Informacja

Drogi użytkowniku, aplikacja do prawidłowego działania wymaga obsługi JavaScript. Proszę włącz obsługę JavaScript w Twojej przeglądarce.

English (EN)·Polski (PL)·Ukraiński (UK)
Przejdź do strony domowej biblioteki Uniwersytet Ekonomiczny we Wrocławiu Link otwiera się w nowym oknie Logo biblioteki Prolib Integro - strona głównaUniwersytet Ekonomiczny we Wrocławiu

Wyszukujesz frazę ""van Woerden GM"" wg kryterium: Autor

  Lista filtrów
Wyrównaj
    Wyświetlanie 1-16 z 16
    Tytuł:
    RHEB/mTOR hyperactivity causes cortical malformations and epileptic seizures through increased axonal connectivity.
    Autorzy:
    Proietti Onori M; Department of Neuroscience, Erasmus Medical Center, Rotterdam, the Netherlands.; The ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, the Netherlands.
    Koene LMC; Department of Neuroscience, Erasmus Medical Center, Rotterdam, the Netherlands.; The ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, the Netherlands.
    Schäfer CB; Department of Neuroscience, Erasmus Medical Center, Rotterdam, the Netherlands.
    Nellist M; Department of Clinical Genetics, Erasmus Medical Center, Rotterdam, Zuid Holland, the Netherlands.
    de Brito van Velze M; Department of Neuroscience, Erasmus Medical Center, Rotterdam, the Netherlands.
    Gao Z; Department of Neuroscience, Erasmus Medical Center, Rotterdam, the Netherlands.
    Elgersma Y; Department of Neuroscience, Erasmus Medical Center, Rotterdam, the Netherlands.; The ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, the Netherlands.; Department of Clinical Genetics, Erasmus Medical Center, Rotterdam, Zuid Holland, the Netherlands.
    van Woerden GM; Department of Neuroscience, Erasmus Medical Center, Rotterdam, the Netherlands.; The ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, the Netherlands.; Department of Clinical Genetics, Erasmus Medical Center, Rotterdam, Zuid Holland, the Netherlands.
    Pokaż więcej
    Źródło:
    PLoS biology [PLoS Biol] 2021 May 26; Vol. 19 (5), pp. e3001279. Date of Electronic Publication: 2021 May 26 (Print Publication: 2021).
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Ras Homolog Enriched in Brain Protein/*metabolism
    Seizures/*metabolism
    TOR Serine-Threonine Kinases/*metabolism
    Animals ; Axons/metabolism ; Brain/metabolism ; Disease Models, Animal ; Epilepsy/metabolism ; Epilepsy/physiopathology ; Mice ; Mice, Inbred C57BL ; Neurons/metabolism ; Seizures/physiopathology ; Signal Transduction ; Somatosensory Cortex/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Assessing the requirements of prenatal UBE3A expression for rescue of behavioral phenotypes in a mouse model for Angelman syndrome.
    Autorzy:
    Sonzogni M; Department of Neuroscience and the ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC University Medical Center, 3015 CN, Rotterdam, The Netherlands.
    Zhai P; Department of Neuroscience and the ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC University Medical Center, 3015 CN, Rotterdam, The Netherlands.; Department of Neurology, The First Affiliated Hospital of Henan University, No.357, Ximendajie Street, Kaifeng City, Henan Province, China.
    Mientjes EJ; Department of Neuroscience and the ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC University Medical Center, 3015 CN, Rotterdam, The Netherlands.
    van Woerden GM; Department of Neuroscience and the ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC University Medical Center, 3015 CN, Rotterdam, The Netherlands.
    Elgersma Y; Department of Neuroscience and the ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC University Medical Center, 3015 CN, Rotterdam, The Netherlands. .
    Pokaż więcej
    Źródło:
    Molecular autism [Mol Autism] 2020 Sep 18; Vol. 11 (1), pp. 70. Date of Electronic Publication: 2020 Sep 18.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Behavior, Animal*
    Gene Expression Regulation, Developmental*
    Phenotype*
    Angelman Syndrome/*genetics
    Ubiquitin-Protein Ligases/*genetics
    Alleles ; Animals ; Brain/embryology ; Brain/pathology ; Disease Models, Animal ; Embryonic Development/genetics ; Gene Silencing ; Integrases/metabolism ; Mice, Inbred C57BL ; Nestin/metabolism ; Ubiquitin-Protein Ligases/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    CaMKII controls neuromodulation via neuropeptide gene expression and axonal targeting of neuropeptide vesicles.
