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Wyszukujesz frazę ""Zhu, K."" wg kryterium: Autor


Tytuł:
EphrinB/EphB forward signaling in Müller cells causes apoptosis of retinal ganglion cells by increasing tumor necrosis factor alpha production in rat experimental glaucomatous model.
Autorzy:
Liu ST; Department of Neurology, Institutes of Brain Science, State Key Laboratory of Medical Neurobiology, Zhongshan Hospital, Fudan University, 131 Dongan Road, Shanghai, 200032, China.
Zhong SM; Department of Neurology, Institutes of Brain Science, State Key Laboratory of Medical Neurobiology, Zhongshan Hospital, Fudan University, 131 Dongan Road, Shanghai, 200032, China.
Li XY; Department of Neurology, Institutes of Brain Science, State Key Laboratory of Medical Neurobiology, Zhongshan Hospital, Fudan University, 131 Dongan Road, Shanghai, 200032, China.
Gao F; Department of Ophthalmology at Eye & ENT Hospital, Institutes of Brain Science, State Key Laboratory of Medical Neurobiology, Fudan University, Shanghai, 200031, China.
Li F; Department of Neurology, Institutes of Brain Science, State Key Laboratory of Medical Neurobiology, Zhongshan Hospital, Fudan University, 131 Dongan Road, Shanghai, 200032, China.
Zhang ML; Department of Neurology, Institutes of Brain Science, State Key Laboratory of Medical Neurobiology, Zhongshan Hospital, Fudan University, 131 Dongan Road, Shanghai, 200032, China.
Zhu K; Department of Ophthalmology at Eye & ENT Hospital, Institutes of Brain Science, State Key Laboratory of Medical Neurobiology, Fudan University, Shanghai, 200031, China.
Sun XH; Department of Ophthalmology at Eye & ENT Hospital, Institutes of Brain Science, State Key Laboratory of Medical Neurobiology, Fudan University, Shanghai, 200031, China.
Wang X; Department of Neurology, Institutes of Brain Science, State Key Laboratory of Medical Neurobiology, Zhongshan Hospital, Fudan University, 131 Dongan Road, Shanghai, 200032, China.
Miao Y; Department of Neurology, Institutes of Brain Science, State Key Laboratory of Medical Neurobiology, Zhongshan Hospital, Fudan University, 131 Dongan Road, Shanghai, 200032, China. .
Yang XL; Department of Neurology, Institutes of Brain Science, State Key Laboratory of Medical Neurobiology, Zhongshan Hospital, Fudan University, 131 Dongan Road, Shanghai, 200032, China.
Wang Z; Department of Neurology, Institutes of Brain Science, State Key Laboratory of Medical Neurobiology, Zhongshan Hospital, Fudan University, 131 Dongan Road, Shanghai, 200032, China. .
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Źródło:
Acta neuropathologica communications [Acta Neuropathol Commun] 2018 Oct 24; Vol. 6 (1), pp. 111. Date of Electronic Publication: 2018 Oct 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*physiology
Ephrin-B1/*metabolism
Glaucoma/*pathology
Receptors, Eph Family/*metabolism
Retinal Ganglion Cells/*pathology
Tumor Necrosis Factor-alpha/*metabolism
Animals ; Animals, Newborn ; Antioxidants/pharmacology ; Apoptosis/drug effects ; Cells, Cultured ; Chromones/pharmacology ; Disease Models, Animal ; Ephrin-B1/pharmacology ; Excitatory Amino Acid Agents/pharmacology ; Glial Fibrillary Acidic Protein/metabolism ; Intercellular Signaling Peptides and Proteins/pharmacology ; Male ; Morpholines/pharmacology ; Phenols/pharmacology ; Piperidines/pharmacology ; Proline/analogs & derivatives ; Proline/pharmacology ; Pyrimidines/pharmacology ; Rats ; Rats, Sprague-Dawley ; Receptors, Eph Family/genetics ; Receptors, N-Methyl-D-Aspartate/metabolism ; Retinal Ganglion Cells/drug effects ; Signal Transduction ; Thiocarbamates/pharmacology ; Tumor Necrosis Factor-alpha/pharmacology
Czasopismo naukowe
Tytuł:
Blockade of RBP-J-Mediated Notch Signaling Pathway Exacerbates Cardiac Remodeling after Infarction by Increasing Apoptosis in Mice.
