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Tytuł :
Heterogeneous melting kinetics in polycrystalline aluminum.
Autorzy :
Liao Y; School of Mechanical Engineering, Southwest Petroleum University, Chengdu, China.
Xiang M; Laboratory of Computational Physics, Institute of Applied Physics and Computational Mathematics, Beijing, China.
Zhu X; School of Mechanical Engineering, Southwest Petroleum University, Chengdu, China.
Chen J; Laboratory of Computational Physics, Institute of Applied Physics and Computational Mathematics, Beijing, China.; Center for Applied Physics and Technology, Peking University, Beijing, China.
Tian X; College of Mechanics and Materials, HoHai University, Nanjing, China.
Ge L; School of Mechanical Engineering, Southwest Petroleum University, Chengdu, China.
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Źródło :
PloS one [PLoS One] 2020 Mar 10; Vol. 15 (3), pp. e0230028. Date of Electronic Publication: 2020 Mar 10 (Print Publication: 2020).
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
Journal Info :
Publisher: Public Library of Science Country of Publication: United States NLM ID: 101285081 Publication Model: eCollection Cited Medium: Internet ISSN: 1932-6203 (Electronic) Linking ISSN: 19326203 NLM ISO Abbreviation: PLoS ONE Subsets: MEDLINE
MeSH Terms :
Phase Transition*
Aluminum/*chemistry
Kinetics ; Models, Molecular ; Transition Temperature
Czasopismo naukowe
Tytuł :
Melting transitions in biomembranes.
Autorzy :
Mužić T; Membrane Biophysics Group, Niels Bohr Institute, University of Copenhagen, Denmark.
Tounsi F; Membrane Biophysics Group, Niels Bohr Institute, University of Copenhagen, Denmark.
Madsen SB; Membrane Biophysics Group, Niels Bohr Institute, University of Copenhagen, Denmark.
Pollakowski D; Membrane Biophysics Group, Niels Bohr Institute, University of Copenhagen, Denmark.
Konrad M; Max-Planck-Institute for Biophysical Chemistry, Am Fassberg 11, Göttingen 37077, Germany.
Heimburg T; Membrane Biophysics Group, Niels Bohr Institute, University of Copenhagen, Denmark. Electronic address: .
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Źródło :
Biochimica et biophysica acta. Biomembranes [Biochim Biophys Acta Biomembr] 2019 Nov 01; Vol. 1861 (11), pp. 183026. Date of Electronic Publication: 2019 Aug 26.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
Journal Info :
Publisher: Elsevier Country of Publication: Netherlands NLM ID: 101731713 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1879-2642 (Electronic) Linking ISSN: 00052736 NLM ISO Abbreviation: Biochim Biophys Acta Biomembr Subsets: MEDLINE
MeSH Terms :
Transition Temperature*
Cell Membrane/*chemistry
Cell Membrane/*metabolism
Animals ; Bacteria ; Bacterial Physiological Phenomena ; Cell Membrane/physiology ; Lipids/analysis ; Mammals/physiology ; Osmolar Concentration ; Phase Transition ; Temperature ; Thermodynamics
Czasopismo naukowe
Tytuł :
Waste-activated sludge disruption by dry ice: bench scale study and evaluation of heat phase transformations.
Autorzy :
Machnicka A; Faculty of Materials, Civil and Environmental Engineering, University of Bielsko-Biala, Willowa 2 STR, 43-300, Bielsko-Biala, Poland.
Grübel K; Faculty of Materials, Civil and Environmental Engineering, University of Bielsko-Biala, Willowa 2 STR, 43-300, Bielsko-Biala, Poland. .
Wacławek S; Centre for Nanomaterials, Advanced Technologies and Innovation, Technical University of Liberec, Liberec, Czech Republic.
Sikora K; Faculty of Mechanical Engineering and Computer Science, University of Bielsko-Biala, Willowa 2 STR, 43-300, Bielsko-Biala, Poland.
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Źródło :
Environmental science and pollution research international [Environ Sci Pollut Res Int] 2019 Sep; Vol. 26 (26), pp. 26488-26499. Date of Electronic Publication: 2019 Jul 09.
Typ publikacji :
Journal Article
Journal Info :
Publisher: Springer Country of Publication: Germany NLM ID: 9441769 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1614-7499 (Electronic) Linking ISSN: 09441344 NLM ISO Abbreviation: Environ Sci Pollut Res Int Subsets: MEDLINE
MeSH Terms :
Dry Ice*
Models, Theoretical*
Phase Transition*
Sewage/*chemistry
Waste Disposal, Fluid/*methods
Ammonia ; Biological Oxygen Demand Analysis ; Freezing ; Sublimation, Chemical ; Transition Temperature
Czasopismo naukowe
Tytuł :
Nose Temperature and Anticorrelation between Recrystallization Kinetics and Molecular Relaxation Dynamics in Amorphous Morniflumate at High Pressure.
Autorzy :
Romanini M; Grup de Caracterització de Materials, Departament de Física and Barcelona Research Center in Multiscale Science and Engineering , Universitat Politècnica de Catalunya, EEBE , Campus Diagonal-Besòs, Av. Eduard Maristany 10-14 , E-08019 Barcelona , Catalonia , Spain.
Rodriguez S; Grup de Caracterització de Materials, Departament de Física and Barcelona Research Center in Multiscale Science and Engineering , Universitat Politècnica de Catalunya, EEBE , Campus Diagonal-Besòs, Av. Eduard Maristany 10-14 , E-08019 Barcelona , Catalonia , Spain.