    Autorzy:
    Moro A; Department of Clinical Genetics, Center for Neurogenomics and Cognitive Research (CNCR), University Medical Center Amsterdam, Amsterdam, the Netherlands.; Department of Functional Genomics, Center for Neurogenomics and Cognitive Research (CNCR), Vrije Universiteit (VU) Amsterdam, Amsterdam, the Netherlands.
    van Woerden GM; Department of Neuroscience, ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC, Rotterdam, the Netherlands.
    Toonen RF; Department of Functional Genomics, Center for Neurogenomics and Cognitive Research (CNCR), Vrije Universiteit (VU) Amsterdam, Amsterdam, the Netherlands.
    Verhage M; Department of Clinical Genetics, Center for Neurogenomics and Cognitive Research (CNCR), University Medical Center Amsterdam, Amsterdam, the Netherlands.; Department of Functional Genomics, Center for Neurogenomics and Cognitive Research (CNCR), Vrije Universiteit (VU) Amsterdam, Amsterdam, the Netherlands.
    Pokaż więcej
    Źródło:
    PLoS biology [PLoS Biol] 2020 Aug 10; Vol. 18 (8), pp. e3000826. Date of Electronic Publication: 2020 Aug 10 (Print Publication: 2020).
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Astrocytes/*metabolism
    Brain-Derived Neurotrophic Factor/*genetics
    Calcium/*metabolism
    Calcium-Calmodulin-Dependent Protein Kinase Type 2/*genetics
    Neurons/*metabolism
    Protein Subunits/*genetics
    Animals ; Astrocytes/cytology ; Brain-Derived Neurotrophic Factor/metabolism ; Calcium-Calmodulin-Dependent Protein Kinase Type 2/deficiency ; Cyclic AMP Response Element-Binding Protein/genetics ; Cyclic AMP Response Element-Binding Protein/metabolism ; Feedback, Physiological ; Gene Expression Regulation ; Hippocampus/cytology ; Hippocampus/metabolism ; Mice ; Mice, Transgenic ; Neurons/cytology ; Phosphorylation ; Primary Cell Culture ; Protein Subunits/deficiency ; Synapses/physiology ; Synaptic Transmission ; Time-Lapse Imaging
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    The role of ubiquitin ligase E3A in polarized contact guidance and rescue strategies in UBE3A-deficient hippocampal neurons.
    Autorzy:
    Tonazzini I; Istituto Nanoscienze- Consiglio Nazionale delle Ricerche (CNR) & Scuola Normale Superiore, NEST, Piazza San Silvestro 12, 56127 Pisa, Italy.; 2Department of Neuroscience, ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC, Wytemaweg 80, 3000 CA Rotterdam, the Netherlands.
    Van Woerden GM; 2Department of Neuroscience, ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC, Wytemaweg 80, 3000 CA Rotterdam, the Netherlands.
    Masciullo C; Istituto Nanoscienze- Consiglio Nazionale delle Ricerche (CNR) & Scuola Normale Superiore, NEST, Piazza San Silvestro 12, 56127 Pisa, Italy.
    Mientjes EJ; 2Department of Neuroscience, ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC, Wytemaweg 80, 3000 CA Rotterdam, the Netherlands.
    Elgersma Y; 2Department of Neuroscience, ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC, Wytemaweg 80, 3000 CA Rotterdam, the Netherlands.
    Cecchini M; Istituto Nanoscienze- Consiglio Nazionale delle Ricerche (CNR) & Scuola Normale Superiore, NEST, Piazza San Silvestro 12, 56127 Pisa, Italy.
    Pokaż więcej
    Źródło:
    Molecular autism [Mol Autism] 2019 Nov 29; Vol. 10, pp. 41. Date of Electronic Publication: 2019 Nov 29 (Print Publication: 2019).
