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Tytuł:
Fractalkine isoforms differentially regulate microglia-mediated inflammation and enhance visual function in the diabetic retina.
Autorzy:
Rodriguez D; Department of Molecular Microbiology and Immunology, UTSA Circle, The University of Texas at San Antonio, San Antonio, TX, 78249, USA.
Church KA; Department of Molecular Microbiology and Immunology, UTSA Circle, The University of Texas at San Antonio, San Antonio, TX, 78249, USA.
Pietramale AN; Department of Molecular Microbiology and Immunology, UTSA Circle, The University of Texas at San Antonio, San Antonio, TX, 78249, USA.
Cardona SM; Department of Molecular Microbiology and Immunology, UTSA Circle, The University of Texas at San Antonio, San Antonio, TX, 78249, USA.
Vanegas D; Department of Molecular Microbiology and Immunology, UTSA Circle, The University of Texas at San Antonio, San Antonio, TX, 78249, USA.
Rorex C; Department of Molecular Microbiology and Immunology, UTSA Circle, The University of Texas at San Antonio, San Antonio, TX, 78249, USA.
Leary MC; Department of Molecular Microbiology and Immunology, UTSA Circle, The University of Texas at San Antonio, San Antonio, TX, 78249, USA.
Muzzio IA; Department of Psychological and Brain Sciences, University of Iowa, Iowa City, IA, 52242, USA.
Nash KR; Department of Molecular Pharmacology and Physiology, University of South Florida, Tampa, FL, 33620, USA.
Cardona AE; Department of Molecular Microbiology and Immunology, UTSA Circle, The University of Texas at San Antonio, San Antonio, TX, 78249, USA. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 04; Vol. 21 (1), pp. 42. Date of Electronic Publication: 2024 Feb 04.
Typ publikacji:
Journal Article
MeSH Terms:
Chemokine CX3CL1*/genetics
Chemokine CX3CL1*/metabolism
CX3C Chemokine Receptor 1*/genetics
CX3C Chemokine Receptor 1*/metabolism
Diabetes Mellitus*/metabolism
Animals ; Humans ; Mice ; Immunologic Factors ; Inflammation/metabolism ; Microglia/metabolism ; Protein Isoforms ; Retina/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CX3CR1 deficiency aggravates amyloid driven neuronal pathology and cognitive decline in Alzheimer's disease.
Autorzy:
Puntambekar SS; Stark Neurosciences Research Institute, Indiana University-School of Medicine, Indianapolis, IN, USA. .; Department of Medical and Molecular Genetics, Indiana University-School of Medicine, Indianapolis, IN, USA. .
Moutinho M; Stark Neurosciences Research Institute, Indiana University-School of Medicine, Indianapolis, IN, USA.; Department of Anatomy, Cell Biology and Physiology, Indiana University-School of Medicine, Indianapolis, IN, USA.
Lin PB; Stark Neurosciences Research Institute, Indiana University-School of Medicine, Indianapolis, IN, USA.; Indiana Biomedical Gateway (IBMG) Program, Indiana University-School of Medicine, Indianapolis, IN, USA.
Jadhav V; Stark Neurosciences Research Institute, Indiana University-School of Medicine, Indianapolis, IN, USA.; Department of Medicine, Division of Gerontology and Geriatric Medicine, University of Washington, Seattle, WA, USA.
Tumbleson-Brink D; Stark Neurosciences Research Institute, Indiana University-School of Medicine, Indianapolis, IN, USA.; Department of Medical and Molecular Genetics, Indiana University-School of Medicine, Indianapolis, IN, USA.
Balaji A; Stark Neurosciences Research Institute, Indiana University-School of Medicine, Indianapolis, IN, USA.; Indiana Clinical and Translational Institute (CTSI), Summer Research Program (SRP), Indianapolis, IN, USA.
Benito MA; Stark Neurosciences Research Institute, Indiana University-School of Medicine, Indianapolis, IN, USA.; Department of Medical and Molecular Genetics, Indiana University-School of Medicine, Indianapolis, IN, USA.
Xu G; Stark Neurosciences Research Institute, Indiana University-School of Medicine, Indianapolis, IN, USA.; Department of Medical and Molecular Genetics, Indiana University-School of Medicine, Indianapolis, IN, USA.
Oblak A; Stark Neurosciences Research Institute, Indiana University-School of Medicine, Indianapolis, IN, USA.; Department of Radiology, Indiana University-School of Medicine, Indianapolis, IN, USA.
Lasagna-Reeves CA; Stark Neurosciences Research Institute, Indiana University-School of Medicine, Indianapolis, IN, USA.; Department of Anatomy, Cell Biology and Physiology, Indiana University-School of Medicine, Indianapolis, IN, USA.
Landreth GE; Stark Neurosciences Research Institute, Indiana University-School of Medicine, Indianapolis, IN, USA.; Department of Anatomy, Cell Biology and Physiology, Indiana University-School of Medicine, Indianapolis, IN, USA.
Lamb BT; Stark Neurosciences Research Institute, Indiana University-School of Medicine, Indianapolis, IN, USA. .; Department of Medical and Molecular Genetics, Indiana University-School of Medicine, Indianapolis, IN, USA. .
