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Tytuł :
Nicotine facilitates pancreatic fibrosis by promoting activation of pancreatic stellate cells via α7nAChR-mediated JAK2/STAT3 signaling pathway in rats.
Autorzy :
Li Z; Department of Gastroenterology, Beijing Chao-Yang Hospital, Capital Medical University, Beijing, 100020, China.
Lu D; Department of Gastroenterology, Beijing Chao-Yang Hospital, Capital Medical University, Beijing, 100020, China.
Jin T; Department of Gastroenterology, Beijing Chao-Yang Hospital, Capital Medical University, Beijing, 100020, China.
Liu X; Department of Gastroenterology, Beijing Chao-Yang Hospital, Capital Medical University, Beijing, 100020, China.
Hao J; Department of Gastroenterology, Beijing Chao-Yang Hospital, Capital Medical University, Beijing, 100020, China. Electronic address: .
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Źródło :
Toxicology letters [Toxicol Lett] 2021 Oct 01; Vol. 349, pp. 84-91. Date of Electronic Publication: 2021 Jun 18.
Typ publikacji :
Journal Article
MeSH Terms :
Janus Kinase 2/*metabolism
Nicotine/*toxicity
Nicotinic Agonists/*toxicity
Pancreatic Stellate Cells/*drug effects
Pancreatitis, Chronic/*chemically induced
STAT3 Transcription Factor/*metabolism
alpha7 Nicotinic Acetylcholine Receptor/*agonists
Animals ; Apoptosis/drug effects ; Cell Proliferation/drug effects ; Cells, Cultured ; Extracellular Matrix/drug effects ; Extracellular Matrix/metabolism ; Extracellular Matrix/pathology ; Fibrosis ; Male ; Pancreatic Stellate Cells/enzymology ; Pancreatic Stellate Cells/pathology ; Pancreatitis, Chronic/enzymology ; Pancreatitis, Chronic/pathology ; Rats, Wistar ; Signal Transduction ; alpha7 Nicotinic Acetylcholine Receptor/metabolism
Czasopismo naukowe
Tytuł :
Relaxin in hepatic fibrosis: What is known and where to head?
Autorzy :
Ezhilarasan D; Department of Pharmacology, The Blue Lab, Molecular Pharmacology and Toxicology Division, Saveetha Dental College, Saveetha Institute of Medical and Technical Sciences (SIMATS), Chennai, Tamil Nadu, 600 077, India. Electronic address: .
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Źródło :
Biochimie [Biochimie] 2021 Aug; Vol. 187, pp. 144-151. Date of Electronic Publication: 2021 Jun 05.
Typ publikacji :
Journal Article; Review
MeSH Terms :
Signal Transduction*
Hepatic Stellate Cells/*metabolism
Liver Cirrhosis/*metabolism
Relaxin/*metabolism
Animals ; Collagen/biosynthesis ; Extracellular Matrix/metabolism ; Hepatic Stellate Cells/pathology ; Humans ; Hypertension, Portal/metabolism ; Hypertension, Portal/pathology ; Hypertension, Portal/therapy ; Liver Cirrhosis/pathology ; Liver Cirrhosis/therapy ; Receptors, G-Protein-Coupled/metabolism ; Receptors, Peptide/metabolism ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Tytuł :
[Endoplasmic reticulum stress in hepatic stellate cells induced by tunicamycin promotes apoptosis and cell cycle arrest].
Autorzy :
Zhang J; Guizhou Provincial Key Laboratory of Pathogensis & Drug Research for Common Chronic Diseases, Department of Pathophysiology, College of Basic Medicine, Guizhou Medical University, Guiyang 550000, China.
Yang L; Guizhou Provincial Key Laboratory of Pathogensis & Drug Research for Common Chronic Diseases, Department of Pathophysiology, College of Basic Medicine, Guizhou Medical University, Guiyang 550000, China.
Han X; Guizhou Provincial Key Laboratory of Pathogensis & Drug Research for Common Chronic Diseases, Department of Pathophysiology, College of Basic Medicine, Guizhou Medical University, Guiyang 550000, China.
Li C; Guizhou Provincial Key Laboratory of Pathogensis & Drug Research for Common Chronic Diseases, Department of Pathophysiology, College of Basic Medicine, Guizhou Medical University, Guiyang 550000, China.
Liu R; Guizhou Provincial Key Laboratory of Pathogensis & Drug Research for Common Chronic Diseases, Department of Pathophysiology, College of Basic Medicine, Guizhou Medical University, Guiyang 550000, China.
Ma Z; Guizhou Provincial Key Laboratory of Pathogensis & Drug Research for Common Chronic Diseases, Department of Pathophysiology, College of Basic Medicine, Guizhou Medical University, Guiyang 550000, China.
Han B; Guizhou Provincial Key Laboratory of Pathogensis & Drug Research for Common Chronic Diseases, Department of Pathophysiology, College of Basic Medicine, Guizhou Medical University, Guiyang 550000, China.
Xie R; Guizhou Provincial Key Laboratory of Pathogensis & Drug Research for Common Chronic Diseases, Department of Pathophysiology, College of Basic Medicine, Guizhou Medical University, Guiyang 550000, China.
Yang Q; Guizhou Provincial Key Laboratory of Pathogensis & Drug Research for Common Chronic Diseases, Department of Pathophysiology, College of Basic Medicine, Guizhou Medical University, Guiyang 550000, China. *Corresponding author, E-mail: .
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Źródło :
Xi bao yu fen zi mian yi xue za zhi = Chinese journal of cellular and molecular immunology [Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi] 2021 Sep; Vol. 37 (9), pp. 794-800.
