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Tytuł:
Stellera chamaejasme L. extract inhibits adipocyte differentiation through activation of the extracellular signal-regulated kinase pathway.
Autorzy:
Yeon J; Department of Biosciences, Mokpo National University, Jeonnam, Republic of Korea.
Kim E; Department of Biomedicine, Health & Life Convergence Sciences, BK21 Four, Biomedical and Healthcare Research Institute, Mokpo National University, Jeonnam, Republic of Korea.
Bazarragchaa B; Natural History Museum of Mongolia, Ulaanbaatar, Mongolia.
Kim SY; International Biological Material Research Center, Korea Research Institute of Bioscience and Biotechnology, Daejeon, Republic of Korea.
Huh JY; Department of Life Science, Sogang University, Seoul, Republic of Korea.
Park H; Department of Obstetrics & Gynecology, Korea University College of Medicine, Seoul, Republic of Korea.
Suh SS; Department of Biosciences, Mokpo National University, Jeonnam, Republic of Korea.; Department of Biomedicine, Health & Life Convergence Sciences, BK21 Four, Biomedical and Healthcare Research Institute, Mokpo National University, Jeonnam, Republic of Korea.
Seo JB; Department of Biosciences, Mokpo National University, Jeonnam, Republic of Korea.; Department of Biomedicine, Health & Life Convergence Sciences, BK21 Four, Biomedical and Healthcare Research Institute, Mokpo National University, Jeonnam, Republic of Korea.
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Źródło:
PloS one [PLoS One] 2024 Mar 21; Vol. 19 (3), pp. e0300520. Date of Electronic Publication: 2024 Mar 21 (Print Publication: 2024).
Typ publikacji:
Journal Article
MeSH Terms:
Extracellular Signal-Regulated MAP Kinases*/metabolism
Adipogenesis*
Mice ; Animals ; Cell Differentiation ; Lipid Metabolism ; Adipocytes/metabolism ; 3T3-L1 Cells ; PPAR gamma/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
mGluR1/IP3/ERK signaling pathway regulates vestibular compensation in ON UBCs of the cerebellar flocculus.
Autorzy:
Liu D; Department of Otorhinolaryngology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Institute of Otorhinolaryngology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Wang J; Department of Otorhinolaryngology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Institute of Otorhinolaryngology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Tian E; Department of Otorhinolaryngology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Institute of Otorhinolaryngology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Chen J; Department of Otorhinolaryngology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Institute of Otorhinolaryngology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Kong W; Department of Otorhinolaryngology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Institute of Otorhinolaryngology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Lu Y; Department of Physiology, School of Basic Medicine, Huazhong University of Science and Technology, Wuhan, China.; Institute of Brain Research, Collaborative Innovation Center for Brain Science, Huazhong University of Science and Technology, Wuhan, China.
Zhang S; Department of Otorhinolaryngology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Institute of Otorhinolaryngology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Feb; Vol. 30 (2), pp. e14419. Date of Electronic Publication: 2023 Aug 25.
Typ publikacji:
Journal Article
MeSH Terms:
Extracellular Signal-Regulated MAP Kinases*
Signal Transduction*
Benzoates*
Receptors, Metabotropic Glutamate*
Glycine/*analogs & derivatives
Methoxyhydroxyphenylglycol/*analogs & derivatives
Rats ; Animals
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Novel predictive model of cell survival/death related effects of Extracellular Signal-Regulated kinase protein.
Autorzy:
Jain S; Department of Electronics and Communications Engineering, Jaypee University of Information Technology, Solan, Himachal Pradesh, India.
Salau AO; Department of Electrical/Electronics and Computer Engineering, Afe Babalola University, Ado-Ekiti, Nigeria.; Saveetha School of Engineering, Saveetha Institute of Medical and Technical Sciences, Tamil Nadu, India.
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Źródło:
Artificial cells, nanomedicine, and biotechnology [Artif Cells Nanomed Biotechnol] 2023 Dec; Vol. 51 (1), pp. 158-169.