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Hippocampus/*pathology
    Neurons/*metabolism
    Ubiquitin-Protein Ligases/*deficiency
    Animals ; Axons/metabolism ; Cells, Cultured ; Cytoskeleton/metabolism ; Dendrites/metabolism ; Female ; Focal Adhesions/metabolism ; Growth Cones/metabolism ; Male ; Mice ; Ubiquitin-Protein Ligases/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Delayed loss of UBE3A reduces the expression of Angelman syndrome-associated phenotypes.
    Autorzy:
    Sonzogni M; 1Department of Neuroscience and the ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC University Medical Center, 3015 CN Rotterdam, The Netherlands.
    Hakonen J; 1Department of Neuroscience and the ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC University Medical Center, 3015 CN Rotterdam, The Netherlands.
    Bernabé Kleijn M; 1Department of Neuroscience and the ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC University Medical Center, 3015 CN Rotterdam, The Netherlands.
    Silva-Santos S; 1Department of Neuroscience and the ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC University Medical Center, 3015 CN Rotterdam, The Netherlands.
    Judson MC; 2Neuroscience Center, Department of Cell Biology and Physiology, and Carolina Institute for Developmental Disabilities, University of North Carolina, Chapel Hill, NC USA.
    Philpot BD; 2Neuroscience Center, Department of Cell Biology and Physiology, and Carolina Institute for Developmental Disabilities, University of North Carolina, Chapel Hill, NC USA.
    van Woerden GM; 1Department of Neuroscience and the ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC University Medical Center, 3015 CN Rotterdam, The Netherlands.
    Elgersma Y; 1Department of Neuroscience and the ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus MC University Medical Center, 3015 CN Rotterdam, The Netherlands.
    Pokaż więcej
    Źródło:
    Molecular autism [Mol Autism] 2019 May 22; Vol. 10, pp. 23. Date of Electronic Publication: 2019 May 22 (Print Publication: 2019).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Angelman Syndrome/*genetics
    Angelman Syndrome/*pathology
    Ubiquitin-Protein Ligases/*deficiency
    Aging/pathology ; Animals ; Female ; Gene Deletion ; Male ; Mice, Inbred C57BL ; Phenotype ; Ubiquitin-Protein Ligases/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    A behavioral test battery for mouse models of Angelman syndrome: a powerful tool for testing drugs and novel Ube3a mutants.
    Autorzy:
    Sonzogni M; 1Department of Neuroscience, Erasmus Medical Center, Rotterdam, Netherlands.; 2ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, Netherlands.
    Wallaard I; 1Department of Neuroscience, Erasmus Medical Center, Rotterdam, Netherlands.; 2ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, Netherlands.
    Santos SS; 1Department of Neuroscience, Erasmus Medical Center, Rotterdam, Netherlands.; 2ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, Netherlands.
    Kingma J; 1Department of Neuroscience, Erasmus Medical Center, Rotterdam, Netherlands.; 2ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, Netherlands.
    du Mee D; 1Department of Neuroscience, Erasmus Medical Center, Rotterdam, Netherlands.; 2ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, Netherlands.
    van Woerden GM; 1Department of Neuroscience, Erasmus Medical Center, Rotterdam, Netherlands.; 2ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, Netherlands.
    Elgersma Y; 1Department of Neuroscience, Erasmus Medical Center, Rotterdam, Netherlands.; 2ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, Netherlands.
    Pokaż więcej
    Źródło:
    Molecular autism [Mol Autism] 2018 Sep 14; Vol. 9, pp. 47. Date of Electronic Publication: 2018 Sep 14 (Print Publication: 2018).
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Behavior Rating Scale*
    Angelman Syndrome/*genetics
    Angelman Syndrome/*psychology
    Ubiquitin-Protein Ligases/*genetics
    Animals ; Behavior, Animal/drug effects ; Disease Models, Animal ; Female ; Levodopa/pharmacology ; Male ; Mice, Mutant Strains ; Minocycline/pharmacology ; Mutation ; Phenotype
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    The molecular, temporal and region-specific requirements of the beta isoform of Calcium/Calmodulin-dependent protein kinase type 2 (CAMK2B) in mouse locomotion.