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Źródło:
Molecular neurodegeneration [Mol Neurodegener] 2022 Jun 28; Vol. 17 (1), pp. 47. Date of Electronic Publication: 2022 Jun 28.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
MeSH Terms:
Alzheimer Disease*/metabolism
Alzheimer Disease*/pathology
Amyloidosis*/metabolism
CX3C Chemokine Receptor 1*/deficiency
CX3C Chemokine Receptor 1*/metabolism
Cognitive Dysfunction*
Amyloidogenic Proteins/metabolism ; Animals ; Mice ; Neurons/metabolism ; Neurons/pathology ; Plaque, Amyloid ; Transforming Growth Factor beta
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Deletion of Slc9a1 in Cx3cr1 cells stimulated microglial subcluster CREB1 signaling and microglia-oligodendrocyte crosstalk.
Autorzy:
Song S; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA. .; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA. .; Veterans Affairs Pittsburgh Health Care System, Pittsburgh, PA, 15213, USA. .
Oft H; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA.; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA.
Metwally S; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA.; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA.
Paruchuri S; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA.; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA.
Bielanin J; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA.; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA.
Fiesler V; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA.; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA.
Sneiderman C; Department of Neurological Surgery, University of Pittsburgh, Pittsburgh, PA, USA.
Kohanbash G; Department of Neurological Surgery, University of Pittsburgh, Pittsburgh, PA, USA.
Sun D; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA. .; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA. .; Veterans Affairs Pittsburgh Health Care System, Pittsburgh, PA, 15213, USA. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Mar 20; Vol. 21 (1), pp. 69. Date of Electronic Publication: 2024 Mar 20.
Typ publikacji:
Journal Article
MeSH Terms:
Brain*/metabolism
Microglia*/metabolism
Sodium-Hydrogen Exchanger 1*/metabolism
Animals ; Mice ; CX3C Chemokine Receptor 1/metabolism ; Macrophages/metabolism ; Oligodendroglia/metabolism ; Signal Transduction/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Involvement of CX3CR1 cells appearing in the abdominal cavity in the immunosuppressive environment immediately after gastric cancer surgery.
Autorzy:
Natsuki S; Department of Gastroenterological Surgery, Osaka City University Graduate School of Medicine, Asahi-machi 1-4-3, Abeno-ku, Osaka, Japan.
Yoshii M; Department of Gastroenterological Surgery, Osaka City University Graduate School of Medicine, Asahi-machi 1-4-3, Abeno-ku, Osaka, Japan. .
Tanaka H; Department of Surgery, Fuchu Hospital, Osaka, Japan.
Mori T; Department of Gastroenterological Surgery, Osaka City University Graduate School of Medicine, Asahi-machi 1-4-3, Abeno-ku, Osaka, Japan.
Deguchi S; Department of Gastroenterological Surgery, Osaka City University Graduate School of Medicine, Asahi-machi 1-4-3, Abeno-ku, Osaka, Japan.
Miki Y; Department of Gastroenterological Surgery, Osaka City University Graduate School of Medicine, Asahi-machi 1-4-3, Abeno-ku, Osaka, Japan.
Tamura T; Department of Gastroenterological Surgery, Osaka City University Graduate School of Medicine, Asahi-machi 1-4-3, Abeno-ku, Osaka, Japan.
Toyokawa T; Department of Gastroenterological Surgery, Osaka City University Graduate School of Medicine, Asahi-machi 1-4-3, Abeno-ku, Osaka, Japan.
Lee S; Department of Gastroenterological Surgery, Osaka City University Graduate School of Medicine, Asahi-machi 1-4-3, Abeno-ku, Osaka, Japan.
Maeda K; Department of Gastroenterological Surgery, Osaka City University Graduate School of Medicine, Asahi-machi 1-4-3, Abeno-ku, Osaka, Japan.
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Źródło:
World journal of surgical oncology [World J Surg Oncol] 2024 Mar 04; Vol. 22 (1), pp. 74. Date of Electronic Publication: 2024 Mar 04.
Typ publikacji:
Journal Article
MeSH Terms:
Stomach Neoplasms*/surgery
Abdominal Cavity*
Humans ; Gastrectomy ; Cytokines ; Immunosuppressive Agents ; Inflammation ; CX3C Chemokine Receptor 1
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
High TPX2 expression results in poor prognosis, and Sp1 mediates the coupling of the CX3CR1/CXCL10 chemokine pathway to the PI3K/Akt pathway through targeted inhibition of TPX2 in endometrial cancer.
Autorzy:
Yang M; Department of Obstetrics and Gynecology, Xiangyang Central Hospital, Affiliated Hospital of Hubei, University of Arts and Science, Xiangyang, China.; Institute of Maternity Disease, Xiangyang Central Hospital, Affiliated Hospital of Hubei University of Arts and Science, Xiangyang, China.
Mao X; Department of Obstetrics and Gynecology, Xiangyang Central Hospital, Affiliated Hospital of Hubei, University of Arts and Science, Xiangyang, China.
Li L; Department of Obstetrics and Gynecology, Xiangyang Central Hospital, Affiliated Hospital of Hubei, University of Arts and Science, Xiangyang, China.
Yang J; Department of Obstetrics and Gynecology, Xiangyang Central Hospital, Affiliated Hospital of Hubei, University of Arts and Science, Xiangyang, China.
Xing H; Department of Obstetrics and Gynecology, Xiangyang Central Hospital, Affiliated Hospital of Hubei, University of Arts and Science, Xiangyang, China.; Institute of Maternity Disease, Xiangyang Central Hospital, Affiliated Hospital of Hubei University of Arts and Science, Xiangyang, China.
Jiang C; Institute of Maternity Disease, Xiangyang Central Hospital, Affiliated Hospital of Hubei University of Arts and Science, Xiangyang, China.; Department of Pathology, Xiangyang Central Hospital, Affiliated Hospital of Hubei University of Arts and Science, Xiangyang, Hubei, China.