Typ publikacji:
Journal Article
MeSH Terms:
Extracellular Signal-Regulated MAP Kinases*/metabolism
Insulins*
Epidermal Growth Factor/pharmacology ; Cell Survival
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Extracellular Signal-Regulated Kinases Play Essential but Contrasting Roles in Osteoclast Differentiation.
Autorzy:
Kim C; BK21 Program in Biomedical Science & Engineering, Laboratory for Leukocyte Signaling Research, Department of Pharmacology, College of Medicine, Inha University, Incheon 22212, Republic of Korea.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Oct 19; Vol. 24 (20). Date of Electronic Publication: 2023 Oct 19.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Osteoclasts*/metabolism
Extracellular Signal-Regulated MAP Kinases*/metabolism
Osteoblasts/metabolism ; Cell Differentiation ; Cytokines/metabolism ; Macrophage Colony-Stimulating Factor/pharmacology ; Macrophage Colony-Stimulating Factor/metabolism ; RANK Ligand/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Computational Investigations on Reaction Mechanisms of the Covalent Inhibitors Ponatinib and Analogs Targeting the Extracellular Signal-Regulated Kinases.
Autorzy:
Tian Y; College of Chemistry and Molecular Sciences, Wuhan University, Wuhan 430072, China.
Zhang M; College of Chemistry and Molecular Sciences, Wuhan University, Wuhan 430072, China.
Heng P; College of Chemistry and Molecular Sciences, Wuhan University, Wuhan 430072, China.
Hou H; College of Chemistry and Molecular Sciences, Wuhan University, Wuhan 430072, China.
Wang B; College of Chemistry and Molecular Sciences, Wuhan University, Wuhan 430072, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Oct 16; Vol. 24 (20). Date of Electronic Publication: 2023 Oct 16.
Typ publikacji:
Journal Article
MeSH Terms:
Extracellular Signal-Regulated MAP Kinases*
Neoplasms*
Humans ; Molecular Dynamics Simulation ; Imidazoles/pharmacology ; Thermodynamics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Modulation of the proteostasis network promotes tumor resistance to oncogenic KRAS inhibitors.
Autorzy:
Lv X; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Lu X; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Cao J; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Luo Q; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Ding Y; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Peng F; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Pataer A; Department of Thoracic and Cardiovascular Surgery, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
Lu D; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Department of Pharmacology and Chemical Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Center for Drug Discovery, Baylor College of Medicine, Houston, TX 77030, USA.
Han D; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Malmberg E; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Chan DW; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Wang X; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Savage SR; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX 77030, USA.
Mao S; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Yu J; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Peng F; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Department of Medicine, Division of Diabetes, Endocrinology and Metabolism, Baylor College of Medicine, Houston, TX 77030, USA.
Yan L; Department of Molecular and Cellular Oncology, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
Meng H; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.
Maneix L; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Huffington Center on Aging, Baylor College of Medicine, USA.
Han Y; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Chen Y; Department of Bioinformatics and Computational Biology, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
Yao W; Department of Translational Molecular Pathology, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
Chang EC; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Catic A; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Huffington Center on Aging, Baylor College of Medicine, USA.
Lin X; Division of Surgical Oncology, Michael E. DeBakey Department of Surgery, Baylor College of Medicine, Houston, TX 77030, USA.
Miles G; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX 77030, USA.
Huang P; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.
Sun Z; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Department of Medicine, Division of Diabetes, Endocrinology and Metabolism, Baylor College of Medicine, Houston, TX 77030, USA.
Burt B; Division of Thoracic Surgery, Michael E. DeBakey Department of Surgery, Baylor College of Medicine, Houston, TX 77030, USA.
Wang H; Department of Pathology, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
Wang J; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Department of Pharmacology and Chemical Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Center for Drug Discovery, Baylor College of Medicine, Houston, TX 77030, USA.
Yao QC; Division of Surgical Oncology, Michael E. DeBakey Department of Surgery, Baylor College of Medicine, Houston, TX 77030, USA.
Zhang B; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX 77030, USA.
Roth JA; Department of Thoracic and Cardiovascular Surgery, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
O'Malley BW; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.
Ellis MJ; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.; Early Oncology, Oncology R&D, AstraZeneca, Gaithersburg, MD, USA.