    Autorzy:
    Kool MJ; Department of Neuroscience, Erasmus University Medical Center, 3015 CN, Rotterdam, The Netherlands.; ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus University Medical Center, 3015 CN, Rotterdam, The Netherlands.
    van de Bree JE; Department of Neuroscience, Erasmus University Medical Center, 3015 CN, Rotterdam, The Netherlands.; ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus University Medical Center, 3015 CN, Rotterdam, The Netherlands.
    Bodde HE; Department of Neuroscience, Erasmus University Medical Center, 3015 CN, Rotterdam, The Netherlands.
    Elgersma Y; Department of Neuroscience, Erasmus University Medical Center, 3015 CN, Rotterdam, The Netherlands.; ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus University Medical Center, 3015 CN, Rotterdam, The Netherlands.
    van Woerden GM; Department of Neuroscience, Erasmus University Medical Center, 3015 CN, Rotterdam, The Netherlands.; ENCORE Expertise Center for Neurodevelopmental Disorders, Erasmus University Medical Center, 3015 CN, Rotterdam, The Netherlands.
    Pokaż więcej
    Źródło:
    Scientific reports [Sci Rep] 2016 May 31; Vol. 6, pp. 26989. Date of Electronic Publication: 2016 May 31.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Mutation*
    Association Learning/*physiology
    Calcium/*metabolism
    Calcium-Calmodulin-Dependent Protein Kinase Type 2/*genetics
    Spatial Learning/*physiology
    Animals ; Brain Mapping ; Calcium-Calmodulin-Dependent Protein Kinase Type 2/deficiency ; Calmodulin/genetics ; Calmodulin/metabolism ; Cerebellum/metabolism ; Cerebellum/physiopathology ; Corpus Striatum/metabolism ; Corpus Striatum/physiopathology ; Female ; Gene Expression ; Isoenzymes/deficiency ; Isoenzymes/genetics ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Transgenic ; Prosencephalon/metabolism ; Prosencephalon/physiopathology ; Rotarod Performance Test
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Neurofibromin regulates HCN activity in Parvalbumin-positive interneurons.
    Autorzy:
    Omrani A; Department of Neuroscience, Erasmus Medical Center, Rotterdam, The Netherlands.; ENCORE Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, The Netherlands.
    van Woerden GM; Department of Neuroscience, Erasmus Medical Center, Rotterdam, The Netherlands.; ENCORE Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, The Netherlands.
    Elgersma Y; Department of Neuroscience, Erasmus Medical Center, Rotterdam, The Netherlands.; ENCORE Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, The Netherlands.
    Pokaż więcej
    Źródło:
    Molecular psychiatry [Mol Psychiatry] 2015 Nov; Vol. 20 (11), pp. 1263.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Interneurons/*drug effects
    Interneurons/*metabolism
    Neurofibromin 1/*metabolism
    Parvalbumins/*metabolism
    Animals ; Hippocampus/cytology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    HCN channels are a novel therapeutic target for cognitive dysfunction in Neurofibromatosis type 1.
    Autorzy:
    Omrani A; Department of Neuroscience, Erasmus Medical Center, Rotterdam, The Netherlands.; ENCORE Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, The Netherlands.
    van der Vaart T; Department of Neuroscience, Erasmus Medical Center, Rotterdam, The Netherlands.; ENCORE Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, The Netherlands.; Department of Pediatrics, Erasmus Medical Center, Sophia Children's Hospital, Rotterdam, The Netherlands.
    Mientjes E; Department of Neuroscience, Erasmus Medical Center, Rotterdam, The Netherlands.; ENCORE Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, The Netherlands.
    van Woerden GM; Department of Neuroscience, Erasmus Medical Center, Rotterdam, The Netherlands.; ENCORE Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, The Netherlands.
    Hojjati MR; Department of Neuroscience, Erasmus Medical Center, Rotterdam, The Netherlands.; Department of Physiology, Shahrekord University of Medical Sciences, Shahrekord, Iran.
    Li KW; Department of Molecular and Cellular Neurobiology, CNCR, Neuroscience Campus Amsterdam, VU University, Amsterdam, The Netherlands.
    Gutmann DH; Department of Neurology, Washington University School of Medicine, St. Louis, MO, USA.
    Levelt CN; Department of Molecular Visual Plasticity, Netherlands Institute for Neuroscience, Royal Netherlands Academy of Arts and Sciences (KNAW), Amsterdam, The Netherlands.