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Źródło:
Cancer medicine [Cancer Med] 2024 Mar; Vol. 13 (5), pp. e6958.
Typ publikacji:
Journal Article
MeSH Terms:
Chemokine CXCL10*
Endometrial Neoplasms*/genetics
Animals ; Mice ; Female ; Humans ; Phosphatidylinositol 3-Kinases ; Proto-Oncogene Proteins c-akt ; Mice, Nude ; CX3C Chemokine Receptor 1 ; Microtubule-Associated Proteins/genetics ; Cell Cycle Proteins/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Real-time ex vivo monitoring of NK cell migration toward obesity-associated oesophageal adenocarcinoma following modulation of CX3CR1.
Autorzy:
Mylod E; Cancer Immunology Research Group, Department of Anatomy, School of Medicine, Trinity Biomedical Sciences Institute and Trinity St. James's Cancer Institute, Trinity College Dublin, Dublin, Ireland.; Cancer Immunology and Immunotherapy Group, Department of Surgery, School of Medicine, Trinity Translational Medicine Institute and Trinity St. James's Cancer Institute, St. James's Hospital, Trinity College Dublin, Dublin, Ireland.
O'Connell F; Department of Surgery, School of Medicine, Trinity Translational Medicine Institute and Trinity St. James's Cancer Institute, Trinity College Dublin, Dublin, Ireland.
Donlon NE; Cancer Immunology and Immunotherapy Group, Department of Surgery, School of Medicine, Trinity Translational Medicine Institute and Trinity St. James's Cancer Institute, St. James's Hospital, Trinity College Dublin, Dublin, Ireland.
Davern M; Cancer Immunology and Immunotherapy Group, Department of Surgery, School of Medicine, Trinity Translational Medicine Institute and Trinity St. James's Cancer Institute, St. James's Hospital, Trinity College Dublin, Dublin, Ireland.
Marion C; Cancer Immunology Research Group, Department of Anatomy, School of Medicine, Trinity Biomedical Sciences Institute and Trinity St. James's Cancer Institute, Trinity College Dublin, Dublin, Ireland.; Cancer Immunology and Immunotherapy Group, Department of Surgery, School of Medicine, Trinity Translational Medicine Institute and Trinity St. James's Cancer Institute, St. James's Hospital, Trinity College Dublin, Dublin, Ireland.
Butler C; Department of Surgery, School of Medicine, Trinity Translational Medicine Institute and Trinity St. James's Cancer Institute, Trinity College Dublin, Dublin, Ireland.
Reynolds JV; Department of Surgery, School of Medicine, Trinity Translational Medicine Institute and Trinity St. James's Cancer Institute, Trinity College Dublin, Dublin, Ireland.
Lysaght J; Cancer Immunology and Immunotherapy Group, Department of Surgery, School of Medicine, Trinity Translational Medicine Institute and Trinity St. James's Cancer Institute, St. James's Hospital, Trinity College Dublin, Dublin, Ireland.
Conroy MJ; Cancer Immunology Research Group, Department of Anatomy, School of Medicine, Trinity Biomedical Sciences Institute and Trinity St. James's Cancer Institute, Trinity College Dublin, Dublin, Ireland. .
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Źródło:
Scientific reports [Sci Rep] 2024 Feb 18; Vol. 14 (1), pp. 4017. Date of Electronic Publication: 2024 Feb 18.
Typ publikacji:
Journal Article
MeSH Terms:
Adenocarcinoma*/therapy
Esophageal Neoplasms*/therapy
Humans ; Cell Movement ; CX3C Chemokine Receptor 1 ; Killer Cells, Natural ; Obesity/complications ; Tumor Microenvironment ; Immunotherapy
SCR Disease Name:
Adenocarcinoma Of Esophagus
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Diphtheria toxin induced but not CSF1R inhibitor mediated microglia ablation model leads to the loss of CSF/ventricular spaces in vivo that is independent of cytokine upregulation.
Autorzy:
Bedolla A; Department of Molecular Genetics and Biochemistry, College of Medicine, University of Cincinnati, Cincinnati, OH, USA.
Taranov A; Department of Molecular Genetics and Biochemistry, College of Medicine, University of Cincinnati, Cincinnati, OH, USA.
Luo F; Department of Molecular Genetics and Biochemistry, College of Medicine, University of Cincinnati, Cincinnati, OH, USA.
Wang J; Department of Molecular Genetics and Biochemistry, College of Medicine, University of Cincinnati, Cincinnati, OH, USA.
Turcato F; Department of Molecular Genetics and Biochemistry, College of Medicine, University of Cincinnati, Cincinnati, OH, USA.
Fugate EM; Imaging Research Center, Cincinnati Children's Hospital Medical Center, Department of Radiology, University of Cincinnati, Cincinnati, USA.
Greig NH; Drug Design & Development Section, Translational Gerontology Branch, Intramural Research Program, National Institute On Aging, National Institutes of Health, Baltimore, USA.
Lindquist DM; Imaging Research Center, Cincinnati Children's Hospital Medical Center, Department of Radiology, University of Cincinnati, Cincinnati, USA.
Crone SA; Division of Neurosurgery, Cincinnati Children's Hospital Medical Center, 3333 Burnet Avenue, Cincinnati, OH, 45229, USA.; Division of Developmental Biology, Cincinnati Children's Hospital Medical Center, 3333 Burnet Avenue, Cincinnati, OH, 45229, USA.; Department of Neurosurgery, College of Medicine, University of Cincinnati, 231 Albert Sabin Way, Cincinnati, OH, 45267, USA.
