; Proto-Oncogene Proteins c-myc/metabolism ; RNA, Messenger/genetics
Czasopismo naukowe
Tytuł :
Colon fibroblasts from Pirc rats (F344/NTac-Apc ) exhibit a proliferative and inflammatory phenotype that could support early stages of colon carcinogenesis.
Autorzy :
Tortora K; NEUROFARBA Department, Pharmacology and Toxicology Section, University of Florence, Florence, Italy.
Margheri F; Department of Experimental and Clinical Biomedical Sciences Mario Serio, University of Florence, Florence, Italy.
Luceri C; NEUROFARBA Department, Pharmacology and Toxicology Section, University of Florence, Florence, Italy.
Mocali A; Department of Experimental and Clinical Biomedical Sciences Mario Serio, University of Florence, Florence, Italy.
Ristori S; NEUROFARBA Department, Pharmacology and Toxicology Section, University of Florence, Florence, Italy.
Magnelli L; Department of Experimental and Clinical Biomedical Sciences Mario Serio, University of Florence, Florence, Italy.
Caderni G; NEUROFARBA Department, Pharmacology and Toxicology Section, University of Florence, Florence, Italy.
Giovannelli L; NEUROFARBA Department, Pharmacology and Toxicology Section, University of Florence, Florence, Italy.
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Źródło :
International journal of cancer [Int J Cancer] 2022 Jan 15; Vol. 150 (2), pp. 362-373. Date of Electronic Publication: 2021 Sep 18.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Proliferation*
DNA Damage*
Genes, APC*
Colon/*pathology
Colonic Neoplasms/*pathology
Fibroblasts/*pathology
Inflammation/*pathology
Animals ; Apoptosis ; Colon/metabolism ; Colonic Neoplasms/etiology ; Colonic Neoplasms/metabolism ; Fibroblasts/metabolism ; Inflammation/etiology ; Inflammation/metabolism ; Mutation ; Phenotype ; Rats ; Rats, Inbred F344
Czasopismo naukowe

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