Valenti S; Grup de Caracterització de Materials, Departament de Física and Barcelona Research Center in Multiscale Science and Engineering , Universitat Politècnica de Catalunya, EEBE , Campus Diagonal-Besòs, Av. Eduard Maristany 10-14 , E-08019 Barcelona , Catalonia , Spain.
Barrio M; Grup de Caracterització de Materials, Departament de Física and Barcelona Research Center in Multiscale Science and Engineering , Universitat Politècnica de Catalunya, EEBE , Campus Diagonal-Besòs, Av. Eduard Maristany 10-14 , E-08019 Barcelona , Catalonia , Spain.
Tamarit JL; Grup de Caracterització de Materials, Departament de Física and Barcelona Research Center in Multiscale Science and Engineering , Universitat Politècnica de Catalunya, EEBE , Campus Diagonal-Besòs, Av. Eduard Maristany 10-14 , E-08019 Barcelona , Catalonia , Spain.
Macovez R; Grup de Caracterització de Materials, Departament de Física and Barcelona Research Center in Multiscale Science and Engineering , Universitat Politècnica de Catalunya, EEBE , Campus Diagonal-Besòs, Av. Eduard Maristany 10-14 , E-08019 Barcelona , Catalonia , Spain.
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Źródło :
Molecular pharmaceutics [Mol Pharm] 2019 Aug 05; Vol. 16 (8), pp. 3514-3523. Date of Electronic Publication: 2019 Jul 18.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
Journal Info :
Publisher: American Chemical Society Country of Publication: United States NLM ID: 101197791 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1543-8392 (Electronic) Linking ISSN: 15438384 NLM ISO Abbreviation: Mol. Pharm. Subsets: MEDLINE
MeSH Terms :
Transition Temperature*
Anti-Inflammatory Agents, Non-Steroidal/*chemistry
Niflumic Acid/*analogs & derivatives
Crystallization ; Dielectric Spectroscopy ; Hydrostatic Pressure ; Kinetics ; Molecular Dynamics Simulation ; Niflumic Acid/chemistry ; Phase Transition
Czasopismo naukowe
Tytuł :
Telomerase inhibition regulates EMT mechanism in breast cancer stem cells.
Autorzy :
Kusoglu A; Ege University Medical School, Department of Medical Biology, Turkey. Electronic address: .
Goker Bagca B; Ege University Medical School, Department of Medical Biology, Turkey.
Ozates Ay NP; Ege University Medical School, Department of Medical Biology, Turkey.
Gunduz C; Ege University Medical School, Department of Medical Biology, Turkey.
Biray Avci C; Ege University Medical School, Department of Medical Biology, Turkey.
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Źródło :
Gene [Gene] 2020 Oct 30; Vol. 759, pp. 145001. Date of Electronic Publication: 2020 Jul 29.
Typ publikacji :
Journal Article
Journal Info :
Publisher: Elsevier/North-Holland Country of Publication: Netherlands NLM ID: 7706761 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1879-0038 (Electronic) Linking ISSN: 03781119 NLM ISO Abbreviation: Gene Subsets: MEDLINE
MeSH Terms :
Epithelial-Mesenchymal Transition*
Aminobenzoates/*pharmacology
Breast Neoplasms/*metabolism
Enzyme Inhibitors/*pharmacology
Naphthalenes/*pharmacology
Neoplastic Stem Cells/*drug effects
Cell Movement ; Cell Proliferation ; Female ; Humans ; MCF-7 Cells ; Neoplastic Stem Cells/physiology ; Telomerase/antagonists & inhibitors
Czasopismo naukowe
Tytuł :
Mangiferin inhibits lipopolysaccharide-induced epithelial-mesenchymal transition (EMT) and enhances the expression of tumor suppressor gene PER1 in non-small cell lung cancer cells.
Autorzy :
Lin YS; Division of Pulmonary and Critical Care Medicine, An Nan Hospital, China Medical University, Tainan, Taiwan.
Tsai KL; Department of Physical Therapy, College of Medicine, National Cheng Kung University, Tainan, Taiwan.
Chen JN; Department of Medical Sciences Industry, College of Health Sciences, Chang Jung Christian University, Tainan, Taiwan.
Wu CS; Department of Medical Sciences Industry, College of Health Sciences, Chang Jung Christian University, Tainan, Taiwan.
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Źródło :
Environmental toxicology [Environ Toxicol] 2020 Oct; Vol. 35 (10), pp. 1070-1081. Date of Electronic Publication: 2020 May 18.
Typ publikacji :
Journal Article
Journal Info :
Publisher: John Wiley & Sons Country of Publication: United States NLM ID: 100885357 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-7278 (Electronic) Linking ISSN: 15204081 NLM ISO Abbreviation: Environ Toxicol Subsets: MEDLINE
MeSH Terms :
Antineoplastic Agents, Phytogenic/*pharmacology
Cell Movement/*drug effects
Cell Proliferation/*drug effects
Epithelial-Mesenchymal Transition/*drug effects
Period Circadian Proteins/*genetics
Xanthones/*pharmacology
A549 Cells ; Antigens, CD/genetics ; Cadherins/genetics ; Carcinoma, Non-Small-Cell Lung/genetics ; Carcinoma, Non-Small-Cell Lung/pathology ; Cell Line, Tumor ; Cell Movement/genetics ; Cell Proliferation/genetics ; Down-Regulation ; Epithelial-Mesenchymal Transition/genetics ; Humans ; Lipopolysaccharides/pharmacology ; Lung Neoplasms/genetics ; Lung Neoplasms/pathology ; RNA, Small Interfering/genetics ; Receptors, CXCR4/genetics ; Signal Transduction ; Vimentin/metabolism
Czasopismo naukowe
Tytuł :
Elevated soluble E-cadherin during the epithelial-mesenchymal transition process and as a diagnostic marker in colorectal cancer.
