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Tytuł:
Involvement of the OTUB1-YAP1 axis in driving malignant behaviors of head and neck squamous cell carcinoma.
Autorzy:
Jin S; Department of Oral Bioscience, Tokushima University Graduate School of Biomedical Sciences, Tokushima, Japan.
Tsunematsu T; Department of Oral Molecular Pathology, Tokushima University Graduate School of Biomedical Sciences, Tokushima, Japan.
Horiguchi T; Department of Oral Bioscience, Tokushima University Graduate School of Biomedical Sciences, Tokushima, Japan.
Mouri Y; Department of Oral Bioscience, Tokushima University Graduate School of Biomedical Sciences, Tokushima, Japan.
Shao W; Department of Oral Bioscience, Tokushima University Graduate School of Biomedical Sciences, Tokushima, Japan.
Miyoshi K; Department of Oral Bioscience, Tokushima University Graduate School of Biomedical Sciences, Tokushima, Japan.
Hagita H; Department of Oral Bioscience, Tokushima University Graduate School of Biomedical Sciences, Tokushima, Japan.
Sarubo M; Department of Oral Bioscience, Tokushima University Graduate School of Biomedical Sciences, Tokushima, Japan.
Fujiwara N; Department of Oral Healthcare Management, Tokushima University Graduate School of Biomedical Sciences, Tokushima, Japan.
Qi G; Guangxi Key Laboratory of Tumor Immunology and Microenvironmental Regulation, Guilin Medical University, Guilin, China.
Ishimaru N; Department of Oral Molecular Pathology, Tokushima University Graduate School of Biomedical Sciences, Tokushima, Japan.
Kudo Y; Department of Oral Bioscience, Tokushima University Graduate School of Biomedical Sciences, Tokushima, Japan.
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Źródło:
Cancer medicine [Cancer Med] 2023 Dec; Vol. 12 (24), pp. 22156-22169. Date of Electronic Publication: 2023 Nov 20.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Head and Neck Neoplasms*/genetics
Squamous Cell Carcinoma of Head and Neck*/genetics
Deubiquitinating Enzymes*/metabolism
YAP-Signaling Proteins*/metabolism
Humans ; Cell Line, Tumor ; Ubiquitin Thiolesterase
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
PPARα activation promotes liver progenitor cell-mediated liver regeneration by suppressing YAP signaling in zebrafish.
Autorzy:
Kim M; Department of Developmental Biology, McGowan Institute for Regenerative Medicine, Pittsburgh Liver Research Center, University of Pittsburgh, 3501 5th Ave. #5063, Pittsburgh, PA, 15260, USA.
So J; Department of Developmental Biology, McGowan Institute for Regenerative Medicine, Pittsburgh Liver Research Center, University of Pittsburgh, 3501 5th Ave. #5063, Pittsburgh, PA, 15260, USA.
Shin D; Department of Developmental Biology, McGowan Institute for Regenerative Medicine, Pittsburgh Liver Research Center, University of Pittsburgh, 3501 5th Ave. #5063, Pittsburgh, PA, 15260, USA. .
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Źródło:
Scientific reports [Sci Rep] 2023 Oct 25; Vol. 13 (1), pp. 18312. Date of Electronic Publication: 2023 Oct 25.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Liver Diseases*/metabolism
PPAR alpha*/metabolism
Zebrafish*/genetics
YAP-Signaling Proteins*
Animals ; beta Catenin/metabolism ; Cell Proliferation ; Hepatocytes/metabolism ; Liver/metabolism ; Liver Regeneration/physiology ; Stem Cells/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
A New Player in the Mechanobiology of Deep Fascia: Yes-Associated Protein (YAP).
Autorzy:
Pirri C; Department of Neurosciences, Institute of Human Anatomy, University of Padova, 35121 Padova, Italy.
Caroccia B; Department of Medicine-DIMED, University of Padova, 35128 Padova, Italy.
Angelini A; Department of Orthopedics and Orthopedic Oncology, University of Padova, 35128 Padova, Italy.
Piazza M; Department of Medicine-DIMED, University of Padova, 35128 Padova, Italy.
Petrelli L; Department of Medicine-DIMED, University of Padova, 35128 Padova, Italy.
Caputo I; Department of Medicine-DIMED, University of Padova, 35128 Padova, Italy.
Montemurro C; Independent Researcher, ty Padova, 35120 Padova, Italy.