    Smit AB; Department of Molecular and Cellular Neurobiology, CNCR, Neuroscience Campus Amsterdam, VU University, Amsterdam, The Netherlands.
    Silva AJ; Department of Neurobiology, Brain Research Institute, University of California Los Angeles, Los Angeles, CA, USA.
    Kushner SA; ENCORE Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, The Netherlands.; Department of Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands.
    Elgersma Y; Department of Neuroscience, Erasmus Medical Center, Rotterdam, The Netherlands.; ENCORE Center for Neurodevelopmental Disorders, Erasmus Medical Center, Rotterdam, The Netherlands.
    Pokaż więcej
    Źródło:
    Molecular psychiatry [Mol Psychiatry] 2015 Nov; Vol. 20 (11), pp. 1311-21. Date of Electronic Publication: 2015 Apr 28.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
    MeSH Terms:
    Cognition Disorders/*drug therapy
    Cognition Disorders/*metabolism
    Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels/*metabolism
    Neurofibromatosis 1/*complications
    Potassium Channels/*metabolism
    Animals ; Cognition Disorders/etiology ; Cognition Disorders/genetics ; Disease Models, Animal ; Excitatory Amino Acid Antagonists/therapeutic use ; Excitatory Postsynaptic Potentials/drug effects ; Excitatory Postsynaptic Potentials/genetics ; Hippocampus/cytology ; Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels/genetics ; Lamotrigine ; Male ; Maze Learning/drug effects ; Maze Learning/physiology ; Mice ; Mice, Inbred C57BL ; Mice, Transgenic ; Motor Activity/drug effects ; Motor Activity/genetics ; Mutation/genetics ; Neural Inhibition/drug effects ; Neural Inhibition/genetics ; Neurofibromatosis 1/genetics ; Neurofibromin 1/genetics ; Neurofibromin 1/metabolism ; Neuronal Plasticity/drug effects ; Neuronal Plasticity/genetics ; Neurons/drug effects ; Neurons/metabolism ; Potassium Channels/genetics ; Pyrimidines/pharmacology ; Signal Transduction/drug effects ; Signal Transduction/genetics ; Triazines/therapeutic use
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Non-associative potentiation of perisomatic inhibition alters the temporal coding of neocortical layer 5 pyramidal neurons.
    Autorzy:
    Lourenço J; European Brain Research Institute, Rome, Italy; Sorbonne Universités UPMC Univ. Paris 06, UMR S 1127, Paris, France; Inserm U 1127, Paris, France; CNRS UMR 7225, Paris, France; ICM- Institut du Cerveau et de la Moelle épinière, Paris, France.
    Pacioni S; European Brain Research Institute, Rome, Italy.
    Rebola N; CNRS UMR 3571, Paris, France; Institut Pasteur, Unit of Dynamic Neuronal Imaging, Paris, France.
    van Woerden GM; European Brain Research Institute, Rome, Italy; Sorbonne Universités UPMC Univ. Paris 06, UMR S 1127, Paris, France; Inserm U 1127, Paris, France; CNRS UMR 7225, Paris, France; ICM- Institut du Cerveau et de la Moelle épinière, Paris, France.
    Marinelli S; European Brain Research Institute, Rome, Italy.
    DiGregorio D; CNRS UMR 3571, Paris, France; Institut Pasteur, Unit of Dynamic Neuronal Imaging, Paris, France.
    Bacci A; European Brain Research Institute, Rome, Italy; Sorbonne Universités UPMC Univ. Paris 06, UMR S 1127, Paris, France; Inserm U 1127, Paris, France; CNRS UMR 7225, Paris, France; ICM- Institut du Cerveau et de la Moelle épinière, Paris, France.
    Pokaż więcej
    Źródło:
    PLoS biology [PLoS Biol] 2014 Jul 08; Vol. 12 (7), pp. e1001903. Date of Electronic Publication: 2014 Jul 08 (Print Publication: 2014).
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Pyramidal Cells/*physiology
    Action Potentials/physiology ; Animals ; Calcium Channels, L-Type/physiology ; Long-Term Potentiation/physiology ; Long-Term Synaptic Depression/physiology ; Mice, Inbred C57BL ; Neocortex/cytology ; Neuronal Plasticity/physiology ; Neurons ; Patch-Clamp Techniques ; gamma-Aminobutyric Acid/physiology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Motor deficits in neurofibromatosis type 1 mice: the role of the cerebellum.