Typ publikacji :
Journal Article
MeSH Terms :
Endoplasmic Reticulum Stress*
Hepatic Stellate Cells*
Animals ; Apoptosis ; Cell Cycle Checkpoints ; Rats ; Tunicamycin/pharmacology
Czasopismo naukowe
Tytuł :
Physalin B attenuates liver fibrosis via suppressing LAP2α-HDAC1-mediated deacetylation of the transcription factor GLI1 and hepatic stellate cell activation.
Autorzy :
Zhu X; State Key Laboratory of Natural Medicines and Jiangsu Key Laboratory of Bioactive Natural Product Research, School of Traditional Chinese Pharmacy, China Pharmaceutical University, Nanjing, China.
Ye S; State Key Laboratory of Natural Medicines and Jiangsu Key Laboratory of Bioactive Natural Product Research, School of Traditional Chinese Pharmacy, China Pharmaceutical University, Nanjing, China.
Yu D; Personalized Drug Therapy Key Laboratory of Sichuan Province, Department of Pharmacy, Sichuan Provincial People's Hospital, School of Medicine, University of Electronic Science and Technology of China, Chengdu, China.
Zhang Y; State Key Laboratory of Natural Medicines and Jiangsu Key Laboratory of Bioactive Natural Product Research, School of Traditional Chinese Pharmacy, China Pharmaceutical University, Nanjing, China.
Li J; State Key Laboratory of Natural Medicines and Jiangsu Key Laboratory of Bioactive Natural Product Research, School of Traditional Chinese Pharmacy, China Pharmaceutical University, Nanjing, China.
Zhang M; State Key Laboratory of Natural Medicines and Jiangsu Key Laboratory of Bioactive Natural Product Research, School of Traditional Chinese Pharmacy, China Pharmaceutical University, Nanjing, China.
Leng Y; State Key Laboratory of Natural Medicines and Jiangsu Key Laboratory of Bioactive Natural Product Research, School of Traditional Chinese Pharmacy, China Pharmaceutical University, Nanjing, China.
Yang T; State Key Laboratory of Natural Medicines and Jiangsu Key Laboratory of Bioactive Natural Product Research, School of Traditional Chinese Pharmacy, China Pharmaceutical University, Nanjing, China.
Luo J; State Key Laboratory of Natural Medicines and Jiangsu Key Laboratory of Bioactive Natural Product Research, School of Traditional Chinese Pharmacy, China Pharmaceutical University, Nanjing, China.
Chen X; State Key Laboratory of Natural Medicines and Jiangsu Key Laboratory of Bioactive Natural Product Research, School of Traditional Chinese Pharmacy, China Pharmaceutical University, Nanjing, China.
Zhang H; State Key Laboratory of Natural Medicines and Jiangsu Key Laboratory of Bioactive Natural Product Research, School of Traditional Chinese Pharmacy, China Pharmaceutical University, Nanjing, China.
Kong L; State Key Laboratory of Natural Medicines and Jiangsu Key Laboratory of Bioactive Natural Product Research, School of Traditional Chinese Pharmacy, China Pharmaceutical University, Nanjing, China.
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Źródło :
British journal of pharmacology [Br J Pharmacol] 2021 Sep; Vol. 178 (17), pp. 3428-3447. Date of Electronic Publication: 2021 May 21.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Hepatic Stellate Cells*
Transcription Factors*
Animals ; Carbon Tetrachloride ; Hedgehog Proteins ; Histone Deacetylase 1 ; Liver/pathology ; Liver Cirrhosis/drug therapy ; Liver Cirrhosis/pathology ; Mice ; Secosteroids ; Zinc Finger Protein GLI1
Czasopismo naukowe
Tytuł :
Inhibiting xCT/SLC7A11 induces ferroptosis of myofibroblastic hepatic stellate cells but exacerbates chronic liver injury.
Autorzy :
Du K; Department of Medicine, Duke University, Durham, NC, USA.
Oh SH; Department of Medicine, Duke University, Durham, NC, USA.
Dutta RK; Department of Medicine, Duke University, Durham, NC, USA.
Sun T; Department of Molecular Genetics and Microbiology, Duke University, Durham, NC, USA.; Center for Genomic and Computational Biology, Duke University, Durham, NC, USA.
Yang WH; Department of Molecular Genetics and Microbiology, Duke University, Durham, NC, USA.; Center for Genomic and Computational Biology, Duke University, Durham, NC, USA.
Chi JT; Department of Molecular Genetics and Microbiology, Duke University, Durham, NC, USA.; Center for Genomic and Computational Biology, Duke University, Durham, NC, USA.
Diehl AM; Department of Medicine, Duke University, Durham, NC, USA.
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Źródło :
Liver international : official journal of the International Association for the Study of the Liver [Liver Int] 2021 Sep; Vol. 41 (9), pp. 2214-2227. Date of Electronic Publication: 2021 Jul 04.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Ferroptosis*
Hepatic Stellate Cells*/pathology
Animals ; Hepatocytes ; Liver/pathology ; Liver Cirrhosis/chemically induced ; Liver Cirrhosis/pathology ; Mice
Czasopismo naukowe
Tytuł :
Hepatic stellate cell-derived exosomes modulate macrophage inflammatory response.
Autorzy :
Benbow JH; Liver Pathobiology Laboratory, Department of Internal Medicine, Carolinas Medical Center, Atrium Health, Charlotte, NC, 28203, USA.
Marrero E; Department of Surgery, Carolinas Medical Center, Atrium Health, Charlotte, NC, 28203, USA.
McGee RM; Liver Pathobiology Laboratory, Department of Internal Medicine, Carolinas Medical Center, Atrium Health, Charlotte, NC, 28203, USA.
Brandon-Warner E; Liver Pathobiology Laboratory, Department of Internal Medicine, Carolinas Medical Center, Atrium Health, Charlotte, NC, 28203, USA.
Attal N; Department of Surgery, Carolinas Medical Center, Atrium Health, Charlotte, NC, 28203, USA.
