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Tytuł:
Control of cell proliferation by memories of mitosis.
Autorzy:
Meitinger F; Department of Cell and Developmental Biology, School of Biological Sciences, University of California San Diego, La Jolla, CA 92093, USA.; Department of Cellular and Molecular Medicine, University of California San Diego, La Jolla, CA 92093, USA.; Ludwig Institute for Cancer Research, La Jolla, CA 92093, USA.; Okinawa Institute of Science and Technology Graduate University, Okinawa 904-0495, Japan.
Belal H; Okinawa Institute of Science and Technology Graduate University, Okinawa 904-0495, Japan.
Davis RL; Small Molecule Discovery Program, Ludwig Institute for Cancer Research, La Jolla, CA 92093, USA.
Martinez MB; Small Molecule Discovery Program, Ludwig Institute for Cancer Research, La Jolla, CA 92093, USA.
Shiau AK; Department of Cell and Developmental Biology, School of Biological Sciences, University of California San Diego, La Jolla, CA 92093, USA.; Small Molecule Discovery Program, Ludwig Institute for Cancer Research, La Jolla, CA 92093, USA.
Oegema K; Department of Cell and Developmental Biology, School of Biological Sciences, University of California San Diego, La Jolla, CA 92093, USA.; Department of Cellular and Molecular Medicine, University of California San Diego, La Jolla, CA 92093, USA.; Ludwig Institute for Cancer Research, La Jolla, CA 92093, USA.
Desai A; Department of Cell and Developmental Biology, School of Biological Sciences, University of California San Diego, La Jolla, CA 92093, USA.; Department of Cellular and Molecular Medicine, University of California San Diego, La Jolla, CA 92093, USA.; Ludwig Institute for Cancer Research, La Jolla, CA 92093, USA.
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Źródło:
Science (New York, N.Y.) [Science] 2024 Mar 29; Vol. 383 (6690), pp. 1441-1448. Date of Electronic Publication: 2024 Mar 28.
Typ publikacji:
Journal Article
MeSH Terms:
Cell Proliferation*/genetics
Mitosis*/drug effects
Mitosis*/genetics
Neoplasms*/genetics
Neoplasms*/pathology
Ubiquitin Thiolesterase*/genetics
Ubiquitin Thiolesterase*/metabolism
Tumor Suppressor p53-Binding Protein 1*/genetics
Tumor Suppressor p53-Binding Protein 1*/metabolism
Humans ; Genomic Instability ; Tumor Suppressor Protein p53/genetics ; Tumor Suppressor Protein p53/metabolism ; Cell Line, Tumor ; Polo-Like Kinase 1/metabolism ; Antimitotic Agents/pharmacology ; Drug Resistance, Neoplasm
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Cytokine Signaling in Pediatric Kidney Tumor Cell Lines WT-CLS1, WT-3ab and G-401.
Autorzy:
Fasler-Kan E; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Milošević M; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Ruggiero S; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Aliu N; Department of Human Genetics, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Cholewa D; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Häcker FM; Department of Pediatric Surgery, Children's Hospital of Eastern Switzerland, CH-9000 St. Gallen, Switzerland.; Faculty of Medicine, University of Basel, CH-4031 Basel, Switzerland.
Dekany G; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Bartenstein A; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Berger SM; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2024 Feb 14; Vol. 25 (4). Date of Electronic Publication: 2024 Feb 14.
Typ publikacji:
Journal Article
MeSH Terms:
Tumor Necrosis Factor-alpha*/metabolism
Kidney Neoplasms*/pathology
Child ; Humans ; Cytokines/metabolism ; Interferon-alpha/metabolism ; Histocompatibility Antigens Class I/metabolism ; HLA Antigens ; Cell Line, Tumor ; STAT1 Transcription Factor/metabolism ; Kidney/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Targeting TRIP13 in favorable histology Wilms tumor with nuclear export inhibitors synergizes with doxorubicin.
Autorzy:
Mittal K; Department of Pediatrics, Emory University School of Medicine, Atlanta, GA, USA.; Aflac Cancer and Blood Disorders Center, Children's Healthcare of Atlanta, Atlanta, GA, USA.
Cooper GW; Department of Pediatrics, Emory University School of Medicine, Atlanta, GA, USA.; Aflac Cancer and Blood Disorders Center, Children's Healthcare of Atlanta, Atlanta, GA, USA.
Lee BP; Department of Pediatrics, Emory University School of Medicine, Atlanta, GA, USA.; Aflac Cancer and Blood Disorders Center, Children's Healthcare of Atlanta, Atlanta, GA, USA.
Su Y; Department of Pediatrics, Emory University School of Medicine, Atlanta, GA, USA.; Aflac Cancer and Blood Disorders Center, Children's Healthcare of Atlanta, Atlanta, GA, USA.
Skinner KT; Department of Pediatrics, Emory University School of Medicine, Atlanta, GA, USA.; Aflac Cancer and Blood Disorders Center, Children's Healthcare of Atlanta, Atlanta, GA, USA.
Shim J; Department of Pediatrics, Emory University School of Medicine, Atlanta, GA, USA.; Aflac Cancer and Blood Disorders Center, Children's Healthcare of Atlanta, Atlanta, GA, USA.; Winship Cancer Institute, Emory University, Atlanta, GA, USA.
Jonus HC; Department of Pediatrics, Emory University School of Medicine, Atlanta, GA, USA.; Aflac Cancer and Blood Disorders Center, Children's Healthcare of Atlanta, Atlanta, GA, USA.