Goto J; Division of Neurosurgery, Cincinnati Children's Hospital Medical Center, 3333 Burnet Avenue, Cincinnati, OH, 45229, USA.; Department of Neurosurgery, College of Medicine, University of Cincinnati, 231 Albert Sabin Way, Cincinnati, OH, 45267, USA.
Luo Y; Department of Molecular Genetics and Biochemistry, College of Medicine, University of Cincinnati, Cincinnati, OH, USA. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2022 Jan 04; Vol. 19 (1), pp. 3. Date of Electronic Publication: 2022 Jan 04.
Typ publikacji:
Journal Article
MeSH Terms:
Brain/*drug effects
CX3C Chemokine Receptor 1/*metabolism
Cytokines/*metabolism
Diphtheria Toxin/*metabolism
Microglia/*drug effects
Animals ; Brain/metabolism ; CX3C Chemokine Receptor 1/genetics ; Female ; Male ; Mice ; Mice, Transgenic ; Microglia/metabolism ; Up-Regulation/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Early activation of the cardiac CX3CL1/CX3CR1 axis delays β-adrenergic-induced heart failure.
Autorzy:
Flamant M; Sorbonne Université, UPMC Univ Paris 06, INSERM, Institute of Cardiometabolism and Nutrition (ICAN), Team 3, UMR_S ICAN 1166 Team 3, 91 bd de l'hôpital, 75013, Paris, France.
Mougenot N; Sorbonne Université, UMS28, Plateforme d'Expérimentation Cœur, Muscles, Vaisseaux (PECMV), 75013, Paris, France.
Balse E; Sorbonne Université, UPMC Univ Paris 06, INSERM, Institute of Cardiometabolism and Nutrition (ICAN), Team 3, UMR_S ICAN 1166 Team 3, 91 bd de l'hôpital, 75013, Paris, France.
Le Fèvre L; Sorbonne Université, UPMC Univ Paris 06, INSERM, Institute of Cardiometabolism and Nutrition (ICAN), Team 3, UMR_S ICAN 1166 Team 3, 91 bd de l'hôpital, 75013, Paris, France.; Medical and Infectious Intensive Care Unit, Bichat hospital, APHP, 46 rue Henri Huchard, 75018, Paris, France.
Atassi F; Sorbonne Université, UPMC Univ Paris 06, INSERM, Institute of Cardiometabolism and Nutrition (ICAN), Team 3, UMR_S ICAN 1166 Team 3, 91 bd de l'hôpital, 75013, Paris, France.
Gautier EL; Sorbonne Université, UPMC Univ Paris 06, INSERM, Institute of Cardiometabolism and Nutrition (ICAN), UMR_S ICAN 1166 Team 5, 75013, Paris, France.
Le Goff W; Sorbonne Université, UPMC Univ Paris 06, INSERM, Institute of Cardiometabolism and Nutrition (ICAN), UMR_S ICAN 1166 Team 4, 75013, Paris, France.
Keck M; Sorbonne Université, UPMC Univ Paris 06, INSERM, Institute of Cardiometabolism and Nutrition (ICAN), Team 3, UMR_S ICAN 1166 Team 3, 91 bd de l'hôpital, 75013, Paris, France.; Département Médicaments et Technologies pour la Santé (DMTS), Université Paris-Saclay, CEA, INRAE, SIMoS, 91191, Gif-sur-Yvette, France.
Nadaud S; Sorbonne Université, UPMC Univ Paris 06, INSERM, Institute of Cardiometabolism and Nutrition (ICAN), Team 3, UMR_S ICAN 1166 Team 3, 91 bd de l'hôpital, 75013, Paris, France.
Combadière C; Sorbonne Université, Inserm, CNRS, Centre d'Immunologie et des Maladies Infectieuses CIMI-Paris, 75013, Paris, France.
Boissonnas A; Sorbonne Université, Inserm, CNRS, Centre d'Immunologie et des Maladies Infectieuses CIMI-Paris, 75013, Paris, France.
Pavoine C; Sorbonne Université, UPMC Univ Paris 06, INSERM, Institute of Cardiometabolism and Nutrition (ICAN), Team 3, UMR_S ICAN 1166 Team 3, 91 bd de l'hôpital, 75013, Paris, France. .
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Źródło:
Scientific reports [Sci Rep] 2021 Sep 09; Vol. 11 (1), pp. 17982. Date of Electronic Publication: 2021 Sep 09.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adrenergic beta-Agonists/*adverse effects
CX3C Chemokine Receptor 1/*metabolism
Chemokine CX3CL1/*metabolism
Heart Failure/*chemically induced
Heart Failure/*metabolism
Isoproterenol/*adverse effects
Macrophages/*metabolism
Myocytes, Cardiac/*metabolism
Signal Transduction/*genetics
Animals ; CX3C Chemokine Receptor 1/genetics ; Cell Communication/genetics ; Cell Proliferation/genetics ; Cells, Cultured ; Chemokine CX3CL1/genetics ; Disease Models, Animal ; Heart Failure/genetics ; Hypertrophy ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Myocytes, Cardiac/pathology ; RNA, Small Interfering/administration & dosage ; RNA, Small Interfering/genetics ; Tumor Necrosis Factor-alpha/metabolism ; Ventricular Remodeling/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Native Low-Density Lipoproteins Act in Synergy with Lipopolysaccharide to Alter the Balance of Human Monocyte Subsets and Their Ability to Produce IL-1 Beta, CCR2, and CX3CR1 In Vitro and In Vivo: Implications in Atherogenesis.