Rimawi MF; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
Ying H; Department of Molecular and Cellular Oncology, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
Chen X; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, TX 77030, USA.; Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA.; Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA.
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Źródło:
Science (New York, N.Y.) [Science] 2023 Sep 08; Vol. 381 (6662), pp. eabn4180. Date of Electronic Publication: 2023 Sep 08.
Typ publikacji:
Journal Article
MeSH Terms:
Endoribonucleases*
Extracellular Signal-Regulated MAP Kinases*
Neoplasms*/drug therapy
Neoplasms*/genetics
Proteostasis*
Proto-Oncogene Proteins p21(ras)*/antagonists & inhibitors
Proto-Oncogene Proteins p21(ras)*/genetics
Drug Resistance, Neoplasm*
Enzyme Inhibitors*/pharmacology
Antineoplastic Agents*/pharmacology
Heat Shock Transcription Factors*/metabolism
Humans ; Protein Serine-Threonine Kinases
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Peptide Lv Promotes Trafficking and Membrane Insertion of K Ca 3.1 through the MEK1-ERK and PI3K-Akt Signaling Pathways.
Autorzy:
Pham DL; Department of Veterinary Integrative Biosciences, School of Veterinary Medicine and Biomedical Sciences, Texas A&M University, College Station, TX 77843, USA.
Niemi A; Department of Veterinary Integrative Biosciences, School of Veterinary Medicine and Biomedical Sciences, Texas A&M University, College Station, TX 77843, USA.
Blank R; Department of Veterinary Integrative Biosciences, School of Veterinary Medicine and Biomedical Sciences, Texas A&M University, College Station, TX 77843, USA.
Lomenzo G; Department of Veterinary Integrative Biosciences, School of Veterinary Medicine and Biomedical Sciences, Texas A&M University, College Station, TX 77843, USA.
Tham J; Department of Veterinary Integrative Biosciences, School of Veterinary Medicine and Biomedical Sciences, Texas A&M University, College Station, TX 77843, USA.
Ko ML; Department of Veterinary Integrative Biosciences, School of Veterinary Medicine and Biomedical Sciences, Texas A&M University, College Station, TX 77843, USA.; Department of Biology, Division of Natural and Physical Sciences, Blinn College, Bryan, TX 77802, USA.
Ko GY; Department of Veterinary Integrative Biosciences, School of Veterinary Medicine and Biomedical Sciences, Texas A&M University, College Station, TX 77843, USA.; Texas A&M Institute for Neuroscience, Texas A&M University, College Station, TX 77843, USA.
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Źródło:
Cells [Cells] 2023 Jun 17; Vol. 12 (12). Date of Electronic Publication: 2023 Jun 17.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Endothelial Cells*/metabolism
Extracellular Signal-Regulated MAP Kinases*/metabolism
Proto-Oncogene Proteins c-akt*/metabolism
MAP Kinase Kinase 1/metabolism ; Peptides ; Phosphatidylinositol 3-Kinase/metabolism ; Phosphatidylinositol 3-Kinases/metabolism ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Generation of pulsatile ERK activity in mouse embryonic stem cells is regulated by Raf activity.
Autorzy:
Toyooka Y; Division of Embryology, National Institute for Basic Biology, National Institutes of Natural Sciences, 5-1 Higashiyama, Myodaiji-Cho, Okazaki, Aichi, 444-8787, Japan. .; Department of Clinical Application, Center for iPS Cell Research and Application (CiRA), Kyoto University, 53 Kawahara-Cho, Shogoin, Sakyo-Ku, Kyoto, 606-8507, Japan. .
Aoki K; Division of Quantitative Biology, National Institute for Basic Biology, Okazaki, Japan.; Quantitative Biology Research Group, Exploratory Research Center on Life and Living Systems (ExCELLS), Okazaki, Aichi, Japan.; Department of Basic Biology, School of Life Science, SOKENDAI (The Graduate University for Advanced Studies), Okazaki, Aichi, Japan.
Usami FM; Division of Embryology, National Institute for Basic Biology, National Institutes of Natural Sciences, 5-1 Higashiyama, Myodaiji-Cho, Okazaki, Aichi, 444-8787, Japan.; Department of Basic Biology, School of Life Science, SOKENDAI (The Graduate University for Advanced Studies), Okazaki, Aichi, Japan.