Kim WJ; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.; Broad Institute of MIT and Harvard, Cambridge, MA, USA.
Doshi M; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.; Broad Institute of MIT and Harvard, Cambridge, MA, USA.
Almanza D; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.; Broad Institute of MIT and Harvard, Cambridge, MA, USA.
Kynnap BD; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.; Broad Institute of MIT and Harvard, Cambridge, MA, USA.
Christie AL; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.
Yang X; Broad Institute of MIT and Harvard, Cambridge, MA, USA.
Cowley GS; Broad Institute of MIT and Harvard, Cambridge, MA, USA.
Leeper BA; Experimental Therapeutics Core and Belfer Center for Applied Cancer Science, Dana-Farber Cancer Institute, Boston, MA, USA.
Morton CL; Department of Surgery, St. Jude Children's Research Hospital, Memphis, TN, USA.
Dwivedi B; Winship Cancer Institute, Emory University, Atlanta, GA, USA.
Lawrence T; Department of Pediatrics, Emory University School of Medicine, Atlanta, GA, USA.; Aflac Cancer and Blood Disorders Center, Children's Healthcare of Atlanta, Atlanta, GA, USA.
Rupji M; Winship Cancer Institute, Emory University, Atlanta, GA, USA.
Keskula P; Broad Institute of MIT and Harvard, Cambridge, MA, USA.
Meyer S; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.
Clinton CM; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.
Bhasin M; Department of Pediatrics, Emory University School of Medicine, Atlanta, GA, USA.; Aflac Cancer and Blood Disorders Center, Children's Healthcare of Atlanta, Atlanta, GA, USA.; Winship Cancer Institute, Emory University, Atlanta, GA, USA.
Crompton BD; Broad Institute of MIT and Harvard, Cambridge, MA, USA.; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.
Tseng YY; Broad Institute of MIT and Harvard, Cambridge, MA, USA.
Boehm JS; Broad Institute of MIT and Harvard, Cambridge, MA, USA.
Ligon KL; Broad Institute of MIT and Harvard, Cambridge, MA, USA.; Department of Pathology, Brigham and Women's Hospital, Boston, MA, USA.; Department of Oncologic Pathology, Dana-Farber Cancer Institute, Boston, MA, USA.
Root DE; Broad Institute of MIT and Harvard, Cambridge, MA, USA.
Murphy AJ; Department of Surgery, St. Jude Children's Research Hospital, Memphis, TN, USA.
Weinstock DM; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.; Broad Institute of MIT and Harvard, Cambridge, MA, USA.; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.; Merck & Co., Rahway, NJ, USA.
Gokhale PC; Experimental Therapeutics Core and Belfer Center for Applied Cancer Science, Dana-Farber Cancer Institute, Boston, MA, USA.
Spangle JM; Winship Cancer Institute, Emory University, Atlanta, GA, USA.; Department of Radiation Oncology, Emory University School of Medicine, Atlanta, GA, USA.
Rivera MN; Broad Institute of MIT and Harvard, Cambridge, MA, USA.; Department of Pathology, Massachusetts General Hospital, Boston, MA, USA.
Mullen EA; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.
Stegmaier K; Broad Institute of MIT and Harvard, Cambridge, MA, USA.; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.
Goldsmith KC; Department of Pediatrics, Emory University School of Medicine, Atlanta, GA, USA.; Aflac Cancer and Blood Disorders Center, Children's Healthcare of Atlanta, Atlanta, GA, USA.; Winship Cancer Institute, Emory University, Atlanta, GA, USA.
Hahn WC; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA, USA. william_.; Broad Institute of MIT and Harvard, Cambridge, MA, USA. william_.
Hong AL; Department of Pediatrics, Emory University School of Medicine, Atlanta, GA, USA. .; Aflac Cancer and Blood Disorders Center, Children's Healthcare of Atlanta, Atlanta, GA, USA. .; Winship Cancer Institute, Emory University, Atlanta, GA, USA. .
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Źródło:
Communications biology [Commun Biol] 2024 Apr 08; Vol. 7 (1), pp. 426. Date of Electronic Publication: 2024 Apr 08.
Typ publikacji:
Journal Article
MeSH Terms:
Wilms Tumor*/drug therapy
Wilms Tumor*/genetics
Kidney Neoplasms*/drug therapy
Kidney Neoplasms*/genetics
Hydrazines*
Triazoles*
Humans ; Exportin 1 Protein ; Active Transport, Cell Nucleus ; Karyopherins/genetics ; Karyopherins/metabolism ; Receptors, Cytoplasmic and Nuclear/genetics ; Receptors, Cytoplasmic and Nuclear/metabolism ; Cell Line, Tumor ; Apoptosis ; Neoplasm Recurrence, Local ; Doxorubicin/pharmacology ; ATPases Associated with Diverse Cellular Activities/metabolism ; Cell Cycle Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Diagnostic Utility of GATA3 and ISL1 in Differentiating Neuroblastoma From Other Pediatric Malignant Small Round Blue Cell Tumors.
Autorzy:
Mohanty SK; Department of Pathology and Laboratory Medicine, Advanced Medical Research Institute, Bhubaneswar, Odisha, India.; Department of Pathology and Laboratory Medicine, CORE Diagnostics, Gurgaon, Delhi, India.
Diwaker P; Department of Pathology, University College of Medical Sciences, Delhi, India.
Mishra SK; Department of Medical Oncology, Advanced Medical Research Institute, Bhubaneswar, Odisha, India.
Jha S; Department of Pathology and Laboratory Medicine, Advanced Medical Research Institute, Bhubaneswar, Odisha, India.