Autorzy:
Manjarrez-Reyna AN; Laboratory of Immunometabolism, Research Division, General Hospital of Mexico 'Dr. Eduardo Liceaga', Mexico City 06720, Mexico.
Martínez-Reyes CP; Laboratory of Immunometabolism, Research Division, General Hospital of Mexico 'Dr. Eduardo Liceaga', Mexico City 06720, Mexico.
Aguayo-Guerrero JA; Laboratory of Immunometabolism, Research Division, General Hospital of Mexico 'Dr. Eduardo Liceaga', Mexico City 06720, Mexico.
Méndez-García LA; Laboratory of Immunometabolism, Research Division, General Hospital of Mexico 'Dr. Eduardo Liceaga', Mexico City 06720, Mexico.
Esquivel-Velázquez M; Laboratory of Immunometabolism, Research Division, General Hospital of Mexico 'Dr. Eduardo Liceaga', Mexico City 06720, Mexico.
León-Cabrera S; Unidad de Biomedicina, Facultad de Estudios Superiores-Iztacala, Universidad Nacional Autónoma de México, Tlalnepantla, Edo. De Mexico 54090, Mexico.; Carrera de Médico Cirujano, Facultad de Estudios Superiores-Iztacala, Universidad Nacional Autónoma de México, Tlalnepantla, Edo. De Mexico 54090, Mexico.
Vargas-Alarcón G; Department of Molecular Biology, Instituto Nacional de Cardiología 'Ignacio Chávez', Juan Badiano 1, Sección XVI, Tlalpan, Mexico City 14080, Mexico.
Fragoso JM; Department of Molecular Biology, Instituto Nacional de Cardiología 'Ignacio Chávez', Juan Badiano 1, Sección XVI, Tlalpan, Mexico City 14080, Mexico.
Carreón-Torres E; Department of Molecular Biology, Instituto Nacional de Cardiología 'Ignacio Chávez', Juan Badiano 1, Sección XVI, Tlalpan, Mexico City 14080, Mexico.
Pérez-Méndez O; Department of Molecular Biology, Instituto Nacional de Cardiología 'Ignacio Chávez', Juan Badiano 1, Sección XVI, Tlalpan, Mexico City 14080, Mexico.; School of Engineering and Sciences Campus CDMX, Tecnológico de Monterrey, Calle Puente 222, Tlalpan, Mexico City 14380, Mexico.
Prieto-Chávez JL; Laboratorio de Citometría de Flujo, Centro de Instrumentos, Coordinación de Investigación en Salud, Hospital de Especialidades del Centro Médico Siglo XXI, Instituto Mexicano del Seguro Social, Mexico City 06720, Mexico.
Escobedo G; Laboratory of Immunometabolism, Research Division, General Hospital of Mexico 'Dr. Eduardo Liceaga', Mexico City 06720, Mexico.
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Źródło:
Biomolecules [Biomolecules] 2021 Aug 07; Vol. 11 (8). Date of Electronic Publication: 2021 Aug 07.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
CX3C Chemokine Receptor 1/*genetics
Cholesterol, LDL/*pharmacology
Interleukin-1beta/*genetics
Lipopolysaccharides/*pharmacology
Monocytes/*drug effects
Receptors, CCR2/*genetics
Acute-Phase Proteins/genetics ; Acute-Phase Proteins/immunology ; Adolescent ; Adult ; C-Reactive Protein/genetics ; C-Reactive Protein/immunology ; CX3C Chemokine Receptor 1/immunology ; Carrier Proteins/genetics ; Carrier Proteins/immunology ; Cell Lineage/drug effects ; Cell Lineage/immunology ; Cholesterol, HDL/blood ; Drug Synergism ; Female ; Flow Cytometry ; Gene Expression ; Healthy Volunteers ; Humans ; Interleukin-1beta/immunology ; Male ; Membrane Glycoproteins/genetics ; Membrane Glycoproteins/immunology ; Middle Aged ; Monocytes/cytology ; Monocytes/immunology ; Primary Cell Culture ; Receptors, CCR2/immunology ; Triglycerides/blood
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
OncoTherad (MRB-CFI-1) Nanoimmunotherapy: A Promising Strategy to Treat Bacillus Calmette-Guérin-Unresponsive Non-Muscle-Invasive Bladder Cancer: Crosstalk among T-Cell CX3CR1, Immune Checkpoints, and the Toll-Like Receptor 4 Signaling Pathway.
Autorzy:
Alonso JCC; Laboratory of Urogenital Carcinogenesis and Immunotherapy (LCURGIN), Universidade Estadual de Campinas (UNICAMP), Campinas 13083-865, São Paulo, Brazil.; Paulínia Municipal Hospital, Paulínia 13140-000, São Paulo, Brazil.
de Souza BR; Obstetrics & Gynecology Department, Ovarian Cancer Research Group University of British Columbia, Vancouver, BC V6Z 2K8, Canada.
Reis IB; Diagnosis and Surgery Department, Dentistry School, São Paulo State University (UNESP), Araraquara 14801-903, São Paulo, Brazil.
de Arruda Camargo GC; Laboratory of Urogenital Carcinogenesis and Immunotherapy (LCURGIN), Universidade Estadual de Campinas (UNICAMP), Campinas 13083-865, São Paulo, Brazil.
de Oliveira G; Laboratory of Urogenital Carcinogenesis and Immunotherapy (LCURGIN), Universidade Estadual de Campinas (UNICAMP), Campinas 13083-865, São Paulo, Brazil.
de Barros Frazão Salmazo MI; Laboratory of Urogenital Carcinogenesis and Immunotherapy (LCURGIN), Universidade Estadual de Campinas (UNICAMP), Campinas 13083-865, São Paulo, Brazil.