Ruggieri P; Department of Orthopedics and Orthopedic Oncology, University of Padova, 35128 Padova, Italy.
De Caro R; Department of Neurosciences, Institute of Human Anatomy, University of Padova, 35121 Padova, Italy.
Stecco C; Department of Neurosciences, Institute of Human Anatomy, University of Padova, 35121 Padova, Italy.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Oct 20; Vol. 24 (20). Date of Electronic Publication: 2023 Oct 20.
Typ publikacji:
Journal Article
MeSH Terms:
Mechanotransduction, Cellular*
YAP-Signaling Proteins*
Adult ; Aged ; Female ; Humans ; Male ; Middle Aged ; Fascia/physiology ; Fibroblasts/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Role of Cytokine-Inducible SH2 Domain-Containing (CISH) Protein in the Regulation of Erythropoiesis.
Autorzy:
Maymand S; School of Medicine, Deakin University, Waurn Ponds, Geelong, VIC 3216, Australia.
Lakkavaram AL; School of Medicine, Deakin University, Waurn Ponds, Geelong, VIC 3216, Australia.
Naser W; School of Medicine, Deakin University, Waurn Ponds, Geelong, VIC 3216, Australia.; College of Science, University of Baghdad, Baghdad 10071, Iraq.
Rasighaemi P; School of Medicine, Deakin University, Waurn Ponds, Geelong, VIC 3216, Australia.
Dlugolenski D; School of Medicine, Deakin University, Waurn Ponds, Geelong, VIC 3216, Australia.
Liongue C; School of Medicine, Deakin University, Waurn Ponds, Geelong, VIC 3216, Australia.; Institute for Mental and Physical Health and Clinical Translation, Deakin University, Waurn Ponds, Geelong, VIC 3216, Australia.
Stambas J; School of Medicine, Deakin University, Waurn Ponds, Geelong, VIC 3216, Australia.; Institute for Mental and Physical Health and Clinical Translation, Deakin University, Waurn Ponds, Geelong, VIC 3216, Australia.
de Koning-Ward TF; School of Medicine, Deakin University, Waurn Ponds, Geelong, VIC 3216, Australia.; Institute for Mental and Physical Health and Clinical Translation, Deakin University, Waurn Ponds, Geelong, VIC 3216, Australia.
Ward AC; School of Medicine, Deakin University, Waurn Ponds, Geelong, VIC 3216, Australia.; Institute for Mental and Physical Health and Clinical Translation, Deakin University, Waurn Ponds, Geelong, VIC 3216, Australia.
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Źródło:
Biomolecules [Biomolecules] 2023 Oct 12; Vol. 13 (10). Date of Electronic Publication: 2023 Oct 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Erythropoiesis*/physiology
Suppressor of Cytokine Signaling Proteins*/metabolism
Animals ; Mice ; Anemia/genetics ; Cytokines ; Signal Transduction/physiology ; src Homology Domains
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Releasing YAP dysfunction-caused replicative toxicity rejuvenates mesenchymal stem cells.
Autorzy:
Yu F; State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, West China Hospital of Stomatology, Sichuan University, Chengdu, China.; Department of Endodontics, West China Hospital of Stomatology, Sichuan University, Chengdu, China.
Yao L; State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, West China Hospital of Stomatology, Sichuan University, Chengdu, China.; Department of Endodontics, West China Hospital of Stomatology, Sichuan University, Chengdu, China.
Li F; State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, West China Hospital of Stomatology, Sichuan University, Chengdu, China.
Wang C; State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, West China Hospital of Stomatology, Sichuan University, Chengdu, China.; Department of Endodontics, West China Hospital of Stomatology, Sichuan University, Chengdu, China.
Ye L; State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, West China Hospital of Stomatology, Sichuan University, Chengdu, China.; Department of Endodontics, West China Hospital of Stomatology, Sichuan University, Chengdu, China.
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Źródło:
Aging cell [Aging Cell] 2023 Sep; Vol. 22 (9), pp. e13913. Date of Electronic Publication: 2023 Jun 20.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Mesenchymal Stem Cells*
YAP-Signaling Proteins*
Humans ; Cell Cycle Proteins/genetics ; Genomic Instability ; Phosphorylation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The Role of Cytokine-Inducible SH2 Domain-Containing Protein (CISH) in the Regulation of Basal and Cytokine-Mediated Myelopoiesis.