Lobo A; Department of Pathology and Laboratory Medicine, Advanced Medical Research Institute, Bhubaneswar, Odisha, India.
Panda SP; Department of Pediatric Oncology, Institute of Medical Sciences and SUM Hospital, Bhubaneswar, Odisha, India.
Sharma S; Department of Pathology and Laboratory Medicine, CORE Diagnostics, Gurgaon, Delhi, India.
Kumar M; Department of Pathology and Laboratory Medicine, CORE Diagnostics, Gurgaon, Delhi, India.
Arora S; Department of Pathology and Laboratory Medicine, CORE Diagnostics, Gurgaon, Delhi, India.
Mallik V; Department of Pathology and Laboratory Medicine, CORE Diagnostics, Gurgaon, Delhi, India.
Jain D; Department of Pathology and Laboratory Medicine, CORE Diagnostics, Gurgaon, Delhi, India.
Jain E; Department of Pathology and Laboratory Medicine, CORE Diagnostics, Gurgaon, Delhi, India.
Chakrabarti I; Department of Pathology, AIIMS, Kalyani, West Bengal, India.
Varshney J; Department of Pathology and Laboratory Medicine, CORE Diagnostics, Gurgaon, Delhi, India.
Beg A; Department of Pathology and Laboratory Medicine, CORE Diagnostics, Gurgaon, Delhi, India.
Dixit M; Department of Pathology and Laboratory Medicine, CORE Diagnostics, Gurgaon, Delhi, India.
Baisakh MR; Department of Pathology, Prolife Diagnostics, Bhubaneswar, Odisha, India.
Naik S; Department of Pathology, Prolife Diagnostics, Bhubaneswar, Odisha, India.
Sahoo SK; Department of Pediatric Surgery, Institute of Medical Sciences and SUM Hospital, Bhubaneswar, Odisha, India.
Akgul M; Department of Pathology, Albany Medical Center, Albany, NY, USA.
Balzer BL; Department of Pathology and Laboratory Medicine, Cedars-Sinai Medical Center, Los Angeles, CA, USA.
Amin MB; Department of Pathology and Laboratory, University of Southern California Keck School, Los Angeles, CA, USA.
Parwani AV; Department of Pathology and Laboratory, Wexner Medical Center, Pathology, Columbus, OH, USA.
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Źródło:
International journal of surgical pathology [Int J Surg Pathol] 2024 Apr; Vol. 32 (2), pp. 294-303. Date of Electronic Publication: 2023 Jun 13.
Typ publikacji:
Journal Article
MeSH Terms:
Medulloblastoma*
Sarcoma, Synovial*/diagnosis
Sarcoma, Synovial*/genetics
Rhabdomyosarcoma, Embryonal*
Neuroblastoma*/diagnosis
Wilms Tumor*/diagnosis
Precursor Cell Lymphoblastic Leukemia-Lymphoma*
Cerebellar Neoplasms*
Kidney Neoplasms*/diagnosis
Kidney Neoplasms*/genetics
Humans ; Child ; Biomarkers, Tumor ; Diagnosis, Differential ; GATA3 Transcription Factor
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
P53 and BCL-2 family proteins PUMA and NOXA define competitive fitness in pluripotent cell competition.
Autorzy:
Valverde-Lopez JA; Cardiovascular Regeneration Program, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.
Li-Bao L; Cardiovascular Regeneration Program, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.
Sierra R; Cardiovascular Regeneration Program, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.; Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
Santos E; Pluripotent Cell Technology Unit, Centro Nacional de Investigaciones Cardiovasculares, CNIC, Madrid, Spain.
Giovinazzo G; Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.; Pluripotent Cell Technology Unit, Centro Nacional de Investigaciones Cardiovasculares, CNIC, Madrid, Spain.
Díaz-Díaz C; Cardiovascular Regeneration Program, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.; Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
Torres M; Cardiovascular Regeneration Program, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.; Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
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Źródło:
PLoS genetics [PLoS Genet] 2024 Mar 15; Vol. 20 (3), pp. e1011193. Date of Electronic Publication: 2024 Mar 15 (Print Publication: 2024).
Typ publikacji:
Journal Article
MeSH Terms:
Cell Competition*/genetics
Proto-Oncogene Proteins c-bcl-2*/genetics
Proto-Oncogene Proteins c-bcl-2*/metabolism
Tumor Suppressor Protein p53*/genetics
Tumor Suppressor Protein p53*/metabolism
Animals ; Apoptosis/genetics ; Apoptosis Regulatory Proteins/metabolism ; Tumor Suppressor Proteins/metabolism ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
NLCECA score: a serum inflammatory-tumor biomarker score to predict survival of advanced perihilar cholangiocarcinoma after hepatic arterial infusion chemotherapy.
Autorzy:
Fu S; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education/Beijing), Department of Interventional Oncology, Peking University Cancer Hospital & Institute, Beijing, 100142, China.
Li J; Center for Medical Device Evaluation, National Medical Products Administration, Beijing, 100053, China.
Fan H; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education/Beijing), Department of Cancer Epidemiology, Peking University Cancer Hospital & Institute, Beijing, 100142, China.
Zheng K; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education/Beijing), Department of Interventional Oncology, Peking University Cancer Hospital & Institute, Beijing, 100142, China.
Leng B; Hebei North University, Hebei, 075000, China.
Cao G; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education/Beijing), Department of Interventional Oncology, Peking University Cancer Hospital & Institute, Beijing, 100142, China.