    Autorzy:
    van der Vaart T; Department of Neuroscience, Erasmus Medical Center, Rotterdam, The Netherlands.
    van Woerden GM
    Elgersma Y
    de Zeeuw CI
    Schonewille M
    Pokaż więcej
    Źródło:
    Genes, brain, and behavior [Genes Brain Behav] 2011 Jun; Vol. 10 (4), pp. 404-9. Date of Electronic Publication: 2011 Mar 22.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Cerebellum/*physiopathology
    Motor Activity/*genetics
    Motor Skills Disorders/*etiology
    Neurofibromatosis 1/*genetics
    Purkinje Cells/*physiology
    Animals ; Eye Movements/genetics ; Genes, Neurofibromatosis 1 ; Hand Strength/physiology ; Heterozygote ; Learning/physiology ; Mice ; Mice, Neurologic Mutants ; Motor Skills Disorders/genetics ; Motor Skills Disorders/physiopathology ; Neurofibromin 1/genetics ; Rotarod Performance Test
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses.
    Autorzy:
    van Woerden GM; Department of Neuroscience, Erasmus University Medical Center, Rotterdam, The Netherlands.
    Hoebeek FE
    Gao Z
    Nagaraja RY
    Hoogenraad CC
    Kushner SA
    Hansel C
    De Zeeuw CI
    Elgersma Y
    Pokaż więcej
    Źródło:
    Nature neuroscience [Nat Neurosci] 2009 Jul; Vol. 12 (7), pp. 823-5. Date of Electronic Publication: 2009 Jun 07.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Calcium-Calmodulin-Dependent Protein Kinase Type 2/*metabolism
    Neuronal Plasticity/*physiology
    Purkinje Cells/*physiology
    Synapses/*physiology
    Animals ; Calcineurin/metabolism ; Calcium-Calmodulin-Dependent Protein Kinase Type 2/genetics ; Cyclosporine/pharmacology ; Electric Stimulation ; Enzyme Inhibitors/pharmacology ; In Vitro Techniques ; Long-Term Potentiation/physiology ; Long-Term Synaptic Depression/drug effects ; Long-Term Synaptic Depression/physiology ; Mice ; Mice, Knockout ; Models, Neurological ; Patch-Clamp Techniques
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Rapid changes in hippocampal CA1 pyramidal cell function via pre- as well as postsynaptic membrane mineralocorticoid receptors.
    Autorzy:
    Olijslagers JE; SILS-CNS, University of Amsterdam, Kruislaan 320, 1098 SM Amsterdam, the Netherlands.
    de Kloet ER
    Elgersma Y
    van Woerden GM
    Joëls M
    Karst H
    Pokaż więcej
    Źródło:
    The European journal of neuroscience [Eur J Neurosci] 2008 May; Vol. 27 (10), pp. 2542-50.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Adrenal Cortex Hormones/*metabolism
    Hippocampus/*metabolism
    Pyramidal Cells/*metabolism
    Receptors, Mineralocorticoid/*metabolism
    Synaptic Membranes/*metabolism
    Synaptic Transmission/*physiology
    Animals ; Enzyme Inhibitors/pharmacology ; Excitatory Postsynaptic Potentials/drug effects ; Excitatory Postsynaptic Potentials/physiology ; Guanosine Diphosphate/analogs & derivatives ; Guanosine Diphosphate/pharmacology ; Hippocampus/ultrastructure ; MAP Kinase Signaling System/drug effects ; MAP Kinase Signaling System/physiology ; Male ; Membrane Potentials/drug effects ; Membrane Potentials/genetics ; Mice ; Mice, Inbred C57BL ; Mitogen-Activated Protein Kinase 3/drug effects ; Mitogen-Activated Protein Kinase 3/metabolism ; Neuronal Plasticity/drug effects ; Neuronal Plasticity/physiology ; Organ Culture Techniques ; Patch-Clamp Techniques ; Pyramidal Cells/drug effects ; Pyramidal Cells/ultrastructure ; Receptors, G-Protein-Coupled/drug effects ; Receptors, G-Protein-Coupled/metabolism ; Receptors, Mineralocorticoid/drug effects ; Stress, Physiological/metabolism ; Stress, Physiological/physiopathology ; Synaptic Membranes/drug effects ; Synaptic Transmission/drug effects ; Thionucleotides/pharmacology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Kinase activity is not required for alphaCaMKII-dependent presynaptic plasticity at CA3-CA1 synapses.