Autorzy:
Naser W; School of Medicine, Deakin University, Geelong, VIC 3216, Australia.; College of Science, University of Baghdad, Baghdad 10071, Iraq.
Maymand S; School of Medicine, Deakin University, Geelong, VIC 3216, Australia.
Dlugolenski D; School of Medicine, Deakin University, Geelong, VIC 3216, Australia.
Basheer F; School of Medicine, Deakin University, Geelong, VIC 3216, Australia.; Institute for Mental and Physical Health and Clinical Translation, Deakin University, Geelong, VIC 3216, Australia.
Ward AC; School of Medicine, Deakin University, Geelong, VIC 3216, Australia.; Institute for Mental and Physical Health and Clinical Translation, Deakin University, Geelong, VIC 3216, Australia.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Aug 14; Vol. 24 (16). Date of Electronic Publication: 2023 Aug 14.
Typ publikacji:
Journal Article
MeSH Terms:
Cytokines*
Suppressor of Cytokine Signaling Proteins*/metabolism
Animals ; Mice ; Granulocyte-Macrophage Colony-Stimulating Factor/metabolism ; Myelopoiesis ; src Homology Domains
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
On the Value of In Vitro Cell Systems for Mechanobiology from the Perspective of Yes-Associated Protein/Transcriptional Co-Activator with a PDZ-Binding Motif and Focal Adhesion Kinase and Their Involvement in Wound Healing, Cancer, Aging, and Senescence.
Autorzy:
Steinberg T; Center for Dental Medicine, Division of Oral Biotechnology, Medical Center-University of Freiburg, Faculty of Medicine, University of Freiburg, Hugstetterstr. 55, 79106 Freiburg, Germany.
Dieterle MP; Center for Dental Medicine, Division of Oral Biotechnology, Medical Center-University of Freiburg, Faculty of Medicine, University of Freiburg, Hugstetterstr. 55, 79106 Freiburg, Germany.
Ramminger I; Center for Dental Medicine, Division of Oral Biotechnology, Medical Center-University of Freiburg, Faculty of Medicine, University of Freiburg, Hugstetterstr. 55, 79106 Freiburg, Germany.; Faculty of Biology, University of Freiburg, Schaenzlestr. 1, 79104 Freiburg, Germany.
Klein C; Center for Dental Medicine, Division of Oral Biotechnology, Medical Center-University of Freiburg, Faculty of Medicine, University of Freiburg, Hugstetterstr. 55, 79106 Freiburg, Germany.
Brossette J; Center for Dental Medicine, Division of Oral Biotechnology, Medical Center-University of Freiburg, Faculty of Medicine, University of Freiburg, Hugstetterstr. 55, 79106 Freiburg, Germany.; Faculty of Biology, University of Freiburg, Schaenzlestr. 1, 79104 Freiburg, Germany.
Husari A; Center for Dental Medicine, Department of Orthodontics, Medical Center-University of Freiburg, Faculty of Medicine, University of Freiburg, Hugstetterstr. 55, 79106 Freiburg, Germany.
Tomakidi P; Center for Dental Medicine, Division of Oral Biotechnology, Medical Center-University of Freiburg, Faculty of Medicine, University of Freiburg, Hugstetterstr. 55, 79106 Freiburg, Germany.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Aug 11; Vol. 24 (16). Date of Electronic Publication: 2023 Aug 11.
Typ publikacji:
Journal Article; Review
MeSH Terms:
YAP-Signaling Proteins*
Neoplasms*/genetics
Humans ; Focal Adhesion Protein-Tyrosine Kinases ; Mechanotransduction, Cellular ; Transcription Factors ; Wound Healing/genetics ; Biophysics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CIS deletion by CRISPR/Cas9 enhances human primary natural killer cell functions against allogeneic glioblastoma.
Autorzy:
Nakazawa T; Grandsoul Research Institute for Immunology, Inc, 8-1 Matsui, Uda, Nara, 634-8522, Japan. .; Clinic Grandsoul Nara, Uda, Nara, Japan. .; Department of Neurosurgery, Nara Medical University, Kashihara, Nara, Japan. .
Morimoto T; Department of Neurosurgery, Nara Medical University, Kashihara, Nara, Japan.
Maeoka R; Department of Neurosurgery, Nara Medical University, Kashihara, Nara, Japan.