Gonçalves JM; Laboratory of Urogenital Carcinogenesis and Immunotherapy (LCURGIN), Universidade Estadual de Campinas (UNICAMP), Campinas 13083-865, São Paulo, Brazil.
de Castro Roston JR; Laboratory of Urogenital Carcinogenesis and Immunotherapy (LCURGIN), Universidade Estadual de Campinas (UNICAMP), Campinas 13083-865, São Paulo, Brazil.
Caria PHF; Laboratory of Urogenital Carcinogenesis and Immunotherapy (LCURGIN), Universidade Estadual de Campinas (UNICAMP), Campinas 13083-865, São Paulo, Brazil.
da Silva Santos A; Laboratory of Urogenital Carcinogenesis and Immunotherapy (LCURGIN), Universidade Estadual de Campinas (UNICAMP), Campinas 13083-865, São Paulo, Brazil.
de Freitas LLL; Pathology Department, Medical School, Universidade Estadual de Campinas (UNICAMP), Campinas 13083-888, São Paulo, Brazil.
Billis A; Pathology Department, Medical School, Universidade Estadual de Campinas (UNICAMP), Campinas 13083-888, São Paulo, Brazil.
Durán N; Laboratory of Urogenital Carcinogenesis and Immunotherapy (LCURGIN), Universidade Estadual de Campinas (UNICAMP), Campinas 13083-865, São Paulo, Brazil.
Fávaro WJ; Laboratory of Urogenital Carcinogenesis and Immunotherapy (LCURGIN), Universidade Estadual de Campinas (UNICAMP), Campinas 13083-865, São Paulo, Brazil.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Dec 15; Vol. 24 (24). Date of Electronic Publication: 2023 Dec 15.
Typ publikacji:
Clinical Trial, Phase II; Clinical Trial, Phase I; Journal Article
MeSH Terms:
Non-Muscle Invasive Bladder Neoplasms*/therapy
Urinary Bladder Neoplasms*/drug therapy
Urinary Bladder Neoplasms*/pathology
Immunotherapy*/methods
Aged ; Humans ; Adjuvants, Immunologic/therapeutic use ; Administration, Intravesical ; BCG Vaccine/therapeutic use ; CX3C Chemokine Receptor 1 ; Neoplasm Invasiveness ; Neoplasm Recurrence, Local/drug therapy ; Signal Transduction ; Toll-Like Receptor 4/therapeutic use ; Tumor Microenvironment ; Nanoparticle Drug Delivery System
Czasopismo naukowe
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Tytuł:
不可复性牙髓炎患者血清可溶性B7-H3、 CX3趋化因子受体1水平变化及其临床意义
Autorzy:
袁媛园
潘露
周绍兰
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Alternatywny tytuł:
Changes in serum sB7-H3 and CX3CR1 levels in patients with irreversible pulpitis and their clinical significance.
Źródło:
Journal of Practical Medicine / Shiyong Yixue Zazhi. 9/10/2023, Vol. 39 Issue 17, p2221-2224. 4p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Respiratory Syncytial Virus (RSV) G Protein Vaccines With Central Conserved Domain Mutations Induce CX3C-CX3CR1 Blocking Antibodies.
Autorzy:
Bergeron HC; Department of Infectious Diseases, University of Georgia, Athens, GA 30605, USA.
Murray J; Department of Infectious Diseases, University of Georgia, Athens, GA 30605, USA.
Nuñez Castrejon AM; Department of Biomolecular Engineering, University of California-Santa Cruz, Santa Cruz, CA 95064, USA.
DuBois RM; Department of Biomolecular Engineering, University of California-Santa Cruz, Santa Cruz, CA 95064, USA.
Tripp RA; Department of Infectious Diseases, University of Georgia, Athens, GA 30605, USA.
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Źródło:
Viruses [Viruses] 2021 Feb 23; Vol. 13 (2). Date of Electronic Publication: 2021 Feb 23.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
CX3C Chemokine Receptor 1/*immunology
Chemokines, CX3C/*immunology
Respiratory Syncytial Virus Infections/*prevention & control
Respiratory Syncytial Virus Vaccines/*immunology
Respiratory Syncytial Virus, Human/*immunology
Viral Envelope Proteins/*genetics
Viral Envelope Proteins/*immunology
Animals ; Antibodies, Blocking/immunology ; Antibodies, Viral/immunology ; CX3C Chemokine Receptor 1/genetics ; Chemokines, CX3C/genetics ; Female ; Humans ; Mice ; Mice, Inbred BALB C ; Mutation ; Protein Domains ; Respiratory Syncytial Virus Infections/immunology ; Respiratory Syncytial Virus Infections/virology ; Respiratory Syncytial Virus Vaccines/administration & dosage ; Respiratory Syncytial Virus Vaccines/chemistry ; Respiratory Syncytial Virus Vaccines/genetics ; Respiratory Syncytial Virus, Human/chemistry ; Respiratory Syncytial Virus, Human/genetics ; Viral Envelope Proteins/chemistry
Czasopismo naukowe
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Tytuł:
Absence in CX3CR1 receptor signaling promotes post‐ischemic stroke cognitive function recovery through suppressed microglial pyroptosis in mice.
Autorzy:
Ge, Yangyang (AUTHOR)
Yang, Juexi (AUTHOR)
Chen, Jiayi (AUTHOR)
Dai, Maosha (AUTHOR)
Dou, Xiaoke (AUTHOR)
Yao, Shanglong (AUTHOR)
Yao, Chenye (AUTHOR)
Lin, Yun (AUTHOR)
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Źródło:
CNS Neuroscience & Therapeutics. Feb2024, Vol. 30 Issue 2, p1-14. 14p.