Xu L; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education/Beijing), Department of Interventional Oncology, Peking University Cancer Hospital & Institute, Beijing, 100142, China.
Zhong Y; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education/Beijing), Department of Interventional Oncology, Peking University Cancer Hospital & Institute, Beijing, 100142, China.
Niu C; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education/Beijing), Department of Interventional Oncology, Peking University Cancer Hospital & Institute, Beijing, 100142, China.
Wang X; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education/Beijing), Department of Interventional Oncology, Peking University Cancer Hospital & Institute, Beijing, 100142, China. .
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Źródło:
Scientific reports [Sci Rep] 2024 Feb 23; Vol. 14 (1), pp. 4466. Date of Electronic Publication: 2024 Feb 23.
Typ publikacji:
Journal Article
MeSH Terms:
Klatskin Tumor*/drug therapy
Klatskin Tumor*/pathology
Cholangiocarcinoma*
Bile Duct Neoplasms*/pathology
Humans ; Biomarkers, Tumor ; Carcinoembryonic Antigen ; CA-19-9 Antigen ; Bile Ducts, Intrahepatic/pathology ; Retrospective Studies
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
High somatic mutations in circulating tumor DNA predict response of metastatic pancreatic ductal adenocarcinoma to first-line nab-paclitaxel plus S-1: prospective study.
Autorzy:
Huang L; Medical Center on Aging of Ruijin Hospital, MCARJH, Shanghai Jiaotong University School of Medicine, 197 Ruijin Er Road, Shanghai, 200025, China. .; Department of Oncology, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China. .
Lv Y; Department of Medical Oncology, Chinese PLA General Hospital, 28 Fuxing Road, Beijing, 100853, China.
Guan S; Department of Medical Oncology, Chinese PLA General Hospital, 28 Fuxing Road, Beijing, 100853, China.
Yan H; Department of Medical Oncology, Chinese PLA General Hospital, 28 Fuxing Road, Beijing, 100853, China.
Han L; Department of Medical Oncology, Chinese PLA General Hospital, 28 Fuxing Road, Beijing, 100853, China.
Wang Z; Department of Medical Oncology, Chinese PLA General Hospital, 28 Fuxing Road, Beijing, 100853, China. .
Han Q; Department of Medical Oncology, Chinese PLA General Hospital, 28 Fuxing Road, Beijing, 100853, China. .
Dai G; Department of Medical Oncology, Chinese PLA General Hospital, 28 Fuxing Road, Beijing, 100853, China. .
Shi Y; Department of General Surgery, Shanghai Seventh People's Hospital, Shanghai University of Traditional Chinese Medicine, 358 Datong Road, Gaoqiao Town, Shanghai, 200137, China. sy_.
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Źródło:
Journal of translational medicine [J Transl Med] 2024 Feb 20; Vol. 22 (1), pp. 184. Date of Electronic Publication: 2024 Feb 20.
Typ publikacji:
Journal Article
MeSH Terms:
Circulating Tumor DNA*/genetics
Pancreatic Neoplasms*/drug therapy
Pancreatic Neoplasms*/genetics
Pancreatic Neoplasms*/pathology
Adenocarcinoma*/genetics
Albumins*
Paclitaxel*
Humans ; Prospective Studies ; Prognosis ; CA-19-9 Antigen ; Proto-Oncogene Proteins p21(ras)/genetics ; Mutation/genetics ; Biomarkers, Tumor/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Circulating TNF receptor levels are associated with estimated glomerular filtration rate even in healthy individuals with normal kidney function.
Autorzy:
Gohda T; Department of Nephrology, Juntendo University Faculty of Medicine, Hongo 2-1-1, Bunkyo-Ku, Tokyo, 113-8421, Japan. .
Murakoshi M; Department of Nephrology, Juntendo University Faculty of Medicine, Hongo 2-1-1, Bunkyo-Ku, Tokyo, 113-8421, Japan.
Shibata T; Department of Nephrology, Juntendo University Faculty of Medicine, Hongo 2-1-1, Bunkyo-Ku, Tokyo, 113-8421, Japan.
Suzuki Y; Department of Nephrology, Juntendo University Faculty of Medicine, Hongo 2-1-1, Bunkyo-Ku, Tokyo, 113-8421, Japan.
Takemura H; Department of Clinical Laboratory, Juntendo University Hospital, Bunkyo-Ku, Tokyo, Japan.
Tsuchiya K; Department of Clinical Laboratory, Juntendo University Hospital, Bunkyo-Ku, Tokyo, Japan.
Okada T; Department of Clinical Laboratory, Juntendo University Hospital, Bunkyo-Ku, Tokyo, Japan.
Wakita M; Department of Clinical Laboratory, Juntendo University Hospital, Bunkyo-Ku, Tokyo, Japan.
Horiuchi Y; Department of Clinical Laboratory Medicine, Juntendo University Faculty of Medicine, Bunkyo-Ku, Tokyo, Japan.
Tabe Y; Department of Clinical Laboratory Medicine, Juntendo University Faculty of Medicine, Bunkyo-Ku, Tokyo, Japan.
Kamei N; Department of Endocrinology and Metabolism, Hiroshima Red Cross Hospital & Atomic-Bomb Survivors Hospital, Hiroshima, Japan.; Institute for Clinical Research, National Hospital Organization, Kure Medical Center and Chugoku Cancer Center, Hiroshima, Japan.
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Źródło:
Scientific reports [Sci Rep] 2024 Mar 27; Vol. 14 (1), pp. 7245. Date of Electronic Publication: 2024 Mar 27.