Feilen NA; Liver Pathobiology Laboratory, Department of Internal Medicine, Carolinas Medical Center, Atrium Health, Charlotte, NC, 28203, USA.
Culberson CR; Liver Pathobiology Laboratory, Department of Internal Medicine, Carolinas Medical Center, Atrium Health, Charlotte, NC, 28203, USA.
McKillop IH; Department of Surgery, Carolinas Medical Center, Atrium Health, Charlotte, NC, 28203, USA. Electronic address: .
Schrum LW; Liver Pathobiology Laboratory, Department of Internal Medicine, Carolinas Medical Center, Atrium Health, Charlotte, NC, 28203, USA.
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Źródło :
Experimental cell research [Exp Cell Res] 2021 Aug 01; Vol. 405 (1), pp. 112663. Date of Electronic Publication: 2021 May 26.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Exosomes/*physiology
Hepatic Stellate Cells/*physiology
Inflammation/*immunology
Macrophages/*immunology
Animals ; Cells, Cultured ; Cytokines/metabolism ; Hepatic Stellate Cells/cytology ; Humans ; Inflammation/metabolism ; Inflammation/pathology ; Macrophages/metabolism ; Male ; Mice ; Mice, Inbred C57BL ; MicroRNAs/genetics ; Rats ; Rats, Sprague-Dawley ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Tytuł :
Immune-regulated IDO1-dependent tryptophan metabolism is source of one-carbon units for pancreatic cancer and stellate cells.
Autorzy :
Newman AC; Institute of Cancer Sciences, Wolfson Wohl Cancer Research Centre, University of Glasgow, Switchback Road, Glasgow G61 1QH, UK.
Falcone M; Institute of Cancer Sciences, Wolfson Wohl Cancer Research Centre, University of Glasgow, Switchback Road, Glasgow G61 1QH, UK.
Huerta Uribe A; Institute of Cancer Sciences, Wolfson Wohl Cancer Research Centre, University of Glasgow, Switchback Road, Glasgow G61 1QH, UK.
Zhang T; Institute of Cancer Sciences, Wolfson Wohl Cancer Research Centre, University of Glasgow, Switchback Road, Glasgow G61 1QH, UK.
Athineos D; Cancer Research UK Beatson Institute, Switchback Road, Glasgow G61 1BD, UK.
Pietzke M; Cancer Research UK Beatson Institute, Switchback Road, Glasgow G61 1BD, UK.
Vazquez A; Institute of Cancer Sciences, Wolfson Wohl Cancer Research Centre, University of Glasgow, Switchback Road, Glasgow G61 1QH, UK; Cancer Research UK Beatson Institute, Switchback Road, Glasgow G61 1BD, UK.
Blyth K; Institute of Cancer Sciences, Wolfson Wohl Cancer Research Centre, University of Glasgow, Switchback Road, Glasgow G61 1QH, UK; Cancer Research UK Beatson Institute, Switchback Road, Glasgow G61 1BD, UK.
Maddocks ODK; Institute of Cancer Sciences, Wolfson Wohl Cancer Research Centre, University of Glasgow, Switchback Road, Glasgow G61 1QH, UK. Electronic address: .
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Źródło :
Molecular cell [Mol Cell] 2021 Jun 03; Vol. 81 (11), pp. 2290-2302.e7. Date of Electronic Publication: 2021 Apr 07.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Carcinoma, Pancreatic Ductal/*genetics
Indoleamine-Pyrrole 2,3,-Dioxygenase/*genetics
Pancreatic Neoplasms/*genetics
Pancreatic Stellate Cells/*metabolism
Tumor Escape/*drug effects
Allografts ; Animals ; Antineoplastic Agents/pharmacology ; Carbon/immunology ; Carbon/metabolism ; Carcinoma, Pancreatic Ductal/drug therapy ; Carcinoma, Pancreatic Ductal/immunology ; Carcinoma, Pancreatic Ductal/mortality ; Cell Line, Tumor ; Formates/immunology ; Formates/metabolism ; Gene Expression Regulation, Neoplastic ; Humans ; Indoleamine-Pyrrole 2,3,-Dioxygenase/immunology ; Interferon-gamma/genetics ; Interferon-gamma/immunology ; Metabolic Networks and Pathways/drug effects ; Metabolic Networks and Pathways/genetics ; Mice ; Mice, Inbred C57BL ; Mice, Nude ; Oximes/pharmacology ; Pancreatic Neoplasms/drug therapy ; Pancreatic Neoplasms/immunology ; Pancreatic Neoplasms/mortality ; Pancreatic Stellate Cells/drug effects ; Pancreatic Stellate Cells/immunology ; Proto-Oncogene Proteins p21(ras)/genetics ; Proto-Oncogene Proteins p21(ras)/immunology ; Serine/immunology ; Serine/metabolism ; Serine/pharmacology ; Signal Transduction ; Sulfonamides/pharmacology ; Tryptophan/immunology ; Tryptophan/metabolism ; Tryptophan/pharmacology ; Tumor Suppressor Protein p53/genetics ; Tumor Suppressor Protein p53/immunology
Czasopismo naukowe
Tytuł :
Carvacrol alleviates liver fibrosis by inhibiting TRPM7 and modulating the MAPK signaling pathway.
Autorzy :
Cai S; Inflammation and Immune Mediated Diseases Laboratory of Anhui, Province, Anhui Institute of Innovative Drugs, School of Pharmacy, Anhui Medical University, China; The Key Laboratory of Anti-inflammatory and Immune Medicines, Ministry of Education, Hefei, China; Institute for Liver Diseases of Anhui Medical University, Hefei, China.
Wu L; Inflammation and Immune Mediated Diseases Laboratory of Anhui, Province, Anhui Institute of Innovative Drugs, School of Pharmacy, Anhui Medical University, China; The Key Laboratory of Anti-inflammatory and Immune Medicines, Ministry of Education, Hefei, China; Institute for Liver Diseases of Anhui Medical University, Hefei, China.