Oka S; Division of Embryology, National Institute for Basic Biology, National Institutes of Natural Sciences, 5-1 Higashiyama, Myodaiji-Cho, Okazaki, Aichi, 444-8787, Japan.
Kato A; Division of Embryology, National Institute for Basic Biology, National Institutes of Natural Sciences, 5-1 Higashiyama, Myodaiji-Cho, Okazaki, Aichi, 444-8787, Japan.
Fujimori T; Division of Embryology, National Institute for Basic Biology, National Institutes of Natural Sciences, 5-1 Higashiyama, Myodaiji-Cho, Okazaki, Aichi, 444-8787, Japan. .; Department of Basic Biology, School of Life Science, SOKENDAI (The Graduate University for Advanced Studies), Okazaki, Aichi, Japan. .
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Źródło:
Scientific reports [Sci Rep] 2023 Jun 10; Vol. 13 (1), pp. 9465. Date of Electronic Publication: 2023 Jun 10.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Mouse Embryonic Stem Cells*/metabolism
Extracellular Signal-Regulated MAP Kinases*/metabolism
Animals ; Mice ; MAP Kinase Signaling System ; Cell Differentiation ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
MAPK-ERK Pathway.
Autorzy:
Park JI; Department of Biochemistry, Medical College of Wisconsin, Milwaukee, WI 53226, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Jun 02; Vol. 24 (11). Date of Electronic Publication: 2023 Jun 02.
Typ publikacji:
Editorial
MeSH Terms:
Extracellular Signal-Regulated MAP Kinases*/metabolism
Signal Transduction*/physiology
Animals ; MAP Kinase Signaling System/physiology ; Phosphorylation ; Mammals/metabolism
Opinia redakcyjna
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Effects of 5G-modulated 3.5 GHz radiofrequency field exposures on HSF1, RAS, ERK, and PML activation in live fibroblasts and keratinocytes cells.
Autorzy:
Joushomme A; Bordeaux University, CNRS, IMS laboratory, UMR5218, F-33400, Talence, France.
Orlacchio R; Limoges University, CNRS, XLIM, UMR 7252, F-87000, Limoges, France.
Patrignoni L; Bordeaux University, CNRS, IMS laboratory, UMR5218, F-33400, Talence, France.
Canovi A; Bordeaux University, CNRS, IMS laboratory, UMR5218, F-33400, Talence, France.
Chappe YL; Bordeaux University, CNRS, IMS laboratory, UMR5218, F-33400, Talence, France.
Poulletier De Gannes F; Bordeaux University, CNRS, IMS laboratory, UMR5218, F-33400, Talence, France.
Hurtier A; Bordeaux University, CNRS, IMS laboratory, UMR5218, F-33400, Talence, France.
Garenne A; Bordeaux University, CNRS, IMS laboratory, UMR5218, F-33400, Talence, France.
Lagroye I; Bordeaux University, CNRS, IMS laboratory, UMR5218, F-33400, Talence, France.; Paris Sciences et Lettres Research University, F-75006, Paris, France.
Moisan F; Bordeaux University, INSERM, BMGIC Laboratory, UMR1035, F-33000, Bordeaux, France.
Cario M; Bordeaux University, INSERM, BMGIC Laboratory, UMR1035, F-33000, Bordeaux, France.
Lévêque P; Limoges University, CNRS, XLIM, UMR 7252, F-87000, Limoges, France.
Arnaud-Cormos D; Limoges University, CNRS, XLIM, UMR 7252, F-87000, Limoges, France.; Institut Universitaire de France (IUF), F-75005, Paris, France.
Percherancier Y; Bordeaux University, CNRS, IMS laboratory, UMR5218, F-33400, Talence, France. .
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Źródło:
Scientific reports [Sci Rep] 2023 May 23; Vol. 13 (1), pp. 8305. Date of Electronic Publication: 2023 May 23.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Electromagnetic Fields*/adverse effects
Extracellular Signal-Regulated MAP Kinases*
Fibroblasts*
Keratinocytes*
Humans
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Downregulation of ROR2 attenuates LPS-induced A549 cell injury through JNK and ERK signaling pathways.