Autorzy :
Zhu S; Department of Clinical Laboratory, Qilu Hospital, Cheeloo College of Medicine, Shandong University, 107 Wenhuaxi Road, Jinan, Shandong 250012, China.
Zhao G; Department of Clinical Laboratory, Qilu Hospital, Cheeloo College of Medicine, Shandong University, 107 Wenhuaxi Road, Jinan, Shandong 250012, China.
Zhao X; Department of Clinical Laboratory, Qilu Hospital, Cheeloo College of Medicine, Shandong University, 107 Wenhuaxi Road, Jinan, Shandong 250012, China.
Zhan X; Department of Pathology, The Affiliated Hospital of Qingdao University, Jiangsu Road, Qingdao, Shandong 266035, China.
Cai M; Department of Clinical Laboratory, Qilu Hospital, Cheeloo College of Medicine, Shandong University, 107 Wenhuaxi Road, Jinan, Shandong 250012, China.
Geng C; Department of Clinical Laboratory, Qilu Hospital(Qingdao), Cheeloo College of Medicine, Shandong University, 758 Hefei Road, Qingdao, Shandong 266035, China.
Pu Q; Department of General Surgery, Qilu Hospital(Qingdao), Cheeloo College of Medicine, Shandong University, 758 Hefei Road, Qingdao, Shandong 266035, China.
Zhao Q; Department of Pathology, Qilu Hospital(Qingdao), Cheeloo College of Medicine, Shandong University, 758 Hefei Road, Qingdao, Shandong 266035, China.
Fu Q; Medical Experimental Center, Qilu Hospital(Qingdao), Cheeloo College of Medicine, Shandong University, 758 Hefei Road, Qingdao, Shandong 266035, China.
Huang C; Department of Clinical Laboratory, Qilu Hospital(Qingdao), Cheeloo College of Medicine, Shandong University, 758 Hefei Road, Qingdao, Shandong 266035, China.
Wang Q; Department of Clinical Laboratory, Qilu Hospital, Cheeloo College of Medicine, Shandong University, 107 Wenhuaxi Road, Jinan, Shandong 250012, China. Electronic address: .
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Źródło :
Gene [Gene] 2020 Sep 05; Vol. 754, pp. 144899. Date of Electronic Publication: 2020 Jun 13.
Typ publikacji :
Journal Article
Journal Info :
Publisher: Elsevier/North-Holland Country of Publication: Netherlands NLM ID: 7706761 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1879-0038 (Electronic) Linking ISSN: 03781119 NLM ISO Abbreviation: Gene Subsets: MEDLINE
MeSH Terms :
Cell Movement*
Cell Proliferation*
Epithelial-Mesenchymal Transition*
Antigens, CD/*blood
Biomarkers, Tumor/*blood
Cadherins/*blood
Colorectal Neoplasms/*pathology
Lung Neoplasms/*secondary
Animals ; Apoptosis ; Colorectal Neoplasms/blood ; Female ; Humans ; Lung Neoplasms/blood ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Neoplasm Invasiveness ; Prognosis ; Signal Transduction ; Tumor Cells, Cultured ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
PADI4 modulates the invasion and migration of osteosarcoma cells by down-regulation of epithelial-mesenchymal transition.
Autorzy :
Zhai Q; Center of Translational Medicine, Zibo Central Hospital, Zibo 255036, China.
Qin J; Department of Oncology, Gaoqing People's Hospital, Zibo 256300, China.
Jin X; Department of Geriatrics, Zibo Central Hospital, Zibo 255036, China.
Sun X; Department of Pathology, Zibo Central Hospital, Zibo 255036, China.
Wang L; Center of Translational Medicine, Zibo Central Hospital, Zibo 255036, China.
Du W; Center of Translational Medicine, Zibo Central Hospital, Zibo 255036, China.
Li T; Center of Translational Medicine, Zibo Central Hospital, Zibo 255036, China. Electronic address: .
Xiang X; Center of Translational Medicine, Zibo Central Hospital, Zibo 255036, China. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2020 Sep 01; Vol. 256, pp. 117968. Date of Electronic Publication: 2020 Jun 13.
Typ publikacji :
Journal Article
Journal Info :
Publisher: Elsevier Country of Publication: Netherlands NLM ID: 0375521 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1879-0631 (Electronic) Linking ISSN: 00243205 NLM ISO Abbreviation: Life Sci. Subsets: MEDLINE
MeSH Terms :
Cell Movement*
Down-Regulation*/genetics
Epithelial-Mesenchymal Transition*/genetics
Osteosarcoma/*enzymology
Osteosarcoma/*pathology
Protein-Arginine Deiminase Type 4/*metabolism
Antigens, CD/metabolism ; Cadherins/metabolism ; Cell Line, Tumor ; Gene Expression Regulation, Neoplastic ; Humans ; Lung Neoplasms/secondary ; Neoplasm Invasiveness ; Osteosarcoma/genetics ; Protein-Arginine Deiminase Type 4/genetics
Czasopismo naukowe
Tytuł :
The vicious cycle between ferritinophagy and ROS production triggered EMT inhibition of gastric cancer cells was through p53/AKT/mTor pathway.
Autorzy :
Xu Z; College of Life Science and Technology, PR China.
Feng J; College of Basic Medical Science, Xinxiang Medical University, Xinxiang, Henan, PR China.
Li Y; Department of Histology and Embryology, PR China.
Guan D; College of Basic Medical Science, Xinxiang Medical University, Xinxiang, Henan, PR China.