Autorzy:
He Y; Department of Cardiology, Zhongda Hospital Affiliated to Southeast University, Nanjing, China.; Medical School of Southeast University, Nanjing, China.
Pang S; Department of Cardiology, Zhongda Hospital Affiliated to Southeast University, Nanjing, China.; Medical School of Southeast University, Nanjing, China.
Huang J; Department of Cardiology, Zhongda Hospital Affiliated to Southeast University, Nanjing, China.; Medical School of Southeast University, Nanjing, China.
Zhu K; Department of Cardiology, Zhongda Hospital Affiliated to Southeast University, Nanjing, China.
Tong J; Department of Cardiology, Zhongda Hospital Affiliated to Southeast University, Nanjing, China.
Tang Y; Department of Cardiovascular Medicine, Georgia Regents University, USA.
Ma G; Department of Cardiology, Zhongda Hospital Affiliated to Southeast University, Nanjing, China.
Chen L; Department of Cardiology, Zhongda Hospital Affiliated to Southeast University, Nanjing, China.
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Źródło:
BioMed research international [Biomed Res Int] 2018 Jul 03; Vol. 2018, pp. 5207031. Date of Electronic Publication: 2018 Jul 03 (Print Publication: 2018).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Immunoglobulin J Recombination Signal Sequence-Binding Protein/*physiology
Myocardial Infarction/*metabolism
Receptors, Notch/*metabolism
Ventricular Remodeling/*physiology
Animals ; Humans ; Mice ; Mice, Knockout ; Mice, Transgenic ; Myocytes, Cardiac ; Signal Transduction
Czasopismo naukowe
Tytuł:
Silencing thioredoxin1 exacerbates damage of astrocytes exposed to OGD/R by aggravating apoptosis through the Actin-Ras2-cAMP-PKA pathway.
Autorzy:
Zhu K; a Department of Pathology , The First People's Hospital of Yibin , Yibin , Sichuan , People's Republic of China.
He Q; b Department of Pathophysiology , Chongqing Medical University , Chongqing , People's Republic of China.; c Institute of Neuroscience , Chongqing Medical University , Chongqing , PR China.
Li L; c Institute of Neuroscience , Chongqing Medical University , Chongqing , PR China.; d Department of Pathology , Chongqing Medical University , Chongqing , People's Republic of China.
Zhao Y; c Institute of Neuroscience , Chongqing Medical University , Chongqing , PR China.; d Department of Pathology , Chongqing Medical University , Chongqing , People's Republic of China.
Zhao J; b Department of Pathophysiology , Chongqing Medical University , Chongqing , People's Republic of China.; c Institute of Neuroscience , Chongqing Medical University , Chongqing , PR China.
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Źródło:
The International journal of neuroscience [Int J Neurosci] 2018 Jun; Vol. 128 (6), pp. 512-519. Date of Electronic Publication: 2017 Nov 16.
Typ publikacji:
Journal Article
MeSH Terms:
Actins/*metabolism
Apoptosis/*physiology
Astrocytes/*metabolism
Cyclic AMP-Dependent Protein Kinases/*metabolism
Neuroprotection/*physiology
Reperfusion Injury/*metabolism
Signal Transduction/*physiology
Thioredoxins/*metabolism
ras Proteins/*metabolism
Animals ; Animals, Newborn ; Disease Models, Animal ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Tytuł:
Non-hemolytic enterotoxin of Bacillus cereus induces apoptosis in Vero cells.