Autorzy:
Wang Z; Department of Anesthesiology, Yongchuan Hospital Affiliated to Chongqing Medical University, Yongchuan, People's Republic of China.
Yang L; Department of Anesthesiology, Yongchuan Hospital Affiliated to Chongqing Medical University, Yongchuan, People's Republic of China.
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Źródło:
Immunity, inflammation and disease [Immun Inflamm Dis] 2023 Apr; Vol. 11 (4), pp. e803.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Acute Lung Injury*/chemically induced
Acute Lung Injury*/metabolism
Extracellular Signal-Regulated MAP Kinases*/genetics
Extracellular Signal-Regulated MAP Kinases*/metabolism
Animals ; Humans ; Mice ; A549 Cells ; Down-Regulation ; Lipopolysaccharides/toxicity ; Receptor Tyrosine Kinase-like Orphan Receptors/genetics ; Receptor Tyrosine Kinase-like Orphan Receptors/metabolism ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Casein Kinase 1α as a Novel Factor Affects Thyrotropin Synthesis via PKC/ERK/CREB Signaling.
Autorzy:
Wang B; College of Veterinary Medicine, Yangzhou University, Yangzhou 225009, China.
Zhang J; Institute of Reproduction and Metabolism, Yangzhou University, Yangzhou 225009, China.; Joint International Research Laboratory of Agriculture and Agri-Product Safety, The Ministry of Education of China, Yangzhou University, Yangzhou 225009, China.
Zhang D; College of Veterinary Medicine, Yangzhou University, Yangzhou 225009, China.; Institute of Reproduction and Metabolism, Yangzhou University, Yangzhou 225009, China.
Lu C; College of Veterinary Medicine, Yangzhou University, Yangzhou 225009, China.
Liu H; College of Veterinary Medicine, Yangzhou University, Yangzhou 225009, China.
Gao Q; Institute of Reproduction and Metabolism, Yangzhou University, Yangzhou 225009, China.
Niu T; College of Veterinary Medicine, Yangzhou University, Yangzhou 225009, China.
Yin M; College of Veterinary Medicine, Yangzhou University, Yangzhou 225009, China.
Cui S; College of Veterinary Medicine, Yangzhou University, Yangzhou 225009, China.; Institute of Reproduction and Metabolism, Yangzhou University, Yangzhou 225009, China.; Jiangsu Co-Innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, Yangzhou 225009, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Apr 11; Vol. 24 (8). Date of Electronic Publication: 2023 Apr 11.
Typ publikacji:
Journal Article
MeSH Terms:
Casein Kinases*/metabolism
Extracellular Signal-Regulated MAP Kinases*/metabolism
Thyrotropin*/metabolism
Animals ; Mice ; Pituitary Gland/metabolism ; Thyrotropin-Releasing Hormone/metabolism ; Thyroxine/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Redundant roles of extra-cellular signal-regulated kinase (ERK) 1 and 2 in the G1-S transition and etoposide-induced G2/M checkpoint in HCT116 cells.
Autorzy:
Erdenebaatar P; Division of Molecular Cell Signaling, Cancer Research Institute, Kanazawa University, Kanazawa, Japan.
Gunarta IK; Division of Molecular Cell Signaling, Cancer Research Institute, Kanazawa University, Kanazawa, Japan.
Suzuki R; Division of Molecular Cell Signaling, Cancer Research Institute, Kanazawa University, Kanazawa, Japan.
Odongoo R; Division of Molecular Cell Signaling, Cancer Research Institute, Kanazawa University, Kanazawa, Japan.
Fujii T; Department of Biochemistry, Faculty of Pharmacy, Osaka Medical and Pharmaceutical University, Takatsuki, Japan.
Fukunaga R; Department of Biochemistry, Faculty of Pharmacy, Osaka Medical and Pharmaceutical University, Takatsuki, Japan.
Kanemaki MT; Department of Chromosome Science, National Institute of Genetics, Research Organization of Information and Systems (ROIS), Mishima, Japan.; Department of Genetics, The Graduate University for Advanced Studies (SOKENDAI), Mishima, Japan.