    Autorzy:
    Hojjati MR; Department of Neuroscience, Erasmus University Medical Center, Dr. Molewaterplein 50, 3015 GE Rotterdam, The Netherlands.
    van Woerden GM
    Tyler WJ
    Giese KP
    Silva AJ
    Pozzo-Miller L
    Elgersma Y
    Pokaż więcej
    Źródło:
    Nature neuroscience [Nat Neurosci] 2007 Sep; Vol. 10 (9), pp. 1125-7. Date of Electronic Publication: 2007 Jul 29.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Calcium-Calmodulin-Dependent Protein Kinase Type 2/*physiology
    Hippocampus/*cytology
    Neuronal Plasticity/*physiology
    Neurons/*physiology
    Presynaptic Terminals/*physiology
    Synapses/*physiology
    Synaptic Transmission/*physiology
    Analysis of Variance ; Animals ; Calcium-Calmodulin-Dependent Protein Kinase Type 2/genetics ; Electric Stimulation ; Enzyme Activation/genetics ; Excitatory Postsynaptic Potentials/genetics ; Mice ; Mice, Inbred C57BL ; Mice, Transgenic ; Microscopy, Electron, Transmission/methods ; Mutagenesis/physiology ; Neurons/radiation effects ; Patch-Clamp Techniques/methods ; Phosphorylation ; Presynaptic Terminals/ultrastructure ; Synapses/ultrastructure ; Synapsins/metabolism ; Synaptic Transmission/genetics ; Synaptic Vesicles/physiology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Rescue of neurological deficits in a mouse model for Angelman syndrome by reduction of alphaCaMKII inhibitory phosphorylation.
    Autorzy:
    van Woerden GM; Erasmus MC, University Medical Centre, Department of Neuroscience, Dr. Molewaterplein 50, PO Box 2040, 3000 CA, Rotterdam, The Netherlands.
    Harris KD
    Hojjati MR
    Gustin RM
    Qiu S
    de Avila Freire R
    Jiang YH
    Elgersma Y
    Weeber EJ
    Pokaż więcej
    Źródło:
    Nature neuroscience [Nat Neurosci] 2007 Mar; Vol. 10 (3), pp. 280-2. Date of Electronic Publication: 2007 Jan 28.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Angelman Syndrome/*complications
    Calcium-Calmodulin-Dependent Protein Kinase Type 2/*metabolism
    Mental Disorders/*etiology
    Mental Disorders/*therapy
    Angelman Syndrome/genetics ; Angelman Syndrome/pathology ; Animals ; Behavior, Animal ; Calcium-Calmodulin-Dependent Protein Kinase Type 2/genetics ; Conditioning, Classical/physiology ; Disease Models, Animal ; Excitatory Postsynaptic Potentials/genetics ; Female ; Freezing Reaction, Cataleptic/physiology ; Hippocampus/physiopathology ; In Vitro Techniques ; Male ; Maze Learning/physiology ; Mental Disorders/genetics ; Mental Disorders/pathology ; Mice ; Mice, Inbred C57BL ; Mice, Neurologic Mutants ; Motor Activity/physiology ; Phosphorylation ; Phosphotransferases/metabolism ; Reaction Time ; Time Factors ; Ubiquitin-Protein Ligases/genetics
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
      Wyświetlanie 1-16 z 16

      Ta witryna wykorzystuje pliki cookies do przechowywania informacji na Twoim komputerze. Pliki cookies stosujemy w celu świadczenia usług na najwyższym poziomie, w tym w sposób dostosowany do indywidualnych potrzeb. Korzystanie z witryny bez zmiany ustawień dotyczących cookies oznacza, że będą one zamieszczane w Twoim komputerze. W każdym momencie możesz dokonać zmiany ustawień dotyczących cookies