Matsuda R; Department of Neurosurgery, Nara Medical University, Kashihara, Nara, Japan.
Nakamura M; Clinic Grandsoul Nara, Uda, Nara, Japan.; Department of Neurosurgery, Nara Medical University, Kashihara, Nara, Japan.
Nishimura F; Department of Neurosurgery, Nara Medical University, Kashihara, Nara, Japan.
Ouji N; Department of Immunology, Nara Medical University, Kashihara, Nara, 634-8522, Japan.
Yamada S; Department of Neurosurgery, Nara Medical University, Kashihara, Nara, Japan.
Nakagawa I; Department of Neurosurgery, Nara Medical University, Kashihara, Nara, Japan.
Park YS; Department of Neurosurgery, Nara Medical University, Kashihara, Nara, Japan.
Ito T; Department of Immunology, Nara Medical University, Kashihara, Nara, 634-8522, Japan.
Nakase H; Department of Neurosurgery, Nara Medical University, Kashihara, Nara, Japan.
Tsujimura T; Grandsoul Research Institute for Immunology, Inc, 8-1 Matsui, Uda, Nara, 634-8522, Japan.; Clinic Grandsoul Nara, Uda, Nara, Japan.
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Źródło:
Journal of experimental & clinical cancer research : CR [J Exp Clin Cancer Res] 2023 Aug 10; Vol. 42 (1), pp. 205. Date of Electronic Publication: 2023 Aug 10.
Typ publikacji:
Journal Article
MeSH Terms:
Brain Neoplasms*/genetics
Brain Neoplasms*/therapy
Carcinoma, Intraductal, Noninfiltrating*
Glioblastoma*/genetics
Glioblastoma*/therapy
Hematopoietic Stem Cell Transplantation*
Suppressor of Cytokine Signaling Proteins*/genetics
Animals ; Humans ; Mice ; CRISPR-Cas Systems ; Killer Cells, Natural
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
OTUB1 inhibits breast cancer by non-canonically stabilizing CCN6.
Autorzy:
Zhao Y; Chemical Biology Research Center, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou, China.; Oujiang Laboratory (Zhejiang Lab for Regenerative Medicine, Vision and Brain Health), Department of Neurological Rehabilitation, The Second Affiliated Hospital and Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, China.
Ruan J; Department of Pathology, The First Affiliated Hospital, Wenzhou Medical University, Wenzhou, China.
Li Z; Chemical Biology Research Center, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou, China.
Su X; Chemical Biology Research Center, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou, China.
Chen K; Chemical Biology Research Center, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou, China.
Lin Y; Chemical Biology Research Center, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou, China.
Cai Y; School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou, China.
Wang P; School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou, China.
Liu B; Oujiang Laboratory (Zhejiang Lab for Regenerative Medicine, Vision and Brain Health), Department of Neurological Rehabilitation, The Second Affiliated Hospital and Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, China.
Schlüter D; Institute of Medical Microbiology and Hospital Epidemiology, Hannover Medical School, Hannover, Germany.; Cluster of Excellence RESIST (EXC 2155), Hannover Medical School, Hannover, Germany.
Liang G; Chemical Biology Research Center, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou, China.; School of Pharmaceutical Sciences, Hangzhou Medical College, Hangzhou, China.
Wang X; Chemical Biology Research Center, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou, China.; Oujiang Laboratory (Zhejiang Lab for Regenerative Medicine, Vision and Brain Health), Department of Neurological Rehabilitation, The Second Affiliated Hospital and Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, China.; Institute of Medical Microbiology and Hospital Epidemiology, Hannover Medical School, Hannover, Germany.
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Źródło:
Clinical and translational medicine [Clin Transl Med] 2023 Aug; Vol. 13 (8), pp. e1385.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Carcinogenesis*
Cell Transformation, Neoplastic*
Deubiquitinating Enzymes*/metabolism
CCN Intercellular Signaling Proteins*/metabolism
Humans ; Blotting, Western ; Cytoplasm ; Proteasome Endopeptidase Complex
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Inhibition of TANK-binding kinase1 attenuates the astrocyte-mediated neuroinflammatory response through YAP signaling after spinal cord injury.
Autorzy:
Zhang W; Department of Orthopedics (Spine Surgery), The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.; School of Pharmacy, and Department of Neurosurgery, The Affiliated Hospital, Hangzhou Normal University, Hangzhou, Zhejiang, China.