Yoshioka K; Division of Molecular Cell Signaling, Cancer Research Institute, Kanazawa University, Kanazawa, Japan.
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Źródło:
Drug discoveries & therapeutics [Drug Discov Ther] 2023 Mar 11; Vol. 17 (1), pp. 10-17. Date of Electronic Publication: 2023 Jan 14.
Typ publikacji:
Journal Article
MeSH Terms:
Extracellular Signal-Regulated MAP Kinases*
Apoptosis*
Humans ; Etoposide ; HCT116 Cells ; G2 Phase Cell Cycle Checkpoints ; Cell Line, Tumor ; Phosphorylation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Caveolae-associated protein 3 promotes adipogenic differentiation of porcine preadipocytes by promoting extracellular signal-regulated kinase phosphorylation.
Autorzy:
Shi M; College of Animal Science, Shanxi Agricultural University, Taigu, China.
Yang S; College of Animal Science, Shanxi Agricultural University, Taigu, China.
Huang X; College of Animal Science, Shanxi Agricultural University, Taigu, China.
Wang S; College of Animal Science, Shanxi Agricultural University, Taigu, China.
Li W; College of Animal Science, Shanxi Agricultural University, Taigu, China.
Yun J; College of Animal Science, Shanxi Agricultural University, Taigu, China.
Lu C; College of Animal Science, Shanxi Agricultural University, Taigu, China.
Yang Y; College of Animal Science, Shanxi Agricultural University, Taigu, China.
Cai C; College of Animal Science, Shanxi Agricultural University, Taigu, China.
Gao P; College of Animal Science, Shanxi Agricultural University, Taigu, China.
Guo X; College of Animal Science, Shanxi Agricultural University, Taigu, China.
Li B; College of Animal Science, Shanxi Agricultural University, Taigu, China.
Cao G; College of Animal Science, Shanxi Agricultural University, Taigu, China.
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Źródło:
Animal science journal = Nihon chikusan Gakkaiho [Anim Sci J] 2023 Jan-Dec; Vol. 94 (1), pp. e13822.
Typ publikacji:
Journal Article
MeSH Terms:
Extracellular Signal-Regulated MAP Kinases*/metabolism
Extracellular Signal-Regulated MAP Kinases*/pharmacology
Caveolae*/metabolism
Swine ; Animals ; Phosphorylation ; Adipocytes/metabolism ; Cell Differentiation/genetics ; Adipogenesis/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Chemokine CCL7 mediates trigeminal neuropathic pain via CCR2/CCR3-ERK pathway in the trigeminal ganglion of mice.
Autorzy:
Zhu LP; Institute of Pain Medicine and Special Environmental Medicine, Co-Innovation Center of Neuroregeneration, Nantong University, Nantong, China.
Xu ML; Institute of Pain Medicine and Special Environmental Medicine, Co-Innovation Center of Neuroregeneration, Nantong University, Nantong, China.
Yuan BT; Institute of Pain Medicine and Special Environmental Medicine, Co-Innovation Center of Neuroregeneration, Nantong University, Nantong, China.
Ma LJ; Institute of Pain Medicine and Special Environmental Medicine, Co-Innovation Center of Neuroregeneration, Nantong University, Nantong, China.
Gao YJ; Institute of Pain Medicine and Special Environmental Medicine, Co-Innovation Center of Neuroregeneration, Nantong University, Nantong, China.
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Źródło:
Molecular pain [Mol Pain] 2023 Jan-Dec; Vol. 19, pp. 17448069231169373.
Typ publikacji:
Journal Article
MeSH Terms:
Chemokine CCL7*/metabolism
Chemokine CCL7*/pharmacology
Extracellular Signal-Regulated MAP Kinases*/metabolism
Neuralgia*/metabolism
Trigeminal Neuralgia*/metabolism
Animals ; Mice ; Chemokine CCL2/metabolism ; Hyperalgesia/metabolism ; Ligands ; MAP Kinase Signaling System ; Trigeminal Ganglion/metabolism ; Receptors, CCR2/metabolism ; Receptors, CCR3/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Mechanism of autophagy induced by activation of the AMPK/ERK/mTOR signaling pathway after TRIM22-mediated DENV-2 infection of HUVECs.