Chen H; College of Basic Medical Science, Xinxiang Medical University, Xinxiang, Henan, PR China.
Zhai X; Experimental Teaching Center of Biology and Basic Medical Sciences, Sanquan College of Xinxiang Medical University, Xinxiang, Henan, PR China.
Zhang L; Experimental Teaching Center of Biology and Basic Medical Sciences, Sanquan College of Xinxiang Medical University, Xinxiang, Henan, PR China.
Li C; College of Basic Medical Science, Xinxiang Medical University, Xinxiang, Henan, PR China; Experimental Teaching Center of Biology and Basic Medical Sciences, Sanquan College of Xinxiang Medical University, Xinxiang, Henan, PR China. Electronic address: .
Li C; College of Basic Medical Science, Xinxiang Medical University, Xinxiang, Henan, PR China.
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Źródło :
Chemico-biological interactions [Chem Biol Interact] 2020 Sep 01; Vol. 328, pp. 109196. Date of Electronic Publication: 2020 Jul 18.
Typ publikacji :
Comparative Study; Journal Article
Journal Info :
Publisher: Elsevier Country of Publication: Ireland NLM ID: 0227276 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1872-7786 (Electronic) Linking ISSN: 00092797 NLM ISO Abbreviation: Chem. Biol. Interact. Subsets: MEDLINE
MeSH Terms :
Autophagy*/drug effects
Epithelial-Mesenchymal Transition*/drug effects
Ferritins/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Reactive Oxygen Species/*metabolism
Stomach Neoplasms/*pathology
TOR Serine-Threonine Kinases/*metabolism
Tumor Suppressor Protein p53/*metabolism
Cell Line, Tumor ; Ditiocarb/analogs & derivatives ; Ditiocarb/pharmacology ; Humans ; Signal Transduction/drug effects ; Stomach Neoplasms/metabolism ; Transforming Growth Factor beta1/pharmacology
Czasopismo naukowe
Tytuł :
Epstein-Barr virus-encoded miR-BART11 promotes tumor-associated macrophage-induced epithelial-mesenchymal transition via targeting FOXP1 in gastric cancer.
Autorzy :
Song Y; Department of Laboratory Medicine; West China Hospital, Sichuan University, Chengdu, Sichuan, China.
Li Q; Clinical Laboratory, The Affiliated Children Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Liao S; Department of Pathology, The Third Xiangya Hospital, Central South University, Changsha, Hunan, China.
Zhong K; Department of Laboratory Medicine; West China Hospital, Sichuan University, Chengdu, Sichuan, China.
Jin Y; Department of Laboratory Medicine; West China Hospital, Sichuan University, Chengdu, Sichuan, China.
Zeng T; Department of Laboratory Medicine; West China Hospital, Sichuan University, Chengdu, Sichuan, China. Electronic address: .
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Źródło :
Virology [Virology] 2020 Sep; Vol. 548, pp. 6-16. Date of Electronic Publication: 2020 Jun 02.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
Journal Info :
Publisher: Academic Press Country of Publication: United States NLM ID: 0110674 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1096-0341 (Electronic) Linking ISSN: 00426822 NLM ISO Abbreviation: Virology Subsets: MEDLINE
MeSH Terms :
Epithelial-Mesenchymal Transition*
Epstein-Barr Virus Infections/*physiopathology
Forkhead Transcription Factors/*metabolism
Herpesvirus 4, Human/*metabolism
Macrophages/*cytology
MicroRNAs/*metabolism
Repressor Proteins/*metabolism
Stomach Neoplasms/*physiopathology
Cell Line, Tumor ; Cell Movement ; Cell Proliferation ; Epstein-Barr Virus Infections/genetics ; Epstein-Barr Virus Infections/metabolism ; Epstein-Barr Virus Infections/virology ; Forkhead Transcription Factors/genetics ; Gene Expression Regulation, Neoplastic ; Herpesvirus 4, Human/genetics ; Host-Pathogen Interactions ; Humans ; Macrophages/metabolism ; MicroRNAs/genetics ; Repressor Proteins/genetics ; Stomach Neoplasms/genetics ; Stomach Neoplasms/metabolism ; Stomach Neoplasms/virology
Czasopismo naukowe
Tytuł :
Arecoline induces epithelial mesenchymal transition in HK2 cells by upregulating the ERK-mediated signaling pathway.
Autorzy :
Hsieh YH; Department of Biochemistry, School of Medicine, Chung Shan Medical University, Taichung, Taiwan.; Institute of Medicine, Chung Shan Medical University, Taichung, Taiwan.; Clinical laboratory, Chung Shan Medical University Hospital, Taichung, Taiwan.
Syu RJ; Division of Nephrology, Department of Internal Medicine, Dalin Tzu Chi Hospital, Buddhist Tzu Chi Medical Foundation, Chiayi, Taiwan.
Lee CC; Department of Medicine Research, Buddhist Dalin Tzu Chi Hospital, Chiayi, Taiwan.
Lin SH; Institute of Biochemistry, Microbiology and Immunology, Chung Shan Medical University, Taichung, Taiwan.
Lee CH; Division of Pediatric Surgery, Department of Surgery, Children's Hospital of China Medical University, Taichung, Taiwan.; School of Chinese Medicine, College of Chinese Medicine, China Medical University, Taichung, Taiwan.
Cheng CW; Institute of Medicine, Chung Shan Medical University, Taichung, Taiwan.; Institute of Biochemistry, Microbiology and Immunology, Chung Shan Medical University, Taichung, Taiwan.