Typ publikacji:
Journal Article
MeSH Terms:
Receptors, Tumor Necrosis Factor, Type II*
Diabetes Mellitus, Type 2*/pathology
Male ; Humans ; Female ; Receptors, Tumor Necrosis Factor, Type I ; Glomerular Filtration Rate/physiology ; Kidney/pathology ; Biomarkers
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
AURKB promotes bladder cancer progression by deregulating the p53 DNA damage response pathway via MAD2L2.
Autorzy:
Li L; Department of Urology, Renmin Hospital of Wuhan University, Wuhan, 430060, China.
Jiang P; Department of Urology, Renmin Hospital of Wuhan University, Wuhan, 430060, China.
Hu W; Department of Urology, Renmin Hospital of Wuhan University, Wuhan, 430060, China.
Zou F; Department of Urology, Renmin Hospital of Wuhan University, Wuhan, 430060, China.
Li M; Department of Urology, Renmin Hospital of Wuhan University, Wuhan, 430060, China.
Rao T; Department of Urology, Renmin Hospital of Wuhan University, Wuhan, 430060, China.
Ruan Y; Department of Urology, Renmin Hospital of Wuhan University, Wuhan, 430060, China.
Yu W; Department of Urology, Renmin Hospital of Wuhan University, Wuhan, 430060, China.
Ning J; Department of Urology, Renmin Hospital of Wuhan University, Wuhan, 430060, China. .
Cheng F; Department of Urology, Renmin Hospital of Wuhan University, Wuhan, 430060, China. .
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Źródło:
Journal of translational medicine [J Transl Med] 2024 Mar 21; Vol. 22 (1), pp. 295. Date of Electronic Publication: 2024 Mar 21.
Typ publikacji:
Journal Article
MeSH Terms:
Tumor Suppressor Protein p53*/metabolism
Urinary Bladder Neoplasms*/genetics
Urinary Bladder Neoplasms*/pathology
Animals ; Humans ; Mice ; Aurora Kinase B/genetics ; Aurora Kinase B/metabolism ; Cell Line, Tumor ; Cell Proliferation/genetics ; DNA Repair ; Gene Expression Regulation, Neoplastic ; Mad2 Proteins/genetics ; Mad2 Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Tumor Necrosis Factor-α Receptor 1 Mediates Borna Disease Virus 1-Induced Changes in Peroxisomal and Mitochondrial Dynamics in Neurons.
Autorzy:
Osei D; Institute for Anatomy and Cell Biology, Justus Liebig University Giessen, 35392 Giessen, Germany.; Institute of Veterinary Pathology, Justus Liebig University Giessen, 35392 Giessen, Germany.
Baumgart-Vogt E; Institute for Anatomy and Cell Biology, Justus Liebig University Giessen, 35392 Giessen, Germany.
Ahlemeyer B; Institute for Anatomy and Cell Biology, Justus Liebig University Giessen, 35392 Giessen, Germany.
Herden C; Institute of Veterinary Pathology, Justus Liebig University Giessen, 35392 Giessen, Germany.; Center for Mind, Brain and Behavior, Justus Liebig University Giessen, 35392 Giessen, Germany.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2024 Feb 03; Vol. 25 (3). Date of Electronic Publication: 2024 Feb 03.
Typ publikacji:
Journal Article
MeSH Terms:
Receptors, Tumor Necrosis Factor, Type I*/genetics
Borna disease virus*
Mice ; Animals ; Tumor Necrosis Factor-alpha/physiology ; Catalase/genetics ; Antioxidants ; Mitochondrial Dynamics ; Mice, Knockout ; Neurons ; Mice, Inbred C57BL ; Mammals
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
SMARCB1/INI1-Deficient Poorly Differentiated Carcinoma of the Colon With Rhabdoid Features-A Rare Tumor With Serrated Phenotype: Case Report and Review of Literature.
Autorzy:
Maurya S; Department of Pathology, All India Institute of Medical Sciences, New Delhi, India.
Yadav S; Department of Pathology, All India Institute of Medical Sciences, New Delhi, India.
Bhowmik S; Department of Pathology, All India Institute of Medical Sciences, New Delhi, India.
Dhal J; Department of Surgery, All India Institute of Medical Sciences, New Delhi, India.
Mehra L; Department of Pathology, All India Institute of Medical Sciences, New Delhi, India.
Sharma R; Department of Radiology, All India Institute of Medical Sciences, New Delhi, India.
Krishna A; Department of Surgery, All India Institute of Medical Sciences, New Delhi, India.
Sharma A; Department of Medical Oncology, All India Institute of Medical Sciences, New Delhi, India.
Barwad A; Department of Pathology, All India Institute of Medical Sciences, New Delhi, India.
Das P; Department of Pathology, All India Institute of Medical Sciences, New Delhi, India.
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Źródło:
International journal of surgical pathology [Int J Surg Pathol] 2024 Feb; Vol. 32 (1), pp. 187-195. Date of Electronic Publication: 2023 May 01.
Typ publikacji:
Review; Case Reports; Journal Article
MeSH Terms:
Colorectal Neoplasms*
Neoplasms, Glandular and Epithelial*
Carcinoma*/genetics
Carcinoma*/pathology
Rhabdoid Tumor*/pathology
Brain Neoplasms*
Neoplastic Syndromes, Hereditary*
Humans ; Phenotype ; Prognosis ; Biomarkers, Tumor/genetics ; SMARCB1 Protein/genetics
SCR Disease Name:
Turcot syndrome
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Circulating Tumor DNA Is a Variant of Liquid Biopsy with Predictive and Prognostic Clinical Value in Breast Cancer Patients.