Jia M; Department of Orthopedics (Spine Surgery), The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.; School of Pharmacy, and Department of Neurosurgery, The Affiliated Hospital, Hangzhou Normal University, Hangzhou, Zhejiang, China.
Lian J; Department of Orthopedics (Spine Surgery), The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.; School of Pharmacy, and Department of Neurosurgery, The Affiliated Hospital, Hangzhou Normal University, Hangzhou, Zhejiang, China.
Lu S; Department of Orthopedics (Spine Surgery), The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.
Zhou J; Department of Orthopedics (Spine Surgery), The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.; School of Pharmacy, and Department of Neurosurgery, The Affiliated Hospital, Hangzhou Normal University, Hangzhou, Zhejiang, China.
Fan Z; Department of Orthopedics (Spine Surgery), The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.
Zhu Z; Department of Orthopedics (Spine Surgery), The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.
He Y; Department of Orthopedics (Spine Surgery), The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.
Huang C; School of Pharmacy, and Department of Neurosurgery, The Affiliated Hospital, Hangzhou Normal University, Hangzhou, Zhejiang, China.
Zhu M; Department of Orthopedics (Spine Surgery), The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.
Wang J; Center for Wound Repair and Regeneration, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.
Wang Y; Department of Clinical Research Center, Affiliated Hangzhou First People's Hospital, Zhejiang University School of Medicine, Hangzhou, Zhejiang, China.
Huang Z; Department of Orthopedics (Spine Surgery), The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.; School of Pharmacy, and Department of Neurosurgery, The Affiliated Hospital, Hangzhou Normal University, Hangzhou, Zhejiang, China.
Teng H; Department of Orthopedics (Spine Surgery), The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.; Center for Wound Repair and Regeneration, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2023 Aug; Vol. 29 (8), pp. 2206-2222. Date of Electronic Publication: 2023 Apr 10.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
NF-kappa B*/metabolism
Spinal Cord Injuries*/pathology
Protein Serine-Threonine Kinases*/metabolism
YAP-Signaling Proteins*
Astrocytes/metabolism ; Inflammation/metabolism ; Lipopolysaccharides/toxicity ; Signal Transduction ; Spinal Cord/metabolism ; Animals
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The oncogenic role of NF1 in gallbladder cancer through regulation of YAP1 stability by direct interaction with YAP1.
Autorzy:
Zhang L; Department of General Surgery, Shanghai Key Laboratory of Biliary Tract Disease Research, Xinhua Hospital, Shanghai Jiao Tong University, Shanghai, 200092, China.; Department of Biliary-Pancreatic Surgery, State Key Laboratory of Oncogenes and Related Genes, Renji Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, 200127, China.
Jiang L; Department of General Surgery, Shanghai Key Laboratory of Biliary Tract Disease Research, Xinhua Hospital, Shanghai Jiao Tong University, Shanghai, 200092, China.; School of Health Science and Engineering, University of Shanghai for Science and Technology, Shanghai, 200093, China.
Zeng L; Department of General Surgery, Shanghai Key Laboratory of Biliary Tract Disease Research, Xinhua Hospital, Shanghai Jiao Tong University, Shanghai, 200092, China.
Jin Z; Department of General Surgery, Shanghai Key Laboratory of Biliary Tract Disease Research, Xinhua Hospital, Shanghai Jiao Tong University, Shanghai, 200092, China.
Dong X; Department of General Surgery, Shanghai Key Laboratory of Biliary Tract Disease Research, Xinhua Hospital, Shanghai Jiao Tong University, Shanghai, 200092, China.
Zhang Y; Department of General Surgery, Shanghai Key Laboratory of Biliary Tract Disease Research, Xinhua Hospital, Shanghai Jiao Tong University, Shanghai, 200092, China.
Chen L; Department of General Surgery, Shanghai Key Laboratory of Biliary Tract Disease Research, Xinhua Hospital, Shanghai Jiao Tong University, Shanghai, 200092, China.
Shu Y; Department of General Surgery, Shanghai Key Laboratory of Biliary Tract Disease Research, Xinhua Hospital, Shanghai Jiao Tong University, Shanghai, 200092, China. .
Liu Y; Department of Biliary-Pancreatic Surgery, State Key Laboratory of Oncogenes and Related Genes, Renji Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, 200127, China. .