Czasopismo naukowe
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Tytuł:
CX3CR1 Deficiency Attenuates DNFB-Induced Contact Hypersensitivity Through Skewed Polarization Towards M2 Phenotype in Macrophages.
Autorzy:
Otobe S; Department of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.
Hisamoto T; Department of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.
Miyagaki T; Department of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.; Department of Dermatology, St. Marianna University School of Medicine, Kanagawa 216-8511, Japan.
Morimura S; Department of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.; Department of Dermatology, International University of Health and Welfare, Chiba 286-0124, Japan.
Suga H; Department of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.
Sugaya M; Department of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.; Department of Dermatology, International University of Health and Welfare, Chiba 286-0124, Japan.
Sato S; Department of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Oct 07; Vol. 21 (19). Date of Electronic Publication: 2020 Oct 07.
Typ publikacji:
Journal Article
MeSH Terms:
CX3C Chemokine Receptor 1/*deficiency
Dermatitis, Contact/*etiology
Dermatitis, Contact/*metabolism
Dinitrofluorobenzene/*pharmacology
Macrophage Activation/*drug effects
Macrophage Activation/*immunology
Macrophages/*drug effects
Macrophages/*physiology
Animals ; Biomarkers ; CX3C Chemokine Receptor 1/metabolism ; Dermatitis, Contact/pathology ; Disease Models, Animal ; Disease Susceptibility ; Immunohistochemistry ; Mice ; Mice, Knockout ; Neutrophil Infiltration/immunology
Czasopismo naukowe
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Tytuł:
Comparative Analysis of the Occurrence and Role of CX3CL1 (Fractalkine) and Its Receptor CX3CR1 in Hemophilic Arthropathy and Osteoarthritis.
Autorzy:
Wojdasiewicz P; Department of General and Experimental Pathology, Centre for Preclinical Research and Technology (CePT), Medical University of Warsaw, Pawińskiego 3C, 02-106 Warsaw, Poland.; Department of Rehabilitation, Eleonora Reicher National Institute of Geriatrics, Rheumatology and Rehabilitation, Spartańska 1, 02-637 Warsaw, Poland.
Poniatowski ŁA; Department of Experimental and Clinical Pharmacology, Centre for Preclinical Research and Technology (CePT), Medical University of Warsaw, Banacha 1B, 02-097 Warsaw, Poland.; Department of Neurosurgery, Maria Skłodowska-Curie Memorial Cancer Center and Institute of Oncology, W. K. Roentgena 5, 02-781 Warsaw, Poland.
Kotela A; Department of Orthopedics and Traumatology, 1st Faculty of Medicine, Medical University of Warsaw, Lindleya 4, 02-005 Warsaw, Poland.; Department of Orthopedics and Traumatology, Central Clinical Hospital of the Ministry of the Interior and Administration, Wołoska 137, 02-507 Warsaw, Poland.
Skoda M; Department of General and Experimental Pathology, Centre for Preclinical Research and Technology (CePT), Medical University of Warsaw, Pawińskiego 3C, 02-106 Warsaw, Poland.
Pyzlak M; Department of General and Experimental Pathology, Centre for Preclinical Research and Technology (CePT), Medical University of Warsaw, Pawińskiego 3C, 02-106 Warsaw, Poland.
Stangret A; Department of General and Experimental Pathology, Centre for Preclinical Research and Technology (CePT), Medical University of Warsaw, Pawińskiego 3C, 02-106 Warsaw, Poland.
Kotela I; Department of Orthopedics and Traumatology, Central Clinical Hospital of the Ministry of the Interior and Administration, Wołoska 137, 02-507 Warsaw, Poland.; Department of Rehabilitation in Disease of the Locomotor System, Faculty of Medicine and Health Sciences, Jan Kochanowski University, Kielce, Poland.
Szukiewicz D; Department of General and Experimental Pathology, Centre for Preclinical Research and Technology (CePT), Medical University of Warsaw, Pawińskiego 3C, 02-106 Warsaw, Poland.
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Źródło:
Journal of immunology research [J Immunol Res] 2020 Aug 20; Vol. 2020, pp. 2932696. Date of Electronic Publication: 2020 Aug 20 (Print Publication: 2020).
Typ publikacji:
Journal Article
MeSH Terms:
Arthritis/*etiology
Arthritis/*metabolism
CX3C Chemokine Receptor 1/*metabolism
Chemokine CX3CL1/*metabolism
Hemophilia A/*complications
Osteoarthritis/*etiology
Osteoarthritis/*metabolism
Antigens, CD/metabolism ; Antigens, Differentiation, Myelomonocytic/metabolism ; Arthritis/diagnosis ; Biomarkers ; Blood Vessels/metabolism ; Blood Vessels/pathology ; CD56 Antigen/metabolism ; CX3C Chemokine Receptor 1/genetics ; Cartilage, Articular/metabolism ; Cartilage, Articular/pathology ; Case-Control Studies ; Chemokine CX3CL1/genetics ; Disease Susceptibility ; Fibrosis ; Gene Expression ; Humans ; Immunohistochemistry ; Osteoarthritis/diagnosis ; Synovial Fluid/metabolism ; Synovial Membrane/metabolism ; Synovial Membrane/pathology
Czasopismo naukowe
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Tytuł:
MiR-1207-5p/CX3CR1 axis regulates the progression of osteoarthritis via the modulation of the activity of NF-κB pathway.