Yuan S; Department of Pharmacy, The First Affiliated Hospital of Anhui Medical University, Hefei, Anhui, China.
Liu G; Department of Pharmacy, The First Affiliated Hospital of Anhui Medical University, Hefei, Anhui, China.
Wang Y; Inflammation and Immune Mediated Diseases Laboratory of Anhui, Province, Anhui Institute of Innovative Drugs, School of Pharmacy, Anhui Medical University, China; The Key Laboratory of Anti-inflammatory and Immune Medicines, Ministry of Education, Hefei, China; Institute for Liver Diseases of Anhui Medical University, Hefei, China.
Fang L; Department of Pharmacy, The First Affiliated Hospital of Anhui Medical University, Hefei, Anhui, China. Electronic address: .
Xu D; Inflammation and Immune Mediated Diseases Laboratory of Anhui, Province, Anhui Institute of Innovative Drugs, School of Pharmacy, Anhui Medical University, China; The Key Laboratory of Anti-inflammatory and Immune Medicines, Ministry of Education, Hefei, China; Institute for Liver Diseases of Anhui Medical University, Hefei, China; Department of Pharmacy, The First Affiliated Hospital of Anhui Medical University, Hefei, Anhui, China. Electronic address: .
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Źródło :
European journal of pharmacology [Eur J Pharmacol] 2021 May 05; Vol. 898, pp. 173982. Date of Electronic Publication: 2021 Feb 26.
Typ publikacji :
Journal Article
MeSH Terms :
Chemical and Drug Induced Liver Injury/*prevention & control
Cymenes/*pharmacology
Hepatic Stellate Cells/*drug effects
Liver/*drug effects
Liver Cirrhosis, Experimental/*prevention & control
Mitogen-Activated Protein Kinases/*metabolism
TRPM Cation Channels/*antagonists & inhibitors
Animals ; Becaplermin/pharmacology ; Carbon Tetrachloride ; Cell Line ; Cell Proliferation/drug effects ; Chemical and Drug Induced Liver Injury/enzymology ; Chemical and Drug Induced Liver Injury/etiology ; Chemical and Drug Induced Liver Injury/pathology ; Collagen/metabolism ; Hepatic Stellate Cells/enzymology ; Hepatic Stellate Cells/pathology ; Liver/enzymology ; Liver/pathology ; Liver Cirrhosis, Experimental/chemically induced ; Liver Cirrhosis, Experimental/enzymology ; Liver Cirrhosis, Experimental/pathology ; Male ; Mice, Inbred C57BL ; Rats ; Signal Transduction ; TRPM Cation Channels/metabolism
Czasopismo naukowe
Tytuł :
The miR-139-5p/peripheral myelin protein 22 axis modulates TGF-β-induced hepatic stellate cell activation and CCl 4 -induced hepatic fibrosis in mice.
Autorzy :
He C; Department of General Surgery, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China.
Shu B; Department of General Surgery, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China.
Zhou Y; Department of Surgical Operation, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China.
Zhang R; Department of General Surgery, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China.
Yang X; Department of General Surgery, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2021 Jul 01; Vol. 276, pp. 119294. Date of Electronic Publication: 2021 Mar 03.
Typ publikacji :
Journal Article
MeSH Terms :
Gene Expression Regulation/*drug effects
Hepatic Stellate Cells/*pathology
Liver Cirrhosis/*pathology
MicroRNAs/*genetics
Myelin Proteins/*metabolism
Transforming Growth Factor beta/*pharmacology
3' Untranslated Regions ; Animals ; Cell Proliferation ; Hepatic Stellate Cells/drug effects ; Hepatic Stellate Cells/metabolism ; Humans ; Liver Cirrhosis/chemically induced ; Liver Cirrhosis/metabolism ; Mice ; Mice, Inbred C57BL ; Myelin Proteins/genetics ; Signal Transduction
Czasopismo naukowe
Tytuł :
Fasudil prevents liver fibrosis via activating natural killer cells and suppressing hepatic stellate cells.
Autorzy :
Han QJ; Institute of Immunopharmaceutical Sciences, School of Pharmaceutical Sciences, Shandong University, Jinan 250012, Shandong Province, China.
Mu YL; Institute of Immunopharmaceutical Sciences, School of Pharmaceutical Sciences, Shandong University, Jinan 250012, Shandong Province, China.
Zhao HJ; Institute of Immunopharmaceutical Sciences, School of Pharmaceutical Sciences, Shandong University, Jinan 250012, Shandong Province, China.
Zhao RR; Institute of Immunopharmaceutical Sciences, School of Pharmaceutical Sciences, Shandong University, Jinan 250012, Shandong Province, China.
Guo QJ; Institute of Immunopharmaceutical Sciences, School of Pharmaceutical Sciences, Shandong University, Jinan 250012, Shandong Province, China.
Su YH; Department of Emergency Surgery, Qilu Hospital of Shandong University, Jinan 250012, Shandong Province, China.
Zhang J; Institute of Immunopharmaceutical Sciences, School of Pharmaceutical Sciences, Shandong University, Jinan 250012, Shandong Province, China. .
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Źródło :
World journal of gastroenterology [World J Gastroenterol] 2021 Jun 28; Vol. 27 (24), pp. 3581-3594.
Typ publikacji :
Journal Article
MeSH Terms :
Hepatic Stellate Cells*/pathology
Matrix Metalloproteinase 2*
1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine/analogs & derivatives ; Animals ; Killer Cells, Natural ; Liver/pathology ; Liver Cirrhosis/chemically induced ; Liver Cirrhosis/drug therapy ; Liver Cirrhosis/prevention & control ; Mice ; Mice, Inbred C57BL ; Transforming Growth Factor beta1
Czasopismo naukowe
Tytuł :
MicroRNA-503 Targets Mothers Against Decapentaplegic Homolog 7 Enhancing Hepatic Stellate Cell Activation and Hepatic Fibrosis.