Autorzy:
Wu N; Chemistry and Biochemistry Laboratory, Guizhou Medical University, Guiyang, China.
Gou X; Chemistry and Biochemistry Laboratory, Guizhou Medical University, Guiyang, China.
Hu P; Chemistry and Biochemistry Laboratory, Guizhou Medical University, Guiyang, China.
Chen Y; Chemistry and Biochemistry Laboratory, Guizhou Medical University, Guiyang, China.
Ji J; Chemistry and Biochemistry Laboratory, Guizhou Medical University, Guiyang, China.
Wang Y; Chemistry and Biochemistry Laboratory, Guizhou Medical University, Guiyang, China.
Zuo L; Department of Immunology, Guizhou Medical University, Guiyang, China. .
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Źródło:
Virology journal [Virol J] 2022 Dec 31; Vol. 19 (1), pp. 228. Date of Electronic Publication: 2022 Dec 31.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
AMP-Activated Protein Kinases*/genetics
AMP-Activated Protein Kinases*/metabolism
Extracellular Signal-Regulated MAP Kinases*/metabolism
Humans ; Human Umbilical Vein Endothelial Cells ; Sirolimus/pharmacology ; Antilymphocyte Serum/pharmacology ; Signal Transduction ; TOR Serine-Threonine Kinases/genetics ; TOR Serine-Threonine Kinases/metabolism ; TOR Serine-Threonine Kinases/pharmacology ; Autophagy ; Tripartite Motif Proteins/genetics ; Tripartite Motif Proteins/pharmacology ; Repressor Proteins/metabolism ; Minor Histocompatibility Antigens/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
NeiyiKangfu tablets control the progression of endometriosis through inhibiting RAF/MEK/ERK signal pathway by targeting RKIP.
Autorzy:
Wen Y; Department of Gynecology, Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Fan L; Department of Gynecology, Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Pang L; Department of Gynecology, Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Zhao T; Department of Gynecology, Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Li R; Department of Gynecology, Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Zhang Y; Department of Gynecology, Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Zhang L; Department of Gynecology, Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Yang W; Department of Gynecology, Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, China.
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Źródło:
Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology [Gynecol Endocrinol] 2022 Dec; Vol. 38 (12), pp. 1136-1146. Date of Electronic Publication: 2023 Jan 02.
Typ publikacji:
Journal Article
MeSH Terms:
Endometriosis*/drug therapy
Extracellular Signal-Regulated MAP Kinases*/metabolism
Phosphatidylethanolamine Binding Protein*/drug effects
Phosphatidylethanolamine Binding Protein*/metabolism
Phosphatidylethanolamine Binding Protein*/pharmacology
Drugs, Chinese Herbal*
Animals ; Female ; Humans ; Mice ; MAP Kinase Signaling System ; Mitogen-Activated Protein Kinase Kinases/metabolism ; Mitogen-Activated Protein Kinase Kinases/pharmacology ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Relating individual cell division events to single-cell ERK and Akt activity time courses.
Autorzy:
Stern AD; Department of Pharmacological Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Smith GR; Department of Neurology, Center for Advanced Research on Diagnostic Assays, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Santos LC; Department of Pharmacological Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Sarmah D; Department of Chemical and Biomolecular Engineering, Clemson University, Clemson, SC, USA.
Zhang X; School of Computing, Clemson University, Clemson, SC, USA.
Lu X; Department of Chemical and Biomolecular Engineering, Clemson University, Clemson, SC, USA.
Iuricich F; School of Computing, Clemson University, Clemson, SC, USA.
Pandey G; Department of Genetics and Genomic Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Iyengar R; Department of Pharmacological Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Birtwistle MR; Department of Pharmacological Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA. .; Department of Chemical and Biomolecular Engineering, Clemson University, Clemson, SC, USA. .
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Źródło:
Scientific reports [Sci Rep] 2022 Oct 27; Vol. 12 (1), pp. 18077. Date of Electronic Publication: 2022 Oct 27.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Proto-Oncogene Proteins c-akt*/metabolism
Extracellular Signal-Regulated MAP Kinases*/metabolism
Animals ; Signal Transduction ; Cell Division ; Cell Cycle ; Mammals/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Increased Levels of Phosphorylated ERK Induce CTGF Expression in Autophagy-Deficient Mouse Hepatocytes.