Tsai JP; Division of Nephrology, Department of Internal Medicine, Dalin Tzu Chi Hospital, Buddhist Tzu Chi Medical Foundation, Chiayi, Taiwan.; School of Medicine, Tzu Chi University, Hualien, Taiwan.
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Źródło :
Environmental toxicology [Environ Toxicol] 2020 Sep; Vol. 35 (9), pp. 1007-1014. Date of Electronic Publication: 2020 May 22.
Typ publikacji :
Journal Article
Journal Info :
Publisher: John Wiley & Sons Country of Publication: United States NLM ID: 100885357 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-7278 (Electronic) Linking ISSN: 15204081 NLM ISO Abbreviation: Environ. Toxicol. Subsets: MEDLINE
MeSH Terms :
Areca/*toxicity
Arecoline/*toxicity
Epithelial-Mesenchymal Transition/*drug effects
Kidney Tubules/*drug effects
MAP Kinase Signaling System/*drug effects
Actins/genetics ; Actins/metabolism ; Antigens, CD/genetics ; Antigens, CD/metabolism ; Areca/chemistry ; Cadherins/genetics ; Cadherins/metabolism ; Cell Cycle/drug effects ; Cell Line ; Cell Movement/drug effects ; Dose-Response Relationship, Drug ; Epithelial-Mesenchymal Transition/genetics ; Fibrosis ; Humans ; Kidney Tubules/metabolism ; Kidney Tubules/pathology ; MAP Kinase Signaling System/genetics ; Phosphorylation ; Renal Insufficiency, Chronic/metabolism ; Renal Insufficiency, Chronic/pathology ; Up-Regulation
Czasopismo naukowe
Tytuł :
Endosulfan triggers epithelial-mesenchymal transition via PTP4A3-mediated TGF-β signaling pathway in prostate cancer cells.
Autorzy :
Wang Y; Institute of Environmental Systems Biology, Environment Science and Engineering College, Dalian Maritime University, Linghai Road 1, Dalian 116026, PR China.
Guo Y; Institute of Environmental Systems Biology, Environment Science and Engineering College, Dalian Maritime University, Linghai Road 1, Dalian 116026, PR China.
Hu Y; Institute of Environmental Systems Biology, Environment Science and Engineering College, Dalian Maritime University, Linghai Road 1, Dalian 116026, PR China.
Sun Y; Institute of Environmental Systems Biology, Environment Science and Engineering College, Dalian Maritime University, Linghai Road 1, Dalian 116026, PR China.
Xu D; Institute of Environmental Systems Biology, Environment Science and Engineering College, Dalian Maritime University, Linghai Road 1, Dalian 116026, PR China. Electronic address: .
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Źródło :
The Science of the total environment [Sci Total Environ] 2020 Aug 20; Vol. 731, pp. 139234. Date of Electronic Publication: 2020 May 07.
Typ publikacji :
Journal Article
Journal Info :
Publisher: Elsevier Country of Publication: Netherlands NLM ID: 0330500 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1879-1026 (Electronic) Linking ISSN: 00489697 NLM ISO Abbreviation: Sci. Total Environ. Subsets: MEDLINE
MeSH Terms :
Endosulfan*
Epithelial-Mesenchymal Transition*
Prostatic Neoplasms*
Cell Line, Tumor ; Humans ; Male ; Neoplasm Proteins ; Protein Tyrosine Phosphatases ; Signal Transduction ; Transforming Growth Factor beta
Czasopismo naukowe
Tytuł :
Cyclic pentapeptide cRGDfK enhances the inhibitory effect of sunitinib on TGF-β1-induced epithelial-to-mesenchymal transition in human non-small cell lung cancer cells.
Autorzy :
Park KY; Department of Integrated Material's Development, CHA Meditech Co., Ltd, Daejeon, Republic of Korea.
Kim J; Department of Medical Laboratory Science, College of Health Science, Dankook University, Cheonan, Republic of Korea.
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Źródło :
PloS one [PLoS One] 2020 Aug 18; Vol. 15 (8), pp. e0232917. Date of Electronic Publication: 2020 Aug 18 (Print Publication: 2020).
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
Journal Info :
Publisher: Public Library of Science Country of Publication: United States NLM ID: 101285081 Publication Model: eCollection Cited Medium: Internet ISSN: 1932-6203 (Electronic) Linking ISSN: 19326203 Subsets: MEDLINE
MeSH Terms :
Carcinoma, Non-Small-Cell Lung/*drug therapy
Epithelial-Mesenchymal Transition/*drug effects
Lung Neoplasms/*drug therapy
Peptides, Cyclic/*administration & dosage
Sunitinib/*administration & dosage
Transforming Growth Factor beta1/*antagonists & inhibitors
A549 Cells ; Adenosine Triphosphate/metabolism ; Antineoplastic Combined Chemotherapy Protocols/administration & dosage ; Biomarkers, Tumor/genetics ; Biomarkers, Tumor/metabolism ; Carcinoma, Non-Small-Cell Lung/metabolism ; Carcinoma, Non-Small-Cell Lung/pathology ; Catalytic Domain ; Cell Line, Tumor ; Cell Survival/drug effects ; Drug Synergism ; Epithelial-Mesenchymal Transition/genetics ; Epithelial-Mesenchymal Transition/physiology ; Humans ; Integrin alphaVbeta3/antagonists & inhibitors ; Lung Neoplasms/metabolism ; Lung Neoplasms/pathology ; Molecular Docking Simulation ; Neoplasm Invasiveness/pathology ; Neoplasm Invasiveness/prevention & control ; Protein-Serine-Threonine Kinases/antagonists & inhibitors ; Protein-Serine-Threonine Kinases/chemistry ; Protein-Serine-Threonine Kinases/metabolism ; Smad Proteins/metabolism ; Wnt Signaling Pathway/drug effects
Czasopismo naukowe
Tytuł :
Gambogenic acid induces ferroptosis in melanoma cells undergoing epithelial-to-mesenchymal transition.