Autorzy :
Xie X; Department of Infectious Diseases, The First Affiliated Hospital of University of South China, Hengyang, 421001, Hunan, China.
Dou CY; Department of Infectious Diseases, The First Affiliated Hospital of University of South China, Hengyang, 421001, Hunan, China.
Zhou Y; Department of Pathology, The Second Affiliated Hospital of University of South China, Hengyang, 421001, China.
Zhou Q; Department of Infectious Diseases, The First Affiliated Hospital of University of South China, Hengyang, 421001, Hunan, China.
Tang HB; Department of Infectious Diseases, The First Affiliated Hospital of University of South China, Hengyang, 421001, Hunan, China. .
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Źródło :
Digestive diseases and sciences [Dig Dis Sci] 2021 Jun; Vol. 66 (6), pp. 1928-1939. Date of Electronic Publication: 2020 Jul 09.
Typ publikacji :
Journal Article
MeSH Terms :
Hepatic Stellate Cells/*metabolism
Liver Cirrhosis/*metabolism
MicroRNAs/*biosynthesis
Smad7 Protein/*biosynthesis
Animals ; Carbon Tetrachloride/toxicity ; Hepatic Stellate Cells/pathology ; Humans ; Liver Cirrhosis/chemically induced ; Liver Cirrhosis/genetics ; Liver Cirrhosis/pathology ; MicroRNAs/genetics ; Rats ; Rats, Sprague-Dawley ; Smad7 Protein/genetics
Czasopismo naukowe
Tytuł :
[Study on mechanism of salidroside against liver fibrosis by regulating CXCL16].
Autorzy :
Ye QN; Shuguang Hospital Affiliated to Shanghai University of Traditional Chinese Medicine Shanghai 201203, China Institute of Hepatology, Shanghai University of Traditional Chinese Medicine Shanghai 201203, China.
Zhao CQ; Shuguang Hospital Affiliated to Shanghai University of Traditional Chinese Medicine Shanghai 201203, China Institute of Hepatology, Shanghai University of Traditional Chinese Medicine Shanghai 201203, China Key Laboratory of Liver and Kidney Diseases, Ministry of Education Shanghai 201203, China.
Ping J; Shuguang Hospital Affiliated to Shanghai University of Traditional Chinese Medicine Shanghai 201203, China Key Laboratory of Liver and Kidney Diseases, Ministry of Education Shanghai 201203, China.
Xu LM; Shuguang Hospital Affiliated to Shanghai University of Traditional Chinese Medicine Shanghai 201203, China Institute of Hepatology, Shanghai University of Traditional Chinese Medicine Shanghai 201203, China Key Laboratory of Liver and Kidney Diseases, Ministry of Education Shanghai 201203, China Shanghai Key Clinical Laboratory of Traditional Chinese Medicine Shanghai 201203, China.
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Źródło :
Zhongguo Zhong yao za zhi = Zhongguo zhongyao zazhi = China journal of Chinese materia medica [Zhongguo Zhong Yao Za Zhi] 2021 Jun; Vol. 46 (11), pp. 2865-2870.
Typ publikacji :
Journal Article
MeSH Terms :
Hepatic Stellate Cells*
Liver Cirrhosis*/drug therapy
Liver Cirrhosis*/genetics
Animals ; Carbon Tetrachloride ; Chemokine CXCL16 ; Glucosides ; Liver/pathology ; Male ; Mice ; Phenols
Czasopismo naukowe
Tytuł :
Synthesis and anti-fibrotic effects of santamarin derivatives as cytotoxic agents against hepatic stellate cell line LX2.
Autorzy :
Wang JP; State Key Laboratory of Phytochemistry and Plant Resources in West China, Kunming Institute of Botany, Chinese Academy of Sciences, Yunnan Key Laboratory of Natural Medicinal Chemistry, Kunming 650201, People's Republic of China; Key Laboratory of Applied Surface and Colloid Chemistry, Ministry of Education and School of Chemistry and Chemical Engineering, Shaanxi Normal University, Xi'an 710062, People's Republic of China.
Li TZ; State Key Laboratory of Phytochemistry and Plant Resources in West China, Kunming Institute of Botany, Chinese Academy of Sciences, Yunnan Key Laboratory of Natural Medicinal Chemistry, Kunming 650201, People's Republic of China.
Huang XY; State Key Laboratory of Phytochemistry and Plant Resources in West China, Kunming Institute of Botany, Chinese Academy of Sciences, Yunnan Key Laboratory of Natural Medicinal Chemistry, Kunming 650201, People's Republic of China.
Geng CA; State Key Laboratory of Phytochemistry and Plant Resources in West China, Kunming Institute of Botany, Chinese Academy of Sciences, Yunnan Key Laboratory of Natural Medicinal Chemistry, Kunming 650201, People's Republic of China.
Shen C; State Key Laboratory of Phytochemistry and Plant Resources in West China, Kunming Institute of Botany, Chinese Academy of Sciences, Yunnan Key Laboratory of Natural Medicinal Chemistry, Kunming 650201, People's Republic of China.
Sun JJ; State Key Laboratory of Phytochemistry and Plant Resources in West China, Kunming Institute of Botany, Chinese Academy of Sciences, Yunnan Key Laboratory of Natural Medicinal Chemistry, Kunming 650201, People's Republic of China; Key Laboratory of Applied Surface and Colloid Chemistry, Ministry of Education and School of Chemistry and Chemical Engineering, Shaanxi Normal University, Xi'an 710062, People's Republic of China.