Autorzy:
Seo HY; Department of Internal Medicine, Keimyung University School of Medicine, Daegu 42601, Korea.; Institute for Medical Science, Keimyung University School of Medicine, Daegu 42601, Korea.
Lee SH; Department of Internal Medicine, Keimyung University School of Medicine, Daegu 42601, Korea.; Institute for Medical Science, Keimyung University School of Medicine, Daegu 42601, Korea.
Han E; Department of Internal Medicine, Keimyung University School of Medicine, Daegu 42601, Korea.; Institute for Medical Science, Keimyung University School of Medicine, Daegu 42601, Korea.
Hwang JS; Department of Internal Medicine, Keimyung University School of Medicine, Daegu 42601, Korea.
Kim MK; Department of Internal Medicine, Keimyung University School of Medicine, Daegu 42601, Korea.; Institute for Medical Science, Keimyung University School of Medicine, Daegu 42601, Korea.
Jang BK; Department of Internal Medicine, Keimyung University School of Medicine, Daegu 42601, Korea.; Institute for Medical Science, Keimyung University School of Medicine, Daegu 42601, Korea.
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Źródło:
Cells [Cells] 2022 Aug 30; Vol. 11 (17). Date of Electronic Publication: 2022 Aug 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autophagy*/genetics
Autophagy*/physiology
Connective Tissue Growth Factor*/genetics
Connective Tissue Growth Factor*/metabolism
Extracellular Signal-Regulated MAP Kinases*/metabolism
Animals ; Hepatocytes/metabolism ; Liver Cirrhosis/metabolism ; Mammals/metabolism ; Mice ; Signal Transduction/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
MAPK15 protects from oxidative stress-dependent cellular senescence by inducing the mitophagic process.
Autorzy:
Franci L; Istituto di Fisiologia Clinica (IFC), Consiglio Nazionale delle Ricerche (CNR), Siena, Italy.; Core Research Laboratory (CRL), Istituto per lo Studio la Prevenzione e la Rete Oncologica (ISPRO), Siena, Italy.; Department of Medical Biotechnologies, University of Siena, Siena, Italy.
Tubita A; Department of Experimental and Clinical Biomedical Sciences, University of Firenze, Firenze, Italy.
Bertolino FM; Istituto di Fisiologia Clinica (IFC), Consiglio Nazionale delle Ricerche (CNR), Siena, Italy.; Core Research Laboratory (CRL), Istituto per lo Studio la Prevenzione e la Rete Oncologica (ISPRO), Siena, Italy.
Palma A; Department of Onco-hematology, Gene and Cell Therapy, Bambino Gesù Children's Hospital-IRCCS, Rome, Italy.
Cannino G; Department of Biomedical Sciences, University of Padova, Padova, Italy.
Settembre C; Telethon Institute of Genetics and Medicine (TIGEM), Pozzuoli, Italy.; Department of Clinical Medicine and Surgery, University of Napoli Federico II, Napoli, Italy.
Rasola A; Department of Biomedical Sciences, University of Padova, Padova, Italy.
Rovida E; Department of Experimental and Clinical Biomedical Sciences, University of Firenze, Firenze, Italy.
Chiariello M; Istituto di Fisiologia Clinica (IFC), Consiglio Nazionale delle Ricerche (CNR), Siena, Italy.; Core Research Laboratory (CRL), Istituto per lo Studio la Prevenzione e la Rete Oncologica (ISPRO), Siena, Italy.
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Źródło:
Aging cell [Aging Cell] 2022 Jul; Vol. 21 (7), pp. e13620. Date of Electronic Publication: 2022 Jun 01.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cellular Senescence*/genetics
Cellular Senescence*/physiology
Extracellular Signal-Regulated MAP Kinases*/metabolism
Mitophagy*/genetics
Mitophagy*/physiology
Autophagy/genetics ; Humans ; Oxidative Stress/genetics ; Oxidative Stress/physiology ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
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