Autorzy:
Zavarykina TM; N.M. Emanuel Institute of Biochemical Physics of the Russian Academy of Sciences, Moscow 119334, Russia.; 'B.I. Kulakov National Medical Research Center of Obstetrics, Gynecology, and Perinatology of Ministry of Health of the Russian Federation, Moscow 117997, Russia.
Lomskova PK; N.M. Emanuel Institute of Biochemical Physics of the Russian Academy of Sciences, Moscow 119334, Russia.
Pronina IV; Institute of General Pathology and Pathophysiology, Moscow 125315, Russia.
Khokhlova SV; 'B.I. Kulakov National Medical Research Center of Obstetrics, Gynecology, and Perinatology of Ministry of Health of the Russian Federation, Moscow 117997, Russia.
Stenina MB; 'N.N. Blokhin National Medical Research Center of Oncology of Ministry of Health of the Russian Federation, Moscow 115522, Russia.
Sukhikh GT; 'B.I. Kulakov National Medical Research Center of Obstetrics, Gynecology, and Perinatology of Ministry of Health of the Russian Federation, Moscow 117997, Russia.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Dec 02; Vol. 24 (23). Date of Electronic Publication: 2023 Dec 02.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Circulating Tumor DNA*/genetics
Circulating Tumor DNA*/analysis
Breast Neoplasms*/diagnosis
Breast Neoplasms*/genetics
Breast Neoplasms*/pathology
Cell-Free Nucleic Acids*/genetics
Humans ; Female ; Prognosis ; Liquid Biopsy/methods ; Biomarkers, Tumor/genetics ; Biomarkers, Tumor/analysis ; Mutation
Czasopismo naukowe
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Tytuł:
Macrophage barrier in the tumor microenvironment and potential clinical applications.
Autorzy:
Ji S; Department of Urinary Surgery, The Shengjing Hospital of China Medical University, Shenyang, 110022, China.
Shi Y; Department of Respiratory Medicine, Shenyang 10th People's Hospital, Shenyang, 110096, China.
Yin B; Department of Urinary Surgery, The Shengjing Hospital of China Medical University, Shenyang, 110022, China. .
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Źródło:
Cell communication and signaling : CCS [Cell Commun Signal] 2024 Jan 26; Vol. 22 (1), pp. 74. Date of Electronic Publication: 2024 Jan 26.
Typ publikacji:
Video-Audio Media; Journal Article; Review
MeSH Terms:
Tumor Microenvironment*
Neoplasms*
Humans ; Macrophages ; Tumor-Associated Macrophages ; Immunotherapy ; Killer Cells, Natural
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Deciphering tumor microenvironment: CXCL9 and SPP1 as crucial determinants of tumor-associated macrophage polarity and prognostic indicators.
Autorzy:
Su X; Key Laboratory of Novel Targets and Drug Study for Neural Repair of Zhejiang Province, School of Medicine, Hangzhou City University, Hangzhou, Zhejiang, China.; Department of Clinical Medicine, Hangzhou City University, Hangzhou, Zhejiang, China.
Liang C; Key Laboratory of Novel Targets and Drug Study for Neural Repair of Zhejiang Province, School of Medicine, Hangzhou City University, Hangzhou, Zhejiang, China.; Department of Clinical Medicine, Hangzhou City University, Hangzhou, Zhejiang, China.
Chen R; Key Laboratory of Novel Targets and Drug Study for Neural Repair of Zhejiang Province, School of Medicine, Hangzhou City University, Hangzhou, Zhejiang, China.; Department of Clinical Medicine, Hangzhou City University, Hangzhou, Zhejiang, China.
Duan S; Key Laboratory of Novel Targets and Drug Study for Neural Repair of Zhejiang Province, School of Medicine, Hangzhou City University, Hangzhou, Zhejiang, China. .; Department of Clinical Medicine, Hangzhou City University, Hangzhou, Zhejiang, China. .
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Źródło:
Molecular cancer [Mol Cancer] 2024 Jan 13; Vol. 23 (1), pp. 13. Date of Electronic Publication: 2024 Jan 13.
Typ publikacji:
Letter; Research Support, Non-U.S. Gov't
MeSH Terms:
Tumor-Associated Macrophages*
Neoplasms*
Humans ; Prognosis ; Phenotype ; Tumor Microenvironment ; Chemokine CXCL9 ; Osteopontin
Raport
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Preliminary analysis of immune-related markers in thymic carcinoid.
Autorzy:
Zhang J; Department of Thoracic Surgery, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, 100730, China.
Guo C; Department of Thoracic Surgery, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, 100730, China.
Liu L; Department of Thoracic Surgery, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, 100730, China.
Zhao K; Department of Thoracic Surgery, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, 100730, China.
Zhou M; Department of Thoracic Surgery, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, 100730, China.
Chen Y; Department of Thoracic Surgery, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, 100730, China. .
Li S; Department of Thoracic Surgery, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, 100730, China. .
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Źródło:
Journal of translational medicine [J Transl Med] 2023 Dec 09; Vol. 21 (1), pp. 894. Date of Electronic Publication: 2023 Dec 09.
Typ publikacji:
Letter
MeSH Terms:
CD8-Positive T-Lymphocytes*/metabolism
Carcinoid Tumor*/metabolism
Humans ; B7-H1 Antigen/metabolism ; Tumor Microenvironment ; Lymphocytes, Tumor-Infiltrating/metabolism ; Prognosis
Opinia redakcyjna
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Knockouts of TNFRSF1A and TNFRSF1B Genes in K562 Cell Line Lead to Diverse Long-Lasting Responses to TNF-α.