Autorzy :
Wang M; Key Laboratory of Xin'an Medicine, Ministry of Education, China.
Li S; Key Laboratory of Xin'an Medicine, Ministry of Education, China.
Wang Y; Key Laboratory of Xin'an Medicine, Ministry of Education, China.
Cheng H; Key Laboratory of Xin'an Medicine, Ministry of Education, China.
Su J; Key Laboratory of Xin'an Medicine, Ministry of Education, China.
Li Q; Key Laboratory of Xin'an Medicine, Ministry of Education, China; Key Laboratory of Chinese Medicial Formula of Anhui Province, Anhui University of Chinese Medicine, Hefei 230038, China. Electronic address: .
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Źródło :
Toxicology and applied pharmacology [Toxicol Appl Pharmacol] 2020 Aug 15; Vol. 401, pp. 115110. Date of Electronic Publication: 2020 Jun 10.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
Journal Info :
Publisher: Academic Press Country of Publication: United States NLM ID: 0416575 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1096-0333 (Electronic) Linking ISSN: 0041008X NLM ISO Abbreviation: Toxicol. Appl. Pharmacol. Subsets: MEDLINE
MeSH Terms :
Drugs, Chinese Herbal/*toxicity
Epithelial-Mesenchymal Transition/*drug effects
Ferroptosis/*drug effects
Melanoma/*metabolism
Skin Neoplasms/*metabolism
Xanthenes/*toxicity
Cell Line, Tumor ; Cell Survival/drug effects ; Cell Survival/physiology ; Drugs, Chinese Herbal/therapeutic use ; Epithelial-Mesenchymal Transition/physiology ; Ferroptosis/physiology ; Humans ; Melanoma/drug therapy ; Melanoma/pathology ; Skin Neoplasms/drug therapy ; Skin Neoplasms/pathology ; Xanthenes/therapeutic use
Czasopismo naukowe
Tytuł :
Bone mesenchymal stem cells are recruited via CXCL8-CXCR2 and promote EMT through TGF-β signal pathways in oral squamous carcinoma.
Autorzy :
Meng L; Department of Oral Pathology, Jilin Provincial Key Laboratory of Tooth Development and Bone Remodeling, School and Hospital of Stomatology, Jilin University, Changchun, China.
Zhao Y; State Key Laboratory of Supramolecular Structure and Material, College of Chemistry, Jilin University, Changchun, China.
Bu W; School and Hospital of Stomatology, China Medical University, Shenyang, China.
Li X; School and Hospital of Stomatology, China Medical University, Shenyang, China.
Liu X; Department of Oral Pathology, Jilin Provincial Key Laboratory of Tooth Development and Bone Remodeling, School and Hospital of Stomatology, Jilin University, Changchun, China.
Zhou D; School and Hospital of Stomatology, China Medical University, Shenyang, China.
Chen Y; Department of Oral Pathology, Jilin Provincial Key Laboratory of Tooth Development and Bone Remodeling, School and Hospital of Stomatology, Jilin University, Changchun, China.
Zheng S; Department of Oral Pathology, Jilin Provincial Key Laboratory of Tooth Development and Bone Remodeling, School and Hospital of Stomatology, Jilin University, Changchun, China.
Lin Q; State Key Laboratory of Supramolecular Structure and Material, College of Chemistry, Jilin University, Changchun, China.
Liu Q; Department of Oral Pathology, Jilin Provincial Key Laboratory of Tooth Development and Bone Remodeling, School and Hospital of Stomatology, Jilin University, Changchun, China.
Sun H; Department of Oral Pathology, Jilin Provincial Key Laboratory of Tooth Development and Bone Remodeling, School and Hospital of Stomatology, Jilin University, Changchun, China.
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Źródło :
Cell proliferation [Cell Prolif] 2020 Aug; Vol. 53 (8), pp. e12859. Date of Electronic Publication: 2020 Jun 26.
Typ publikacji :
Journal Article
Journal Info :
Publisher: Published for the Cell Kinetics Society, the European Study Group for Cell Proliferation, and the International Cell Cycle Society by Blackwell Scientific Publications Country of Publication: England NLM ID: 9105195 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1365-2184 (Electronic) Linking ISSN: 09607722 NLM ISO Abbreviation: Cell Prolif. Subsets: MEDLINE
MeSH Terms :
Epithelial-Mesenchymal Transition*
Signal Transduction*
Carcinoma, Squamous Cell/*pathology
Gene Expression Regulation, Neoplastic/*genetics
Mesenchymal Stem Cells/*cytology
Mouth Neoplasms/*genetics
Transforming Growth Factor beta/*metabolism
Animals ; Cadherins/metabolism ; Carcinoma, Squamous Cell/genetics ; Cell Movement/physiology ; Cell Proliferation/physiology ; Interleukin-8/metabolism ; Male ; Mice, Nude ; Mouth Neoplasms/immunology ; Receptors, Interleukin-8B/metabolism ; Tumor Microenvironment/physiology
Czasopismo naukowe
Tytuł :
[Effect of salvianolic acid B on high-glucose induced renal tubular epithelial-mesenchymal transition in rats and its mechanism].
Autorzy :
Sun L; Department of Pathophysiology, School of Basic Medical Sciences, Guizhou Medical University Guiyang 550025, China Guizhou Provincial Key Laboratory of Pathogenesis & Drug Research on Common Chronic Diseases, Guizhou Medical University Guiyang 550025, China.