Huang Y; Department of General Surgery, Shanghai Key Laboratory of Biliary Tract Disease Research, Xinhua Hospital, Shanghai Jiao Tong University, Shanghai, 200092, China. .
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Źródło:
Journal of translational medicine [J Transl Med] 2023 May 05; Vol. 21 (1), pp. 306. Date of Electronic Publication: 2023 May 05.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gallbladder Neoplasms*/genetics
Neurofibromin 1*/genetics
Neurofibromin 1*/metabolism
YAP-Signaling Proteins*/genetics
YAP-Signaling Proteins*/metabolism
Animals ; Mice ; Cell Line, Tumor ; Cell Proliferation ; Gene Expression Regulation, Neoplastic ; Mice, Nude ; RNA, Small Interfering ; Signal Transduction ; Humans
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
YES-associated protein-regulated Smad7 worsen epithelial barrier injury of chronic sinusitis with nasal polyps.
Autorzy:
Jiang X; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
Shu L; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
Liu Y; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
Shen Y; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
Ke X; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
Liu J; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
Yang Y; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
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Źródło:
Immunity, inflammation and disease [Immun Inflamm Dis] 2023 Jun; Vol. 11 (6), pp. e907.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Nasal Polyps*
Sinusitis*
YAP-Signaling Proteins*/genetics
Smad7 Protein*/genetics
Humans ; Cadherins ; Chronic Disease ; Transcription Factors/genetics ; Transforming Growth Factor beta1
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
New insights into the ambivalent role of YAP/TAZ in human cancers.
Autorzy:
Luo J; Scientific Research Center, The Seventh Affiliated Hospital of Sun Yat-sen University, No. 628 Zhenyuan Road, Shenzhen, 518107, Guangdong, People's Republic of China.
Deng L; Department of General Surgery, The Seventh Affiliated Hospital of Sun Yat-sen University, No. 628 Zhenyuan Road, Shenzhen, 518107, Guangdong, People's Republic of China.
Zou H; Scientific Research Center, The Seventh Affiliated Hospital of Sun Yat-sen University, No. 628 Zhenyuan Road, Shenzhen, 518107, Guangdong, People's Republic of China.
Guo Y; Scientific Research Center, The Seventh Affiliated Hospital of Sun Yat-sen University, No. 628 Zhenyuan Road, Shenzhen, 518107, Guangdong, People's Republic of China.
Tong T; Scientific Research Center, The Seventh Affiliated Hospital of Sun Yat-sen University, No. 628 Zhenyuan Road, Shenzhen, 518107, Guangdong, People's Republic of China.; Department of Urology, Pelvic Floor Disorders Center, The Seventh Affiliated Hospital of Sun Yat-sen University, No. 628 Zhenyuan Road, Shenzhen, 518107, Guangdong, People's Republic of China.
Huang M; Department of General Surgery, The Seventh Affiliated Hospital of Sun Yat-sen University, No. 628 Zhenyuan Road, Shenzhen, 518107, Guangdong, People's Republic of China.
Ling G; Department of Neurosurgery, The Seventh Affiliated Hospital of Sun Yat-sen University, No. 628 Zhenyuan Road, Shenzhen, 518107, Guangdong, People's Republic of China.
Li P; Scientific Research Center, The Seventh Affiliated Hospital of Sun Yat-sen University, No. 628 Zhenyuan Road, Shenzhen, 518107, Guangdong, People's Republic of China. .; Guangdong Provincial Key Laboratory of Digestive Cancer Research, The Seventh Affiliated Hospital of Sun Yat-sen University, No. 628 Zhenyuan Road, Shenzhen, 518107, Guangdong, People's Republic of China. .
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Źródło:
Journal of experimental & clinical cancer research : CR [J Exp Clin Cancer Res] 2023 May 22; Vol. 42 (1), pp. 130. Date of Electronic Publication: 2023 May 22.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Adaptor Proteins, Signal Transducing*/genetics
Adaptor Proteins, Signal Transducing*/metabolism
Neoplasms*/pathology
Transcriptional Coactivator with PDZ-Binding Motif Proteins*
YAP-Signaling Proteins*
Humans ; Phosphoproteins/metabolism ; Protein Serine-Threonine Kinases/metabolism ; Signal Transduction ; Trans-Activators/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Inflammatory Skin Diseases: Focus on the Role of Suppressors of Cytokine Signaling (SOCS) Proteins.