Xue D; Key Laboratory of Applied Surface and Colloid Chemistry, Ministry of Education and School of Chemistry and Chemical Engineering, Shaanxi Normal University, Xi'an 710062, People's Republic of China. Electronic address: .
Chen JJ; State Key Laboratory of Phytochemistry and Plant Resources in West China, Kunming Institute of Botany, Chinese Academy of Sciences, Yunnan Key Laboratory of Natural Medicinal Chemistry, Kunming 650201, People's Republic of China; University of Chinese Academy of Sciences, Beijing 100049, People's Republic of China. Electronic address: .
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Źródło :
Bioorganic & medicinal chemistry letters [Bioorg Med Chem Lett] 2021 Jun 01; Vol. 41, pp. 127994. Date of Electronic Publication: 2021 Mar 26.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cytotoxins/*pharmacology
Hepatic Stellate Cells/*drug effects
Liver Cirrhosis/*drug therapy
Sesquiterpenes/*pharmacology
Cell Line ; Cytotoxins/chemical synthesis ; Cytotoxins/chemistry ; Dose-Response Relationship, Drug ; Hepatic Stellate Cells/metabolism ; Hepatic Stellate Cells/pathology ; Humans ; Liver Cirrhosis/metabolism ; Liver Cirrhosis/pathology ; Molecular Structure ; Sesquiterpenes/chemical synthesis ; Sesquiterpenes/chemistry ; Structure-Activity Relationship
Czasopismo naukowe
Tytuł :
Previous liver regeneration induces fibro-protective mechanisms during thioacetamide-induced chronic liver injury.
Autorzy :
Gratte FD; School of Veterinary and Life Sciences, Murdoch University, Perth, WA, Australia; Curtin Medical School, Curtin Health Innovation Research Institute, Curtin University, Perth, Australia. Electronic address: .
Pasic S; Curtin Medical School, Curtin Health Innovation Research Institute, Curtin University, Perth, Australia. Electronic address: .
Abu Bakar NDB; Curtin Medical School, Curtin Health Innovation Research Institute, Curtin University, Perth, Australia. Electronic address: .
Gogoi-Tiwari J; School of Veterinary and Life Sciences, Murdoch University, Perth, WA, Australia; Curtin Medical School, Curtin Health Innovation Research Institute, Curtin University, Perth, Australia. Electronic address: .
Liu X; Department of Surgery, University of California, San Diego, La Jolla, CA, USA. Electronic address: .
Carlessi R; Curtin Medical School, Curtin Health Innovation Research Institute, Curtin University, Perth, Australia. Electronic address: .
Kisseleva T; Department of Surgery, University of California, San Diego, La Jolla, CA, USA. Electronic address: .
Brenner DA; School of Medicine, University of California, San Diego, La Jolla, CA, USA. Electronic address: .
Ramm GA; QIMR Berghofer Medical Research Institute, Brisbane, QLD, Australia; The University of Queensland, Brisbane, QLD, Australia. Electronic address: .
Olynyk JK; Fiona Stanley and Fremantle Hospital Group, Perth, WA, Australia; School of Medical and Health Sciences, Edith Cowan University, Perth, WA, Australia. Electronic address: .
Tirnitz-Parker JEE; Curtin Medical School, Curtin Health Innovation Research Institute, Curtin University, Perth, Australia. Electronic address: .
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Źródło :
The international journal of biochemistry & cell biology [Int J Biochem Cell Biol] 2021 May; Vol. 134, pp. 105933. Date of Electronic Publication: 2021 Feb 01.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Chemical and Drug Induced Liver Injury/*pathology
Hepatic Stellate Cells/*cytology
Liver/*cytology
Liver Cirrhosis/*pathology
Liver Regeneration/*physiology
Stem Cells/*cytology
Thioacetamide/*toxicity
Animals ; Cells, Cultured ; Chemical and Drug Induced Liver Injury/etiology ; Chemical and Drug Induced Liver Injury/metabolism ; Coculture Techniques ; Disease Models, Animal ; Hepatic Stellate Cells/metabolism ; Liver/metabolism ; Liver Cirrhosis/etiology ; Liver Cirrhosis/metabolism ; Male ; Mice ; Mice, Inbred C57BL ; Stem Cells/metabolism
Czasopismo naukowe
Tytuł :
CD8 tissue-resident memory T cells promote liver fibrosis resolution by inducing apoptosis of hepatic stellate cells.
Autorzy :
Koda Y; Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo, Japan.; Mitsubishi Tanabe Pharma Corporation, Kanagawa, Japan.
Teratani T; Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo, Japan.
Chu PS; Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo, Japan.
Hagihara Y; Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo, Japan.
Mikami Y; Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo, Japan.
Harada Y; Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo, Japan.
Tsujikawa H; Department of Pathology, Keio University School of Medicine, Tokyo, Japan.
Miyamoto K; Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo, Japan.
Suzuki T; Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo, Japan.
Taniki N; Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo, Japan.
Sujino T; Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo, Japan.
Sakamoto M; Department of Pathology, Keio University School of Medicine, Tokyo, Japan.
Kanai T; Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo, Japan. .; Japan Agency for Medical Research and Development, AMED, Tokyo, Japan. .
Nakamoto N; Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo, Japan. .
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Źródło :
Nature communications [Nat Commun] 2021 Jul 22; Vol. 12 (1), pp. 4474. Date of Electronic Publication: 2021 Jul 22.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
CD8-Positive T-Lymphocytes/*immunology
Hepatic Stellate Cells/*pathology
Liver Cirrhosis/*immunology
Liver Cirrhosis/*pathology
Non-alcoholic Fatty Liver Disease/*immunology
Non-alcoholic Fatty Liver Disease/*pathology
Adoptive Transfer ; Adult ; Aged ; Aged, 80 and over ; Animals ; Apoptosis/immunology ; Disease Models, Animal ; Disease Progression ; Female ; Hepatic Stellate Cells/immunology ; Humans ; Immunologic Memory ; Interleukin-15/immunology ; Liver Cirrhosis/therapy ; Male ; Mice ; Mice, Inbred C57BL ; Middle Aged ; Non-alcoholic Fatty Liver Disease/therapy ; Receptors, CCR5/immunology ; Young Adult
Czasopismo naukowe
Tytuł :
Trefoil factor 2 secreted from damaged hepatocytes activates hepatic stellate cells to induce fibrogenesis.