Autorzy:
Perik-Zavodskaia O; Laboratory of Molecular Immunology, Research Institute of Fundamental and Clinical Immunology, 630099 Novosibirsk, Russia.
Alrhmoun S; Laboratory of Molecular Immunology, Research Institute of Fundamental and Clinical Immunology, 630099 Novosibirsk, Russia.; Laboratory of Immune Engineering, Federal State Autonomous Educational Institution of Higher Education I.M. Sechenov First Moscow State Medical University of the Ministry of Health of the Russian Federation (Sechenov University), 119991 Moscow, Russia.
Perik-Zavodskii R; Laboratory of Molecular Immunology, Research Institute of Fundamental and Clinical Immunology, 630099 Novosibirsk, Russia.; Laboratory of Immune Engineering, Federal State Autonomous Educational Institution of Higher Education I.M. Sechenov First Moscow State Medical University of the Ministry of Health of the Russian Federation (Sechenov University), 119991 Moscow, Russia.
Zhukova J; Laboratory of Molecular Immunology, Research Institute of Fundamental and Clinical Immunology, 630099 Novosibirsk, Russia.; Laboratory of Immune Engineering, Federal State Autonomous Educational Institution of Higher Education I.M. Sechenov First Moscow State Medical University of the Ministry of Health of the Russian Federation (Sechenov University), 119991 Moscow, Russia.
Lopatnikova J; Laboratory of Molecular Immunology, Research Institute of Fundamental and Clinical Immunology, 630099 Novosibirsk, Russia.; Laboratory of Immune Engineering, Federal State Autonomous Educational Institution of Higher Education I.M. Sechenov First Moscow State Medical University of the Ministry of Health of the Russian Federation (Sechenov University), 119991 Moscow, Russia.
Volynets M; Laboratory of Molecular Immunology, Research Institute of Fundamental and Clinical Immunology, 630099 Novosibirsk, Russia.
Alshevskaya A; Laboratory of Immune Engineering, Federal State Autonomous Educational Institution of Higher Education I.M. Sechenov First Moscow State Medical University of the Ministry of Health of the Russian Federation (Sechenov University), 119991 Moscow, Russia.
Sennikov S; Laboratory of Molecular Immunology, Research Institute of Fundamental and Clinical Immunology, 630099 Novosibirsk, Russia.; Laboratory of Immune Engineering, Federal State Autonomous Educational Institution of Higher Education I.M. Sechenov First Moscow State Medical University of the Ministry of Health of the Russian Federation (Sechenov University), 119991 Moscow, Russia.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Dec 06; Vol. 24 (24). Date of Electronic Publication: 2023 Dec 06.
Typ publikacji:
Journal Article
MeSH Terms:
Receptors, Tumor Necrosis Factor, Type I*/genetics
Receptors, Tumor Necrosis Factor, Type I*/metabolism
Tumor Necrosis Factor-alpha*/metabolism
Humans ; Receptors, Tumor Necrosis Factor, Type II/metabolism ; K562 Cells ; Cytokines/metabolism
Czasopismo naukowe
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Tytuł:
Cyclin B2 impairs the p53 signaling in nasopharyngeal carcinoma.
Autorzy:
Liu Q; Department of Otolaryngology, Qingdao Municipal Hospital, NO. 1, Shibei District, Jiaozhou Road, 266011, Qingdao, Shandong, P.R. China.
Yuan Y; Department of Otolaryngology, Qingdao Municipal Hospital, NO. 1, Shibei District, Jiaozhou Road, 266011, Qingdao, Shandong, P.R. China.
Shang X; Department of Otolaryngology, Qingdao Municipal Hospital, NO. 1, Shibei District, Jiaozhou Road, 266011, Qingdao, Shandong, P.R. China.
Xin L; Department of Otolaryngology, Qingdao Municipal Hospital, NO. 1, Shibei District, Jiaozhou Road, 266011, Qingdao, Shandong, P.R. China. .
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Źródło:
BMC cancer [BMC Cancer] 2024 Jan 02; Vol. 24 (1), pp. 25. Date of Electronic Publication: 2024 Jan 02.
Typ publikacji:
Journal Article
MeSH Terms:
Cyclin B2*/genetics
Cyclin B2*/metabolism
Nasopharyngeal Carcinoma*/genetics
Nasopharyngeal Carcinoma*/pathology
Nasopharyngeal Neoplasms*/genetics
Nasopharyngeal Neoplasms*/pathology
Tumor Suppressor Protein p53*/genetics
Tumor Suppressor Protein p53*/metabolism
Humans ; Cell Line, Tumor ; Cell Movement/genetics ; Cell Proliferation/genetics ; Epithelial-Mesenchymal Transition/genetics ; Gene Expression Regulation, Neoplastic ; Jumonji Domain-Containing Histone Demethylases/metabolism ; Signal Transduction/genetics
Czasopismo naukowe
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Tytuł:
Circulating Tumor DNA Reflects Histologic and Clinical Characteristics of Various Lymphoma Subtypes.
Autorzy:
Kim JJ; Department of Laboratory Medicine, Yongin Severance Hospital, Yonsei University College of Medicine, Yongin, Korea.
Kim HM; Department of Pathology, Yongin Severance Hospital, Yonsei University College of Medicine, Yongin, Korea.