Autorzy:
Cianciulli A; Department of Biosciences, Biotechnologies and Environment, University of Bari, I-70125 Bari, Italy.
Calvello R; Department of Biosciences, Biotechnologies and Environment, University of Bari, I-70125 Bari, Italy.
Porro C; Department of Clinical and Experimental Medicine, University of Foggia, I-71100 Foggia, Italy.
Lofrumento DD; Department of Biological and Environmental Sciences and Technologies, Section of Human Anatomy, University of Salento, I-73100 Lecce, Italy.
Panaro MA; Department of Biosciences, Biotechnologies and Environment, University of Bari, I-70125 Bari, Italy.
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Źródło:
Cells [Cells] 2024 Mar 13; Vol. 13 (6). Date of Electronic Publication: 2024 Mar 13.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Dermatitis, Atopic*
Psoriasis*
Humans ; Suppressor of Cytokine Signaling Proteins/genetics ; Suppressor of Cytokine Signaling Proteins/metabolism ; Signal Transduction/genetics ; Cytokines/metabolism ; Inflammation
Czasopismo naukowe
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Tytuł:
CSN6‐SPOP‐HMGCS1 Axis Promotes Hepatocellular Carcinoma Progression via YAP1 Activation.
Autorzy:
Li, Kai (AUTHOR)
Zhang, Jiayu (AUTHOR)
Lyu, Haiwen (AUTHOR)
Yang, Jinneng (AUTHOR)
Wei, Wenxia (AUTHOR)
Wang, Yuzhi (AUTHOR)
Luo, Haidan (AUTHOR)
Zhang, Yijing (AUTHOR)
Jiang, Xin (AUTHOR)
Yi, Hairong (AUTHOR)
Wang, Mengan (AUTHOR)
Zhang, Caiyun (AUTHOR)
Wu, Kang (AUTHOR)
Xiao, Lishi (AUTHOR)
Wen, Weijie (AUTHOR)
Xu, Hui (AUTHOR)
Li, Guolin (AUTHOR)
Wan, Yunle (AUTHOR)
Yang, Fang (AUTHOR)
Yang, Runxiang (AUTHOR)
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Źródło:
Advanced Science. 4/10/2024, Vol. 11 Issue 14, p1-17. 17p.
Czasopismo naukowe
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Tytuł:
External Mechanical Stability Regulates Hematoma Vascularization in Bone Healing Rather than Endothelial YAP/TAZ Mechanotransduction.
Autorzy:
Mehl, Julia (AUTHOR)
Farahani, Saeed Khomeijani (AUTHOR)
Brauer, Erik (AUTHOR)
Klaus‐Bergmann, Alexandra (AUTHOR)
Thiele, Tobias (AUTHOR)
Ellinghaus, Agnes (AUTHOR)
Bartels‐Klein, Eireen (AUTHOR)
Koch, Katharina (AUTHOR)
Schmidt‐Bleek, Katharina (AUTHOR)
Petersen, Ansgar (AUTHOR)
Gerhardt, Holger (AUTHOR)
Vogel, Viola (AUTHOR)
Duda, Georg N. (AUTHOR)
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Źródło:
Advanced Science. 4/3/2024, Vol. 11 Issue 13, p1-18. 18p.
Czasopismo naukowe
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Tytuł:
SUMOylation of TEAD1 Modulates the Mechanism of Pathological Cardiac Hypertrophy.
Autorzy:
Shi, Xin (AUTHOR)
Dang, Xuening (AUTHOR)
Huang, Zhenyu (AUTHOR)
Lu, Yanqiao (AUTHOR)
Tong, Huan (AUTHOR)
Liang, Feng (AUTHOR)
Zhuang, Fei (AUTHOR)
Li, Yi (AUTHOR)
Cai, Zhaohua (AUTHOR)
Huo, Huanhuan (AUTHOR)
Jiang, Zhaolei (AUTHOR)
Pan, Changqing (AUTHOR)
Wang, Xia (AUTHOR)
Gu, Chang (AUTHOR)
He, Ben (AUTHOR)
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Źródło:
Advanced Science. 3/27/2024, Vol. 11 Issue 12, p1-18. 18p.
Czasopismo naukowe
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Wyświetlanie 1-20 z 6374

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