Autorzy :
Zhang B; Department of Cellular and Molecular Physiology, Yale University, New Haven, Connecticut, USA.
Lapenta K; Program in Integrative Cell Signaling and Neurobiology of Metabolism, Department of Comparative Medicine, Yale University School of Medicine, New Haven, Connecticut, USA.
Wang Q; Department of Cellular and Molecular Physiology, Yale University, New Haven, Connecticut, USA.
Nam JH; Department of Public Health Sciences, Medical University of South Carolina, Charleston, South Carolina, USA.
Chung D; Department of Biomedical Informatics, College of Medicine, Ohio State University, Columbus, Ohio, USA.
Robert ME; Department of Pathology, Yale University School of Medicine, New Haven, Connecticut, USA.
Nathanson MH; Section of Digestive Diseases, Department of Internal Medicine, Yale University School of Medicine, New Haven, Connecticut, USA.
Yang X; Department of Cellular and Molecular Physiology, Yale University, New Haven, Connecticut, USA; Program in Integrative Cell Signaling and Neurobiology of Metabolism, Department of Comparative Medicine, Yale University School of Medicine, New Haven, Connecticut, USA. Electronic address: .
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Źródło :
The Journal of biological chemistry [J Biol Chem] 2021 Jul; Vol. 297 (1), pp. 100887. Date of Electronic Publication: 2021 Jun 17.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Hepatic Stellate Cells/*metabolism
Hepatocytes/*metabolism
Liver Cirrhosis/*metabolism
Trefoil Factor-2/*metabolism
Animals ; Carbon Tetrachloride/toxicity ; Cell Line ; Cells, Cultured ; Exocytosis ; Hepatic Stellate Cells/pathology ; Hepatocytes/pathology ; Humans ; Liver Cirrhosis/etiology ; Mice ; N-Acetylglucosaminyltransferases/deficiency ; N-Acetylglucosaminyltransferases/genetics ; N-Acetylglucosaminyltransferases/metabolism ; Necroptosis ; Signal Transduction ; Trefoil Factor-2/genetics
Czasopismo naukowe
Tytuł :
[Effects of Notch3 on gene expression and signal pathway of pancreatic stellate cell activation].
Autorzy :
Song HY; School of Basic Medical Sciences, Xinxiang Medical University, Xinxiang 453003.
Zhou ZX; Xinxiang Medical University, Xinxiang 453003.
Zhang YX; Department of Biochemistry and Molecular Biology, Capital Medical University, Beijing 100069, China.
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Źródło :
Zhongguo ying yong sheng li xue za zhi = Zhongguo yingyong shenglixue zazhi = Chinese journal of applied physiology [Zhongguo Ying Yong Sheng Li Xue Za Zhi] 2021 Jul; Vol. 37 (4), pp. 349-353.
Typ publikacji :
Journal Article
MeSH Terms :
Pancreatic Stellate Cells*
Signal Transduction*
Animals ; Cells, Cultured ; Collagen Type I ; Gene Expression ; Mice ; RNA, Small Interfering/genetics ; Receptor, Notch3
Czasopismo naukowe
Tytuł :
SOD3 deficiency induces liver fibrosis by promoting hepatic stellate cell activation and epithelial-mesenchymal transition.
Autorzy :
Sun YL; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.; Institute of Hepatobiliary and Pancreatic Diseases, Zhengzhou University, Zhengzhou, China.; Key Lab of Hepatobiliary and Pancreatic Diseases, Zhengzhou, China.
Bai T; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.; Institute of Hepatobiliary and Pancreatic Diseases, Zhengzhou University, Zhengzhou, China.; Key Lab of Hepatobiliary and Pancreatic Diseases, Zhengzhou, China.; Department of Vascular and Endovascular Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
Zhou L; Institute of Hepatobiliary and Pancreatic Diseases, Zhengzhou University, Zhengzhou, China.; Key Lab of Hepatobiliary and Pancreatic Diseases, Zhengzhou, China.; Department of Digestive, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
Zhu RT; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.; Institute of Hepatobiliary and Pancreatic Diseases, Zhengzhou University, Zhengzhou, China.; Key Lab of Hepatobiliary and Pancreatic Diseases, Zhengzhou, China.
Wang WJ; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.; Institute of Hepatobiliary and Pancreatic Diseases, Zhengzhou University, Zhengzhou, China.; Key Lab of Hepatobiliary and Pancreatic Diseases, Zhengzhou, China.
Liang RP; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.; Institute of Hepatobiliary and Pancreatic Diseases, Zhengzhou University, Zhengzhou, China.; Key Lab of Hepatobiliary and Pancreatic Diseases, Zhengzhou, China.
Li J; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.; Institute of Hepatobiliary and Pancreatic Diseases, Zhengzhou University, Zhengzhou, China.; Key Lab of Hepatobiliary and Pancreatic Diseases, Zhengzhou, China.
Zhang CX; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.; Institute of Hepatobiliary and Pancreatic Diseases, Zhengzhou University, Zhengzhou, China.; Key Lab of Hepatobiliary and Pancreatic Diseases, Zhengzhou, China.
Gou JJ; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.; Institute of Hepatobiliary and Pancreatic Diseases, Zhengzhou University, Zhengzhou, China.; Key Lab of Hepatobiliary and Pancreatic Diseases, Zhengzhou, China.