Kim H; Department of Laboratory Medicine, Chung-Ang University Gwangmyung Hospital, Chung-Ang University College of Medicine, Gwangmyeong, Korea.
Kim SJ; Division of Hemato-Oncology, Department of Internal Medicine, Yongin Severance Hospital, Yonsei University College of Medicine, Yongin, Korea.
Lee ST; Department of Laboratory Medicine, Severance Hospital, Yonsei University College of Medicine, Seoul, Korea.
Choi JR; Department of Laboratory Medicine, Severance Hospital, Yonsei University College of Medicine, Seoul, Korea.
Shin S; Department of Laboratory Medicine, Severance Hospital, Yonsei University College of Medicine, Seoul, Korea.
Hwang DY; Division of Hematology, Department of Internal Medicine, Severance Hospital, Yonsei University College of Medicine, Seoul, Korea.
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Źródło:
Cancer research and treatment [Cancer Res Treat] 2024 Jan; Vol. 56 (1), pp. 314-323. Date of Electronic Publication: 2023 Jul 17.
Typ publikacji:
Journal Article
MeSH Terms:
Circulating Tumor DNA*/genetics
Lymphoma*
Humans ; Liquid Biopsy ; Mutation ; Biomarkers, Tumor/genetics ; High-Throughput Nucleotide Sequencing
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Soluble CD137: A Potential Prognostic Biomarker in Critically Ill Patients.
Autorzy:
Räth U; Department of Internal Medicine I, Gastroenterology, Hepatology, Endocrinology, Rheumatology, and Infectious Diseases, University Hospital Regensburg, 93053 Regensburg, Germany.
Mester P; Department of Internal Medicine I, Gastroenterology, Hepatology, Endocrinology, Rheumatology, and Infectious Diseases, University Hospital Regensburg, 93053 Regensburg, Germany.
Schwarz H; Department of Physiology, and Immunology Programme, Yong Loo Lin School of Medicine, National University of Singapore, MD9, Singapore 117597, Singapore.
Schmid S; Department of Internal Medicine I, Gastroenterology, Hepatology, Endocrinology, Rheumatology, and Infectious Diseases, University Hospital Regensburg, 93053 Regensburg, Germany.
Müller M; Department of Internal Medicine I, Gastroenterology, Hepatology, Endocrinology, Rheumatology, and Infectious Diseases, University Hospital Regensburg, 93053 Regensburg, Germany.
Buechler C; Department of Internal Medicine I, Gastroenterology, Hepatology, Endocrinology, Rheumatology, and Infectious Diseases, University Hospital Regensburg, 93053 Regensburg, Germany.
Pavel V; Department of Internal Medicine I, Gastroenterology, Hepatology, Endocrinology, Rheumatology, and Infectious Diseases, University Hospital Regensburg, 93053 Regensburg, Germany.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Dec 15; Vol. 24 (24). Date of Electronic Publication: 2023 Dec 15.
Typ publikacji:
Journal Article
MeSH Terms:
Critical Illness*
Sepsis*/diagnosis
Tumor Necrosis Factor Receptor Superfamily, Member 9*/blood
Tumor Necrosis Factor Receptor Superfamily, Member 9*/chemistry
Humans ; Biomarkers ; Prognosis ; Receptors, Tumor Necrosis Factor ; Renal Dialysis ; Systemic Inflammatory Response Syndrome
Czasopismo naukowe
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Tytuł:
Prognostic value of circulating tumor DNA in operable non-small cell lung cancer: a systematic review and reconstructed individual patient-data based meta-analysis.
Autorzy:
Chen D; Department of Cardiothoracic Surgery, the First Affiliated Hospital of Chongqing Medical University, 1# Youyi Road, Yuzhong District, Chongqing, 400016, The People's Republic of China. chen_.
Guo J; Department of Cardiothoracic Surgery, the First Affiliated Hospital of Chongqing Medical University, 1# Youyi Road, Yuzhong District, Chongqing, 400016, The People's Republic of China.
Huang H; Department of Cardiothoracic Surgery, the First Affiliated Hospital of Chongqing Medical University, 1# Youyi Road, Yuzhong District, Chongqing, 400016, The People's Republic of China.
Tian L; Department of Cardiothoracic Surgery, the First Affiliated Hospital of Chongqing Medical University, 1# Youyi Road, Yuzhong District, Chongqing, 400016, The People's Republic of China.
Xie Y; Department of Cardiothoracic Surgery, the First Affiliated Hospital of Chongqing Medical University, 1# Youyi Road, Yuzhong District, Chongqing, 400016, The People's Republic of China.
Wu Q; Department of Cardiothoracic Surgery, the First Affiliated Hospital of Chongqing Medical University, 1# Youyi Road, Yuzhong District, Chongqing, 400016, The People's Republic of China. .
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Źródło:
BMC medicine [BMC Med] 2023 Nov 27; Vol. 21 (1), pp. 467. Date of Electronic Publication: 2023 Nov 27.
Typ publikacji:
Systematic Review; Meta-Analysis; Journal Article
MeSH Terms:
Carcinoma, Non-Small-Cell Lung*/genetics
Carcinoma, Non-Small-Cell Lung*/surgery
Carcinoma, Non-Small-Cell Lung*/drug therapy
Circulating Tumor DNA*/genetics
Lung Neoplasms*/diagnosis
Lung Neoplasms*/genetics
Lung Neoplasms*/surgery
Humans ; Prognosis ; Progression-Free Survival ; Biomarkers, Tumor/genetics
Czasopismo naukowe
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Wyświetlanie 1-20 z 1072951

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