Tian PP; Department of Pathophysiology, School of Basic Medical Sciences, Guizhou Medical University Guiyang 550025, China Guizhou Provincial Key Laboratory of Pathogenesis & Drug Research on Common Chronic Diseases, Guizhou Medical University Guiyang 550025, China.
Zhang F; Department of Pathophysiology, School of Basic Medical Sciences, Guizhou Medical University Guiyang 550025, China Guizhou Provincial Key Laboratory of Pathogenesis & Drug Research on Common Chronic Diseases, Guizhou Medical University Guiyang 550025, China.
Xiao Y; Department of Pathophysiology, School of Basic Medical Sciences, Guizhou Medical University Guiyang 550025, China Guizhou Provincial Key Laboratory of Pathogenesis & Drug Research on Common Chronic Diseases, Guizhou Medical University Guiyang 550025, China.
Guo B; Department of Pathophysiology, School of Basic Medical Sciences, Guizhou Medical University Guiyang 550025, China Guizhou Provincial Key Laboratory of Pathogenesis & Drug Research on Common Chronic Diseases, Guizhou Medical University Guiyang 550025, China.
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Źródło :
Zhongguo Zhong yao za zhi = Zhongguo zhongyao zazhi = China journal of Chinese materia medica [Zhongguo Zhong Yao Za Zhi] 2020 Aug; Vol. 45 (16), pp. 3922-3930.
Typ publikacji :
Journal Article
Journal Info :
Publisher: Zhongguo yao xue hui : Zhongguo Zhong yi yan jiu yuan Zhong yao yan jiu suo Country of Publication: China NLM ID: 8913656 Publication Model: Print Cited Medium: Print ISSN: 1001-5302 (Print) Linking ISSN: 10015302 NLM ISO Abbreviation: Zhongguo Zhong Yao Za Zhi Subsets: MEDLINE
MeSH Terms :
Epithelial-Mesenchymal Transition*
Transforming Growth Factor beta1*
Animals ; Benzofurans ; Epithelial Cells ; Glucose ; Rats
Czasopismo naukowe
Tytuł :
Tumor budding is an adverse prognostic marker in intestinal-type sinonasal adenocarcinoma and seems to be unrelated to epithelial-mesenchymal transition.
Autorzy :
Maffeis V; Department of Medicine (DIMED), Surgical Pathology & Cytopathology Unit, University of Padova, Via Aristide Gabelli, 61, 35121, Padua, Italy. .
Cappellesso R; Department of Medicine (DIMED), Surgical Pathology & Cytopathology Unit, University of Padova, Via Aristide Gabelli, 61, 35121, Padua, Italy.
Galuppini F; Department of Medicine (DIMED), Surgical Pathology & Cytopathology Unit, University of Padova, Via Aristide Gabelli, 61, 35121, Padua, Italy.
Guzzardo V; Department of Medicine (DIMED), Surgical Pathology & Cytopathology Unit, University of Padova, Via Aristide Gabelli, 61, 35121, Padua, Italy.
Zanon A; Department of Neurosciences, Otorhinolaryngology Section, University of Padova, Padua, Italy.
Cazzador D; Department of Neurosciences, Otorhinolaryngology Section, University of Padova, Padua, Italy.; Department of Neurosciences, Section of Human Anatomy, University of Padova, Padua, Italy.
Emanuelli E; Department of Neurosciences, Otorhinolaryngology Section, University of Padova, Padua, Italy.
Ventura L; Department of Statistics, University of Padova, Padua, Italy.
Martini A; Department of Neurosciences, Otorhinolaryngology Section, University of Padova, Padua, Italy.
Fassina A; Department of Medicine (DIMED), Surgical Pathology & Cytopathology Unit, University of Padova, Via Aristide Gabelli, 61, 35121, Padua, Italy.
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Źródło :
Virchows Archiv : an international journal of pathology [Virchows Arch] 2020 Aug; Vol. 477 (2), pp. 241-248. Date of Electronic Publication: 2020 Jan 24.
Typ publikacji :
Journal Article
Journal Info :
Publisher: Springer International Country of Publication: Germany NLM ID: 9423843 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1432-2307 (Electronic) Linking ISSN: 09456317 NLM ISO Abbreviation: Virchows Arch. Subsets: MEDLINE
MeSH Terms :
Epithelial-Mesenchymal Transition*
Adenocarcinoma/*pathology
Biomarkers, Tumor/*metabolism
Colorectal Neoplasms/*pathology
Neoplasm Recurrence, Local/*pathology
Paranasal Sinus Neoplasms/*pathology
Adult ; Aged ; Aged, 80 and over ; Cadherins/metabolism ; Female ; Humans ; Male ; Middle Aged ; Neoplasm Recurrence, Local/diagnosis ; Prognosis ; Snail Family Transcription Factors/metabolism
Czasopismo naukowe
Tytuł :
Coupling of Membrane Nanodomain Formation and Enhanced Electroporation near Phase Transition.
Autorzy :
Kirsch SA; Computational Biology, Friedrich-Alexander University of Erlangen-Nürnberg, Erlangen, Germany.
Böckmann RA; Computational Biology, Friedrich-Alexander University of Erlangen-Nürnberg, Erlangen, Germany. Electronic address: .