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Źródło :
Journal of cellular physiology [J Cell Physiol] 2021 Jun; Vol. 236 (6), pp. 4313-4329. Date of Electronic Publication: 2020 Nov 23.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Epithelial-Mesenchymal Transition*
Chemical and Drug Induced Liver Injury/*enzymology
Collagen Type I/*metabolism
Hepatic Stellate Cells/*enzymology
Liver/*enzymology
Liver Cirrhosis, Experimental/*enzymology
Superoxide Dismutase/*deficiency
AMP-Activated Protein Kinases/metabolism ; Animals ; Carbon Tetrachloride ; Cells, Cultured ; Chemical and Drug Induced Liver Injury/etiology ; Chemical and Drug Induced Liver Injury/genetics ; Chemical and Drug Induced Liver Injury/pathology ; Hepatic Stellate Cells/pathology ; Liver/pathology ; Liver Cirrhosis, Experimental/chemically induced ; Liver Cirrhosis, Experimental/genetics ; Liver Cirrhosis, Experimental/pathology ; Male ; Mice, Inbred C57BL ; Mice, Knockout ; Signal Transduction ; Sirtuin 1/metabolism ; Superoxide Dismutase/genetics
Czasopismo naukowe
Tytuł :
Endoplasmic reticulum stress and autophagy are involved in adipocyte-induced fibrosis in hepatic stellate cells.
Autorzy :
Liu Y; Institute of Cerebrovascular Diseases, The Affiliated Hospital of Qingdao University, Qingdao, Shandong, China.; Medical College, Qingdao University, Qingdao, 266071, China.
Wu X; Institute of Cerebrovascular Diseases, The Affiliated Hospital of Qingdao University, Qingdao, Shandong, China.
Wang Y; Institute of Cerebrovascular Diseases, The Affiliated Hospital of Qingdao University, Qingdao, Shandong, China.
Guo Y; Institute of Cerebrovascular Diseases, The Affiliated Hospital of Qingdao University, Qingdao, Shandong, China. .; Medical College, Qingdao University, Qingdao, 266071, China. .
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Źródło :
Molecular and cellular biochemistry [Mol Cell Biochem] 2021 Jun; Vol. 476 (6), pp. 2527-2538. Date of Electronic Publication: 2021 Feb 26.
Typ publikacji :
Journal Article
MeSH Terms :
Autophagy*
Endoplasmic Reticulum Stress*
Adipocytes/*metabolism
Hepatic Stellate Cells/*metabolism
Liver Cirrhosis/*metabolism
3T3-L1 Cells ; Adipocytes/pathology ; Animals ; Hepatic Stellate Cells/pathology ; Humans ; Liver Cirrhosis/pathology ; Mice
Czasopismo naukowe
Tytuł :
Tumor restriction by type I collagen opposes tumor-promoting effects of cancer-associated fibroblasts.
Autorzy :
Bhattacharjee S; Department of Medicine, Columbia University, New York, New York, USA.
Hamberger F; Department of Gastroenterology, Hepatology, and Endocrinology, Hannover Medical School, Hanover, Germany.
Ravichandra A; Department of Medicine, Columbia University, New York, New York, USA.
Miller M; Department of Biochemistry and Microbiology, Rutgers University, New Brunswick, New Jersey, USA.
Nair A; Department of Medicine, Columbia University, New York, New York, USA.
Affo S; Department of Medicine, Columbia University, New York, New York, USA.
Filliol A; Department of Medicine, Columbia University, New York, New York, USA.
Chin L; Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, USA.
Savage TM; Department of Microbiology and Immunology, Columbia University, New York, New York, USA.
Yin D; Department of Medicine, Columbia University, New York, New York, USA.
Wirsik NM; Department of General, Visceral and Transplantation Surgery, University Hospital Heidelberg, Heidelberg University, Heidelberg, Germany.
Mehal A; Department of Medicine, Columbia University, New York, New York, USA.
Arpaia N; Department of Microbiology and Immunology, Columbia University, New York, New York, USA.; Herbert Irving Comprehensive Cancer Center, Columbia University, New York, New York, USA.
Seki E; Department of Medicine, Division of Digestive and Liver Diseases, Cedars-Sinai Medical Center, Los Angeles, California, USA.
Mack M; Department of Nephrology, University Hospital Regensburg, Regensburg, Germany.
Zhu D; Department of Pharmacology, Minhang Hospital and School of Pharmacy, Fudan University, Shanghai, China.
Sims PA; Department of Systems Biology, Columbia University, New York, New York, USA.
Kalluri R; Department of Cancer Biology, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA.
Stanger BZ; Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, USA.
Olive KP; Department of Medicine, Columbia University, New York, New York, USA.
Schmidt T; Department of General, Visceral and Transplantation Surgery, University Hospital Heidelberg, Heidelberg University, Heidelberg, Germany.
Wells RG; Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, USA.
Mederacke I; Department of Gastroenterology, Hepatology, and Endocrinology, Hannover Medical School, Hanover, Germany.
Schwabe RF; Department of Medicine, Columbia University, New York, New York, USA.; Institute of Human Nutrition, Columbia University, New York, New York, USA.
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Źródło :
The Journal of clinical investigation [J Clin Invest] 2021 Jun 01; Vol. 131 (11).
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Mechanotransduction, Cellular*
Cancer-Associated Fibroblasts/*metabolism
Collagen Type I/*metabolism
Hepatic Stellate Cells/*metabolism
Liver Neoplasms, Experimental/*metabolism
Animals ; Cancer-Associated Fibroblasts/pathology ; Cell Line, Tumor ; Collagen Type I/genetics ; Hepatic Stellate Cells/pathology ; Humans ; Liver Neoplasms, Experimental/genetics ; Liver Neoplasms, Experimental/pathology ; Mice, Knockout ; Neoplasm Metastasis
Czasopismo naukowe

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