Autorzy:
Liu X; Beijing Advanced Innovation Center for Food Nutrition and Human Health, College of Veterinary Medicine, China Agricultural University, Beijing, China.; National Center for Veterinary Drug Safety Evaluation, College of Veterinary Medicine, China Agricultural University, Beijing, China.
Ding S; National Center for Veterinary Drug Safety Evaluation, College of Veterinary Medicine, China Agricultural University, Beijing, China.
Shi P; The Children's Hospital of Fudan University, Shanghai, China.
Dietrich R; Institute of Food Safety, Department of Veterinary Sciences, Ludwig-Maximilians-University Munich, Oberschleißheim, Germany.
Märtlbauer E; Institute of Food Safety, Department of Veterinary Sciences, Ludwig-Maximilians-University Munich, Oberschleißheim, Germany.
Zhu K; Beijing Advanced Innovation Center for Food Nutrition and Human Health, College of Veterinary Medicine, China Agricultural University, Beijing, China.
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Źródło:
Cellular microbiology [Cell Microbiol] 2017 Apr; Vol. 19 (4). Date of Electronic Publication: 2016 Dec 30.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*immunology
Bacillus cereus/*physiology
Enterotoxins/*physiology
Animals ; Bacterial Toxins/pharmacology ; Calcium Signaling ; Chlorocebus aethiops ; Enterotoxins/pharmacology ; G1 Phase Cell Cycle Checkpoints ; Oxidative Stress ; Receptors, Death Domain/metabolism ; Signal Transduction ; Vero Cells
Czasopismo naukowe
Tytuł:
Dab1 Contributes to Angiotensin II-Induced Apoptosis via p38 Signaling Pathway in Podocytes.
Autorzy:
Gao Z; Division of Nephrology, Renmin Hospital of Wuhan University, 238 Jiefang Rd., Wuhan, Hubei 430060, China.
Chen X; Division of Nephrology, Renmin Hospital of Wuhan University, 238 Jiefang Rd., Wuhan, Hubei 430060, China.
Zhu K; Division of Nephrology, Renmin Hospital of Wuhan University, 238 Jiefang Rd., Wuhan, Hubei 430060, China.
Zeng P; Division of Nephrology, Renmin Hospital of Wuhan University, 238 Jiefang Rd., Wuhan, Hubei 430060, China.
Ding G; Division of Nephrology, Renmin Hospital of Wuhan University, 238 Jiefang Rd., Wuhan, Hubei 430060, China.
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Źródło:
BioMed research international [Biomed Res Int] 2017; Vol. 2017, pp. 2484303. Date of Electronic Publication: 2017 Jun 05.
Typ publikacji:
Journal Article
MeSH Terms:
Adaptor Proteins, Signal Transducing/*metabolism
Angiotensin II/*pharmacology
Apoptosis/*drug effects
MAP Kinase Signaling System/*drug effects
Nerve Tissue Proteins/*metabolism
Podocytes/*metabolism
p38 Mitogen-Activated Protein Kinases/*metabolism
Animals ; Male ; Podocytes/pathology ; Rats ; Rats, Sprague-Dawley ; Reelin Protein
Czasopismo naukowe
Tytuł:
Differential regulation of cellular target genes by p53 devoid of the PXXP motifs with impaired apoptotic activity.
Autorzy:
Zhu J; Institute of Molecular Medicine and Genetics, Medical College of Georgia, Augusta 30912, USA.
Jiang J
Zhou W
Zhu K
Chen X
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Źródło:
Oncogene [Oncogene] 1999 Mar 25; Vol. 18 (12), pp. 2149-55.
Typ publikacji:
Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
MeSH Terms:
Gene Expression Regulation*
Apoptosis/*genetics
Proline/*genetics
Repetitive Sequences, Amino Acid/*genetics
Tumor Suppressor Protein p53/*genetics
Cell Cycle/genetics ; Humans ; Sequence Deletion
Czasopismo naukowe

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