Autorzy:
Liu XC; Department of Joint Surgery, Shandong Provincial Hospital, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, China.; Department of Orthopedics, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan, Shandong, China.
Xu L; Department of Orthopedic Surgery, Affiliated Hospital of Shandong Academy of Medical Sciences, Jinan, Shandong, China.; Department of Orthopedics, Shandong Chest Hospital, Jinan, Shandong, China.
Cai YL; Department of Orthopedics, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan, Shandong, China.
Zheng ZY; Department of Orthopedics, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan, Shandong, China.
Dai EN; Department of Orthopedic Surgery, Affiliated Hospital of Shandong Academy of Medical Sciences, Jinan, Shandong, China.
Sun S; Department of Joint Surgery, Shandong Provincial Hospital, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, China.
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Źródło:
International journal of rheumatic diseases [Int J Rheum Dis] 2020 Aug; Vol. 23 (8), pp. 1057-1065. Date of Electronic Publication: 2020 Jun 29.
Typ publikacji:
Journal Article
MeSH Terms:
CX3C Chemokine Receptor 1/*metabolism
Chondrocytes/*metabolism
MicroRNAs/*metabolism
NF-kappa B/*metabolism
Osteoarthritis/*metabolism
Apoptosis ; CX3C Chemokine Receptor 1/genetics ; Cell Line ; Cell Proliferation ; Chondrocytes/drug effects ; Chondrocytes/immunology ; Chondrocytes/pathology ; Databases, Genetic ; Disease Progression ; Extracellular Matrix/metabolism ; Extracellular Matrix/pathology ; Gene Expression Regulation ; Humans ; Lipopolysaccharides/pharmacology ; MicroRNAs/genetics ; Osteoarthritis/genetics ; Osteoarthritis/immunology ; Osteoarthritis/pathology ; Phosphorylation ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Exploring the role of miR-200 family in regulating CX3CR1 and CXCR1 in lung adenocarcinoma tumor microenvironment: implications for therapeutic intervention.
Autorzy:
Sharma A; Translational Research Lab, Department of Biotechnology, Faculty of Natural Sciences, Jamia Millia Islamia, New Delhi, 110025, India.
Singh P; Centre for Interdisciplinary Research in Basic Sciences, Jamia Millia Islamia, New Delhi, 110025, India.
Jha R; Centre for Interdisciplinary Research in Basic Sciences, Jamia Millia Islamia, New Delhi, 110025, India.
Almatroodi SA; Department of Medical Laboratories, College of Applied Medical Sciences, Qassim University, 51452, Buraydah, Saudi Arabia.
Alrumaihi F; Department of Medical Laboratories, College of Applied Medical Sciences, Qassim University, 51452, Buraydah, Saudi Arabia.
Rahmani AH; Department of Medical Laboratories, College of Applied Medical Sciences, Qassim University, 51452, Buraydah, Saudi Arabia.
Alharbi HO; Department of Medical Laboratories, College of Applied Medical Sciences, Qassim University, 51452, Buraydah, Saudi Arabia.
Dohare R; Centre for Interdisciplinary Research in Basic Sciences, Jamia Millia Islamia, New Delhi, 110025, India. .
Syed MA; Translational Research Lab, Department of Biotechnology, Faculty of Natural Sciences, Jamia Millia Islamia, New Delhi, 110025, India. .
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Źródło:
Scientific reports [Sci Rep] 2023 Sep 28; Vol. 13 (1), pp. 16333. Date of Electronic Publication: 2023 Sep 28.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Lung Neoplasms*/genetics
Lung Neoplasms*/therapy
Lung Neoplasms*/metabolism
Adenocarcinoma of Lung*/pathology
MicroRNAs*/genetics
MicroRNAs*/metabolism
Adenocarcinoma*/genetics
Humans ; Tumor Microenvironment/genetics ; CX3C Chemokine Receptor 1/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Fractalkine/CX 3 CR1 in Dilated Cardiomyopathy: A Potential Future Target for Immunomodulatory Therapy?
Autorzy:
Jeyalan V; Academic Cardiovascular Unit, The James Cook University Hospital, Middlesbrough TS4 3BW, UK.; Translational and Clinical Research Institute, Newcastle University, Newcastle upon Tyne NE2 4HH, UK.
Austin D; Academic Cardiovascular Unit, The James Cook University Hospital, Middlesbrough TS4 3BW, UK.; Population Health Science Institute, Newcastle University, Newcastle upon Tyne NE1 7RU, UK.
Loh SX; Department of Cardiology, Freeman Hospital, Newcastle upon Tyne NHS Foundation Trust, Newcastle upon Tyne NE7 7DN, UK.
Wangsaputra VK; Translational and Clinical Research Institute, Newcastle University, Newcastle upon Tyne NE2 4HH, UK.; Faculty of Medicine, Universitas Indonesia, Central Jakarta 10430, Indonesia.
Spyridopoulos I; Translational and Clinical Research Institute, Newcastle University, Newcastle upon Tyne NE2 4HH, UK.; Department of Cardiology, Freeman Hospital, Newcastle upon Tyne NHS Foundation Trust, Newcastle upon Tyne NE7 7DN, UK.
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Źródło:
Cells [Cells] 2023 Sep 28; Vol. 12 (19). Date of Electronic Publication: 2023 Sep 28.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
Cardiomyopathy, Dilated*/pathology
Humans ; CX3C Chemokine Receptor 1 ; Chemokine CX3CL1 ; Inflammation ; Immunomodulation ; Receptors, Complement 3b
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Wyświetlanie 1-20 z 1285

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