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Źródło :
Biophysical journal [Biophys J] 2019 Jun 04; Vol. 116 (11), pp. 2131-2148. Date of Electronic Publication: 2019 Apr 30.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
Journal Info :
Publisher: Cell Press Country of Publication: United States NLM ID: 0370626 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1542-0086 (Electronic) Linking ISSN: 00063495 NLM ISO Abbreviation: Biophys. J. Subsets: MEDLINE
MeSH Terms :
Electroporation*
Phase Transition*
Cell Membrane/*chemistry
Nanostructures/*chemistry
Models, Molecular ; Transition Temperature
Czasopismo naukowe
Tytuł :
Long noncoding RNA H19 acts as a miR-340-3p sponge to promote epithelial-mesenchymal transition by regulating YWHAZ expression in paclitaxel-resistant breast cancer cells.
Autorzy :
Yan L; Anhui Province Key Laboratory of Translational Cancer Research, Bengbu Medical College, Bengbu, Anhui, China.
Yang S; Anhui Province Key Laboratory of Translational Cancer Research, Bengbu Medical College, Bengbu, Anhui, China.
Yue CX; Anhui Province Key Laboratory of Translational Cancer Research, Bengbu Medical College, Bengbu, Anhui, China.
Wei XY; Anhui Province Key Laboratory of Translational Cancer Research, Bengbu Medical College, Bengbu, Anhui, China.
Peng W; Anhui Province Key Laboratory of Translational Cancer Research, Bengbu Medical College, Bengbu, Anhui, China.; Department of Biotechnology, Bengbu Medical College, Bengbu, Anhui, China.
Dong ZY; Anhui Province Key Laboratory of Translational Cancer Research, Bengbu Medical College, Bengbu, Anhui, China.
Xu HN; Anhui Province Key Laboratory of Translational Cancer Research, Bengbu Medical College, Bengbu, Anhui, China.
Chen SL; Anhui Province Key Laboratory of Translational Cancer Research, Bengbu Medical College, Bengbu, Anhui, China.; Department of Biochemistry and Molecular Biology, Bengbu Medical College, Bengbu, Anhui, China.
Wang WR; Anhui Province Key Laboratory of Translational Cancer Research, Bengbu Medical College, Bengbu, Anhui, China.; Department of Biotechnology, Bengbu Medical College, Bengbu, Anhui, China.
Chen CJ; Anhui Province Key Laboratory of Translational Cancer Research, Bengbu Medical College, Bengbu, Anhui, China.; Department of Biochemistry and Molecular Biology, Bengbu Medical College, Bengbu, Anhui, China.
Yang QL; Anhui Province Key Laboratory of Translational Cancer Research, Bengbu Medical College, Bengbu, Anhui, China.; Department of Biochemistry and Molecular Biology, Bengbu Medical College, Bengbu, Anhui, China.
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Źródło :
Environmental toxicology [Environ Toxicol] 2020 Sep; Vol. 35 (9), pp. 1015-1028. Date of Electronic Publication: 2020 May 18.
Typ publikacji :
Journal Article
Journal Info :
Publisher: John Wiley & Sons Country of Publication: United States NLM ID: 100885357 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-7278 (Electronic) Linking ISSN: 15204081 NLM ISO Abbreviation: Environ. Toxicol. Subsets: MEDLINE
MeSH Terms :
14-3-3 Proteins/*metabolism
Antineoplastic Agents, Phytogenic/*pharmacology
Drug Resistance, Neoplasm/*genetics
Epithelial-Mesenchymal Transition/*drug effects
MicroRNAs/*genetics
Paclitaxel/*pharmacology
RNA, Long Noncoding/*genetics
Apoptosis/drug effects ; Apoptosis/genetics ; Cell Movement/drug effects ; Cell Movement/genetics ; Cell Proliferation/drug effects ; Cell Proliferation/genetics ; Epithelial-Mesenchymal Transition/genetics ; Female ; Gene Expression Regulation, Neoplastic/drug effects ; Humans ; MCF-7 Cells ; MicroRNAs/metabolism ; RNA, Long Noncoding/metabolism ; Wnt Signaling Pathway/drug effects ; Wnt Signaling Pathway/genetics
Czasopismo naukowe
Tytuł :
Long noncoding RNAs AC026904.1 is essential for TGF-β-induced migration and epithelial-mesenchymal transition through functioning as an enhancer of Slug in lung cancer cells.
Autorzy :
Yao J; Department of Thoracic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, China.
Lu X; Department of Thoracic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, China.
Wang Y; Department of Thoracic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, China.
Li J; Department of Thoracic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, China.
Ni B; Department of Thoracic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, China.
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Źródło :
Environmental toxicology [Environ Toxicol] 2020 Sep; Vol. 35 (9), pp. 942-951. Date of Electronic Publication: 2020 Apr 09.
Typ publikacji :
Journal Article
Journal Info :
Publisher: John Wiley & Sons Country of Publication: United States NLM ID: 100885357 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-7278 (Electronic) Linking ISSN: 15204081 NLM ISO Abbreviation: Environ. Toxicol. Subsets: MEDLINE
MeSH Terms :
Cell Movement/*genetics
Epithelial-Mesenchymal Transition/*genetics
Lung/*pathology
Lung Neoplasms/*pathology
RNA, Long Noncoding/*genetics
Snail Family Transcription Factors/*metabolism
Transforming Growth Factor beta/*metabolism
A549 Cells ; Cell Line, Tumor ; Cell Movement/drug effects ; Epithelial-Mesenchymal Transition/drug effects ; Female ; Gene Expression Regulation, Neoplastic/drug effects ; Humans ; Lung/metabolism ; Lung Neoplasms/genetics ; Lung Neoplasms/metabolism ; Neoplasm Metastasis ; RNA, Small Interfering/genetics ; Signal Transduction ; Snail Family Transcription Factors/genetics ; Transforming Growth Factor beta/pharmacology ; Up-Regulation
Czasopismo naukowe

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